Cardizem diet

44M 5'11". Starting and highest weight 333, target - honestly 250 would be a huge win and I'm not trying to be greedy beyond that. I was keeping many years of 300-305 pre-Covid, then sigh, home baking and junk food took hold.
Comorbidites - distinctly ungood (160/95 range) BP even with meds (losartan, Cardizem). And this last year got a big wake up call from my liver. Enzymes only mildly elevated but ultrasound looked scary. Biopsy and MRI called it stage 3 NASH. Not cirrhosis, yet, but s**t's gotta change. Just barely pre-diabetic (5.8 A1c) but both my parents were full-blown diabetics (and both over 40 BMI... and neither lived past 65). I've literally had a doctor say they were shocked I wasn't T2D.
Insurance - Wegovy is on the preferred list, Mounjaro on the diabetes list. I suspect and hope Zepbound will be on the Jan 24 revised list. Really preferred Zepbound to semaglutide for better (preliminary results) liver fat reduction. PA denied, paid $550 (coupon) for my first box. My work plan is pretty high deductible so I expect to pay roughly that for at least the first 6-8 months of '24 anyway. I'm also seriously looking at going freelance in a year or two, on an ACA plan presumably, and none of those (in my area) cover any anti-obesity meds. So, long-term, I'm just penciling the full price into my long-term budget. Fortunately, between tech jobs and frugality (FIRE), that's not impossible. If it really works... keeps me down in a saner weight and off the liver transplant list, I'll absolutely pay it.
So far - First 2.5 on 12/10 (Sunday evening). Side effects - barely any so far. Some burps and hiccups, but not unpleasantly sulfurous. Dry mouth in the morning (even with really trying to increase water). Some mood elevation and (maybe) a bit more... emotional? Like not in a depressive way, more of an "Ol' Yeller" way? Incredible appetite suppression Monday-Wednesday. Starts fading by Thursday, but I know 2.5 is just a starter dose. Down to 325-ish, though I've been trying to be in 'diet mode' for a week or two before my first shot. I'm sorta counting my 2.5mg month and the holidays as warmup, and really measuring with 5mg and the new year.
Strategy - I'm a big spreadsheet guy, so I tend to weigh every day but really look at a weighted moving average (see "The Hackers Diet"). That's worked for me in the past. And trying to bump up my walking/cycling. I've always been reasonably active for my size, but been bad in the last few years about using my e-bike when I should pedal. Diet - not aiming for keto or anything particular, just healthier. I love salad and tofu. I know how to eat a healthy balanced Mediterranean diet. It's just that if a monster plate of french fries or tortilla chips is in front of me at a restaurant, I will literally never stop eating them.
Anyway, enjoying this group, good luck to us all in 2024.

I’ve been on cardizem 120mg ER for 30 days. While this med did clear my chest pain and deeply relaxed me (like taking a Xanax but better!), it has not controlled my PVCs completely. Because my burden is quite high 20-30%, Dr prescribed Flecinaide 50mg x 2 per day.
Omg I have been PVC free since starting the flecainide. This feels like a miracle drug.
For the longest time I tried to manage these without meds…acupuncture, diet, meditation, box breathing, magnesium, lots of medical testing and making sure my electrolytes are balanced! However, I only continued to get worse!!!!
Having a high burden of PVCs put you at risk of developing cardiomyopathy. This is why my group of cardiologists really pushed meds on me. I fought and fought to remain med free but it was a futile battle.
I am celebrating a PVC-free life at the moment and wanted to share my immediate success with flecinaide. There is hope for us suffering.

I’m currently 12 weeks post op. Surgery was great and post op I continued to follow the diet regimen (including Premier Protein cartons), 64 oz or little more fluid intake, vitamin recommendations, follow up visits, etc. At 10 weeks post op, I ended up in the emergency room 4 times within 7 days for rapid pulse (150-160), foggy mind, dry mouth. I was diagnosed with supraventricular tachycardia (fast heart rate) and given 3 bags of IV fluids, IV Lopressor with positive effects but my pulse elevated right before discharge resulting in overnight observation. ALL my labs came back perfect (bloodwork, CAT scan, EKG, urine). Discharged home with Cardizem as needed for any future episodes of heart rate greater then 140. 2 days later same thing happened and no effect with Cardizem. Went to ER 2 more times afterwards. Doctors couldn’t figure out what was wrong. Cardiologist ordered a Holter monitor and stress test. In the meantime, I noticed that I wasn’t taking in enough Iron, B vitamins (especially B12). I continued to take Bariatric Fusion Multivitamin, Bariatric Fusion Soft Chew Calcium and added Flinstone Multivitamin with iron, Vitamin D3, Vitamin B12. I also added the following drinks to ensure my daily protein and fluid intake (all sugar free): Isopure, Protein2O, Powerade Powerwater with electrolytes, Propel electrolytes, Powerade zero sugar). This has made a drastic difference in my alertness, energy level, and health. Please listen to your body and despite lab results being normal, sometimes additional vitamins is what you need! Take care!

This is me, following up with all of you, regarding my recent surgery with Dr. Goldstein of Bespoke Surgical in NY. I had laser removal and cleanup of irritation and scar tissue, I had 3 anal glands cleanses as well as Botox injected into the superficial skin on Friday November 12th, 2021z My recovery. I flew home the same afternoon, under anesthesia and was okay until arriving at Memphis airport. I took a Percocet. Nov. 13, 2021. I was right as rain, I felt comfortable if a little sore, discharge was coming (and was expected) no bm. I took docusate sodium, miralax, drink a lot of water, Tylenol (off percocet). I ate brown rice, avocados and smoothies. Nov. 14, 2021. I woke feeling okay, I had my first bm after lunch. I inserted a calmol suppository before using the restroom, it went surprisingly well. I continued miralax, docusate sodium (ds) and my high fiber diet. Later in the afternoon, I had another bm, which I had no time for the suppository. It hurt, bleeding, stinging and throbbing. I sat in the bath for the 4th time that day. I noticed a large lump on the perianal site. Nov. 15th, 2021. I woke sore, the sore lump was still there, I put on my cardizem compound, that I had been alternating with lidocaine and went about my day. Taking it easier than the day before. Around 3 I had a bm, plenty of time to put in suppository and use the restroom comfortable. I took a bath. Around 5pm I had the urge for a bm, I tried inserting a suppository to no success. I tried quickly making it to the restroom, I failed and used the bathroom on myself. Misery. But it still didn’t hurt as bad as yesterday. It’s the little things. I’ll try to keep you guys updated.

TDLR: Heart palpitations, SVT, stage 2 hypertension, stomach bloating, and overall body aches since increasing Synthroid. Never had this issue before, thyroid levels normal. No known cause. Symptoms began in the last 30 days. Synthroid increased 6 weeks ago.
EDIT: August 3rd, 2021. I am closer to an answer. I still don’t know with certainty, but it seems I actually did not need to be medicated at all. My thyroid numbers, ALL of them have been perfect since I made this post. I’ve been told by a cardiologist that Synthroid caused me to develop POTS/dysautonomia. The POTS took 4-5 months to go away, but I’ve been symptom free. My heart still races at times, but I’m on a beta-blocker, metoprolol which has helped. I find out next week if the dysautonomia is still present (seeing a neurologist). Will update this post after the appointment.
Hello everyone. This is my first time ever posting on Reddit. While I understand that it’s not good to get medical advice from the internet, I am truly at a loss and don’t know what to do. I’m hoping that in sharing my story, someone else may have some feedback, suggestions, or insights that may help. Also, I am committed to updating this post once I have this issue resolved so that it may help someone else going through similar issues.
In September 2020, I was diagnosed with Hypothyroidism. I had well over 100 strange and seemingly unrelated systems. My doctor ran DOZENS of labs and discovered many things that were out of whack: - Ferritin: 27 (ref range 15-150) - B12: 1971 (ref range 232-1245) - Vitamin D, 25 Hydroxy: 23 (ref range 30-100) - TSH: 7.21 (ref range 0.45-4.50) - Hemoglobin A1C: 5.7 (ref range 4.8- 5.6) - Iron Saturation: 14 (ref range 15-55) - Cholesterol Total: 278 (ref range 100-199)
Everything else was in range.
I was immediately started on 25 mcg of Synthroid, daily. I started taking medical-grade Vitamin D (50,000), once per week, as instructed. I was told to eat more iron-rich food, which I have done, and I take an iron tablet twice, weekly. I also cut my sugar consumption by more than half, and returned to a vegan diet (I was vegetarian at the time).
To be honest, I never started feeling better, although my hair did stop falling out, I stopped gaining weight, and I had slightly more energy.
Then, in mid-December, my doctor told me to take my same dose, but take 2, 25 mcg Synthroid every other day. This is without testing my levels; he based this on my lack of full improvement in symptoms. Two weeks later, ALL my most severe issues started.
What I initially thought were anxiety attacks became a daily occurrence. I always felt out of breath. I had constant heart palpitations. My arms and legs started hurting. Then, the scariest of all symptoms happened. One night at 2am, I was on my way to bed, and I felt like I was going to pass out. I felt hot, nauseous, pain and tension were radiating down both arms, I felt like I was about to poop on myself, and I felt my heart racing like crazy. I put on my Pulse Oximeter and watched my heart rate go from 130 to 160 in a matter of two minutes.
My partner and I both freaked out and called 911. The EMS arrived within 10 minutes and hooked me up to an EKG. They said my oxygen level was 100, my BP was 173/100, and I was in sinus tachycardia. I’ve never had issues with blood pressure before, in fact, it was often lower than normal.
I was advised that I was probably over-medicated, OR that my Synthroid dose was simply settling in. I declined going to the hospital, with this in mind, as I had read about this very thing, in this group. I called my doctor the next day and he said, “Oh, you’re just adjusting. Give it a month. We can do your bloodwork again in 30 days.”
This issue never stopped. In fact, it got worse. Initially, every night around 1am it would start, but once I took magnesium, drank ice water, made a bowel movement, and laid completely still, it would stop. Usually taking about 20-30 minutes, from start to finish.
On January 13, 2021, it started from the moment I woke up, and refused to let up. By 5pm, I was trembling, unable to walk, unable to move more than a few inches, and felt horrible. I checked my BP and it was 180/123, heart rate 163. I called my doctor and he advised me to call 911. Once again, they came and said it was sinus tachycardia, which was never an issue, previously. They also said I was in hypertension crisis, and needed to go to the ER. I was hospitalized for 24 hours.
I was given a chest X-ray, echocardiogram, multiple EKGs, and a CT Scan. Bloodwork was taken. Everything checked out perfectly. And of course, my blood pressure decided to stay at 120/79 the entire time I was in the hospital. They dismissed my issue as an anxiety attack and sent me home. They suggested I start taking a beta-blocker, wrote a prescription, but said it was optional. They also advised me to lose weight (I’ve been fat all my life; this has never been an issue).
My primary doctor got me set up to see a Cardiologist, and said don’t take the beta-blocker until I talked with her. However, five days later, I had another episode and ended up in the hospital. This time, they said it was anxiety, AND because my TSH was now showing up at 1.9, advised that I stop taking Synthroid for at least 30 days to purge any excess from my system.
I saw an endocrinologist during my stay at the hospital, and she said I was probably subclinical and didn’t need to be medicated yet. I asked if she thought it was a cortisol issue because these episodes only happened right when I was going to bed. She wrote me orders to get bloodwork for various things, but said I needed to wait until I had been off Synthroid for 30 days. I was discharged.
I had my appointment with the Cardiologist and was placed on a 48 hour holter monitor. Of COURSE, my blood pressure was mostly normal the entire time and my heart rate never exceeded 130. HOWEVER, it was clear that my resting heart rate has now risen to 100-110. Before taking Synthroid, it was 90. Also, when I sleep, my heart rate normally goes down to the 70s, Now, it’s 95.
Just after I removed the holter, I had a really intense episode (Of COURSE it didn’t get recorded!). I felt deep pain in my upper back, I was dizzy, trembling, my heart rate shot up to 173 while I was sitting on the couch watching TV, I felt like I had to vomit, and I just felt bad everywhere. I went to take a nap, as resting is the only thing that allows the episode to pass.
My partner woke me up 30 minutes later. She said I was pale, (I’m a brown-skinned Black woman). My whole left side felt cold and numb. I was freezing, despite being under the blanket, and my hands looked washed out and blue-ish at the fingertips. My throat and neck got really tight. My partner called 911. When they arrived and hooked me up to the EKG, they said I was having an SVT (supraventricular tachycardia) episode and needed to be rushed to the hospital. At the time, my BP was also 184/123 and I could hardly breathe.
At the hospital, they were afraid I'd had a mini-stroke, so they admitted me and immediately started running all the tests. My heart enzymes were fine. X-Ray normal. Head MRI normal. TSH was 2.9. Potassium, magnesium, calcium, Vitamin D, sodium, kidney function, and liver function, all normal. And once again, my blood pressure remained stable. The only thing they captured was tachycardia. They never even saw the SVT on the EKG, and the ambulance forgot to leave the EKG printing they had taken at my house. I was prescribed Cardizem 60 mg and sent home the next day.
An hour after I got home, I ended up having to call 911 yet again. Something about walking around my house or sitting on the couch triggers my heart rate despite having a very peaceful, loving home with my partner. I was having another SVT episode, but it quickly shifted to sinus tachycardia. This time, I refused being taken to the hospital, because after being hospitalized three times between Jan 13-23, I was fed up.
Also worth noting, I am gluten-free, vegan, and now apparently borderline diabetic. The hospital had very limited food for me to eat, so I just ate a small serving of pineapples, diced potatoes, a tiny side salad, and soy milk while I was there. My hunger annoyed me and made me just want to go home. I’m also a smoker and the nicotine withdrawals drove me nuts. I AM trying to quit, but I’m not there yet.
Anyway, fast-forward to today, my resting heart rate is still 110. I vacillate between being freezing cold or too hot all day. I have a low-grade headache most of the day. I have nightmares two or three times per week. While my anxiety has increased, this is not anxiety-related, nor a panic attack, which the EMT’s confirmed. My feet, arms, upper back, and neck ache. Feels like I pulled a muscle, which I have not. I am tired all the time.
I can’t even do basic household tasks for more than 10 minutes at a time. My heart rate still spikes 1-3 times per day to the point of feeling like I’m going to pass out. My primary doctor keeps telling me to wait for the results of the holter monitor, (won’t have for another day or two, as I had to mail the holter monitor back due to distance and COVID).
I NEVER had these issues until my Synthroid was increased. I have been off of Synthroid for two full weeks AND taking Cardizem twice per day, yet I’m still having these episodes. My blood pressure now averages 145/93, which was NEVER an issue before. My body feels run down, my eyes look unwell, and I just feel horrible. I also have ringing in my ears when my blood pressure is too high. Also, my appetite is half of what it used to be. I now eat 1-2 meals per day, which I often don’t finish, a smoothie, and NO snacks, (which is unlike me).
I have not been given a full thyroid panel yet, but based on what I got at the hospital a few days ago, here are my current numbers:

• TSH with reflex: 2.330 (ref range 0.465- 4.680)
• T3 Free: 3.47 (ref range 2.30-4.8)
• T3 Reverse: 26 (ref range 10-24)
• Also, my B-12 has gone down to 1,000 (ref range 239-931)

At this point, my primary doctor has given me Hydroyzine 25 mg to manage anxiety, which I can’t necessarily take now that I’m on Cardizem (calcium-channel blocker). So that’s out. My cardiologist is suggesting a beta-blocker (Amlodipine 5 mg), but can’t take that either because I’m on Cardizem.
So basically, I’m stuck sitting on the couch all day, forced to take naps to calm my heart down, can’t eat a full meal (because also, the new thing is my stomach feels TOTALLY bloated and full all the time now), and I’m actually afraid to even go to the grocery store because I might faint or have an episode.
I couldn’t even take a shower or bath for a WEEK which is UNHEARD of because I felt so bad. I had the sticky residue from the EKGs, yet it still wasn’t impetus to bathe, because it was literally impossible. My partner offered to help me, but I was just too sick to try. I finally took a bath yesterday and had to wash my hair while sitting down. I do NOT want to live like this. This is not normal for me AT all.
I've started eating better since taking Synthroid, so it’s NOT my diet. I am vegan, and am very careful to ensure I get ALL nutrients. I take Dr. Amen’s Brain MD vitamins and supplements, as I have for the last 10 months because I have ADHD. They have selenium, Vitamin D, and basically all the things we need to be healthy. They are also low iodine, but I get the normal dose of iodine needed by eating seaweed, weekly.
I am vegan, with the exception of D3 and fish oil (because it’s the only thing that helps my ADHD brain). My nutritional levels are on point. I stopped taking the supplements last week, so as to not trip up my thyroid panel with biotin. I can’t exercise at all because I have exercise intolerance and now a high heart rate.
Other details in case you need them: - Black female living in the U.S. - Age: 36 - Height: 5’6", Weight: 225 (gained eight pounds since stopping Synthroid) - I’ve probably had hypo since I was a pre-teen/teenager, because I have had the same hypo symptoms since then and JUST got diagnosed, finally. - I have obstructive sleep apnea, which has been treated via CPAP for almost two years. - I’m a half-a-pack a day smoker. - I meditate, do breath work, stretch daily, drink plenty of ice water, and warm water, and do vagal maneuvers to calm down my sympathetic nervous system. - I have a mostly sedentarily lifestyle; I work from home, as a writer. But I did go for walks and hikes, until it got too cold here. - I do not drink alcohol or do drugs. - My libido has been crap for the last three months. - My skin is dry, and my hair is falling out in clumps again.
No other health problems other than occasional asthma and allergies to shrimp and eggs, which I do not consume. Also, sugar now sends my blood pressure sky high, so I do not add sugar to anything, and have stopped eating sweets, other than vegan yogurt, almond milk creamer in my decaf coffee, and granola bars.
I have also suddenly developed an intolerance to caffeine over the last 30 days, so I avoid black tea and caffeinated coffee. Drinking tea is what probably sent me to the hospital the first time.
I know this is a LOT, but any and all advice is totally welcome. Has anyone else dealt with this? Anyone been able to resolve this? Are there any other tests I should ask for? (My doctor is good about letting me get what I need). I am so frustrated and scared, I just don’t know how to manage anymore. But I can’t live like this. Thanks in advance for reading and for any insights.
P.S. I’ve done some research and it seems my symptoms mimic Postural Orthostatic Tachycardia Syndrome (POTS), particularly Hyperadrenergic POTS since I increased my Synthroid dose. I do have c-PTSD, so it seems my sympathetic nervous system is going haywire. But this was NOT an issue before, except for c-PTSD triggers. But I notice that any effort to move around, even walking from one room to the next now triggers my heart.

I have been employed at a hospital for about 8 months now. It’s my first job out of nursing school, it’s telemetry med-surg. The acuity of the patients on a med surg floor is crazy compared to other places. We take amnio, cardizem , tridil gtts and more. I was recently in a bad code. I take care of 5-6 patients most times 6. I was so frustrated during my shift the other day constantly called an NP abt the same patient. Constantly being talked down to and getting push back when I’m advocating for a patient. She comes in with brain cancer and has been a DNR in the past extremely lethargic and bedbound. I could hear crackles without my stethoscope. And they have full code, a diet order, and pills. No IV acess even the ER couldn’t get her and the PA’s tapped her femoral artery. The NP is telling me have 3 people try anyway for an IV. Yeah let’s torture the women ? Fuck that. I lied and said we did . Finally the NP orders a midline. Then the critical care team calls me and asks me if she really needs it, keep trying to get blood from her. WHAT THE FUCK. Finally get a DNR order. Basically my whole day was arguing with people to do their job. It’s an exhausting struggle. I have a potential job opportunity I have to go for another interview it’s an outpatient mon-fri type gig seems like a much less stressful environment. I feel guilty leaving with being here less than a year like I would come off as ungrateful to my educator and manager. I am barely off probation. But I feel myself getting more and more burnt out, frustrated, and flat out angry. I don’t want to be that nasty jaded nurse taking it out on their co workers. I feel myself slowly getting there. Also everyone who leaves here goes on to a much bigger and better place. I’d be going to a small vascular surgery office. I’m worried I’m not leaving for a big enough opportunity. Then again I don’t want to just pass up on it I could see myself thriving there. Right now I’m just surviving my job. Thoughts ? Sorry for the essay. Thanks.

Fatty liver disease means you have extra fat in your liver. You might hear your doctor call it hepatic steatosis.
Heavy drinking makes you more likely to get it. Over time, too much alcohol leads to a buildup of fat inside your liver cells. This makes it harder for your liver to work.
But you can get fatty liver disease even if you don’t drink a lot of alcohol.
What is Fatty Liver Disease? and How to Get Rid of It?
In the U.S., it’s estimated that fatty liver disease affects 20 to 40 percent of the population. The scary and surprising fact is that this type of liver disease often doesn’t cause any obvious fatty liver disease symptoms. Many people don’t even realize their livers are fatty until they have testing (such as a CT scan or ultrasound) for another health concern or they experience an obvious alert such as pain in the abdomen.
There are two main types of this disease: nonalcoholic fatty liver disease (NAFLD) and alcoholic fatty liver disease. Having a fatty liver can leave you feeling tired or cause mild abdominal discomfort (fatty liver pain), but otherwise you may not have any symptoms. Signs of fatty liver can also include a feeling of fullness in the middle or upper right side of the abdomen, nausea, weight loss and a decrease in appetite. These are just some of the possible symptoms of fatty liver.
Is a fatty liver dangerous? It can be! The more severe type of NAFLD is NASH, which stands for non-alcoholic steatohepatitis. NASH causes the liver to swell and become damaged. According to the American Liver Foundation, NASH is one of the top causes of cirrhosis in adults in the United States and up to 25 percent of adults with NASH may already have cirrhosis. NAFLD is also linked to an increased risk of liver cancer.
Before you jump to the conclusion that a fatty liver may be a likely part of your future or a diagnosis you can’t do anything about, the good news is that the liver is highly regenerative — in fact, it’s the only organ that can regenerate itself.
That’s why it’s not that surprising that with the right fatty liver disease diet and some powerful yet easy-to-do fatty liver home remedies, you may be able to improve the state of your liver starting today!

What Is Fatty Liver Disease?

The human body’s largest organ is the liver. This vital organ stores energy, helps to digest food and removes toxins and poisons.
What is fatty liver disease? Fatty liver disease is a common health problem that results from a buildup of extra fat in the liver. It’s totally normal for the liver to contain a small amount of fat, but when fat storage reaches 5 to 15 percent of the liver’s total weight, then a person is said to have fatty liver disease.
The severity of a fatty liver (also called steatosis) can be a grade 1, grade 2 or grade 3 fatty liver. A fatty liver grade 2 is more serious and more likely to cause symptoms as compared to fatty liver grade 1. The more fat in the liver, the higher the grade and the more likely for the fatty liver to cause problems. There are also different kinds of fatty liver disease.

Nonalcoholic Fatty Liver Disease vs. Alcoholic Fatty Liver Disease

There are two main forms of fatty liver disease: non-alcoholic fatty liver disease (NAFLD) and alcoholic fatty liver disease. What causes fatty liver? In NAFLD, people have a buildup of fat in the liver that is not caused by alcohol use.
However, the build up of fat and damage to the liver is similar to what occurs in a case of alcoholic fatty liver disease, which is directly linked to consuming large quantities of alcohol. A “large amount” of alcohol is typically usually said to be more than one drink per day on average for women and more than two drinks per day on average for men.

Fatty Liver Symptoms

Now that you know what is fatty liver, it’s time to take a look at some of the ways you can identify this disease by knowing the common nonalcoholic fatty liver disease symptoms. First, it’s important to note that it is possible to have non-alcoholic fatty liver disease and exhibit no symptoms. This is actually quite common. When symptoms do occur, there are several possibilities.
Signs of a fatty liver (due to alcohol or other causes) can include:

• Fatigue
• Weakness
• Loss of appetite
• Nausea
• Abdominal pain especially in the upper right abdomen
• Swelling in the upper abdomen
• Spider-like blood vessels
• Itching
• Jaundice (yellowing of the skin and eyes)
• A build up of fluid and swelling of the legs (edema) and abdomen (ascites)
• Mental confusion

Fatty Liver Causes

What causes a fatty liver? An excessive accumulation of fat leads to a fatty liver. In a diagnosis of alcoholic fatty liver disease, the main cause of fat accumulation is consuming large amounts of alcohol.
Suspected causes of nonalcoholic fatty liver disease (NAFLD) as well as non-alcoholic steatohepatitis (NASH) include:

• Being overweight or obese
• Insulin resistance
• High blood sugar (hyperglycemia), indicating prediabetes or actual type 2 diabetes
• High levels of fats, specifically triglycerides, in the blood

All of these possible causes seem to encourage excess deposits of fat within the liver with obesity likely being the most prevalent cause.
According to American College of Gastroenterology,

NAFLD is a very common disorder affecting and may affect as many as one in three to one in five adults and around one in ten children in the United States. Obesity is thought to be the most common cause of fatty infiltration of the liver. Some experts estimate that about two thirds of obese adults and half of obese children may have fatty liver.

Fatty Liver Risk Factors

What are the risk factors for fatty liver disease? Nonalcoholic fatty liver (NAFLD) is more common in people who exhibit any of these health problems or features:

• Obesity
• Type 2 diabetes
• Prediabetes
• Metabolic syndrome or another metabolic disorder
• Hispanic
• Rapid weight loss
• High blood pressure
• Middle aged or older (NAFLD is most prevalent in people who are middle-aged or older, but younger people including children can also have NAFLD)
• Certain infections like hepatitis C
• Take certain drugs including corticosteroids and certain cancer drugs
• Exposure to some toxins

According to the Cleveland Clinic, “Some genetic metabolic conditions or prescription medications, including amiodarone (Cordarone®), diltiazem (Cardizem®), steroids, and tamoxifen (Nolvadex®) also may increase the risk of non-alcoholic fatty liver disease. If you are taking one of these medications and are diagnosed with fatty liver, your doctor might substitute another drug.”
Is having a fatty liver life threatening? If left untreated and it progresses, a fatty liver can definitely be life threatening. Let’s take a look at possible fatty liver health complications.

Fatty Liver Complications

Non-alcoholic fatty liver disease (NAFLD) is the most frequent cause of liver disease in the Western world. Having fatty liver disease means that you have fat deposits within your liver and these undesirable deposits can prevent the liver from doing its important job (and what’s so crucial to optimal health) — to remove toxins from your blood. This puts someone with this liver disease at serious risk for toxic buildup and all kinds of unpleasant symptoms of a poorly functioning liver including jaundice, edema, chronic fatigue, nausea and more.
When non-alcoholic fatty liver disease progresses and becomes severe, it is then non-alcoholic steatohepatitis (NASH). According to the National Institutes of Health (NIH), between 7 and 30 percent of people with NAFLD will develop NASH and at least one-third of people with NASH eventually develop cirrhosis.
The late-stage scarring of the liver known as cirrhosis is the main complication of NAFLD and NASH. When cirrhosis occurs, liver cells are progressively replaced by scar tissue and liver function deteriorates more and more.
According to Mayo Clinic, approximately 20 percent of people with NASH will progress to cirrhosis, which can lead to:

• Abdominal fluid buildup
• Swelling of veins in your esophagus, which can rupture and bleed
• Confusion, drowsiness and slurred speech
• Liver cancer
• End-stage liver failure (the liver has stopped functioning)
• The need for a liver transplant

People with NAFLD, NASH and cirrhosis are all believed to be at an increased risk of developing liver cancer.

Conventional Treatment of Fatty Liver

To diagnose a fatty liver, your doctor will likely perform blood and imaging tests. A liver biopsy may also be necessary.
Common conventional treatment recommendations for fatty liver disease involve eliminating or controlling the cause(s). For example, according to the National Institute of Diabetes and Digestive and Kidney Diseases, conventional fatty liver treatment may includes a doctor’s recommendation to lose weight loss since which helps to decrease fat in the liver as well as inflammation and fibrosis.
Rapid weight loss, however, is not a good idea since this can only make things worse. Gradually losing weight is the smart way to go. Some doctors may advise aiming to lose around 7 percent of body weight over a one year time frame.
Other recommendations may include:

• Stop taking any drug that may be causing fatty liver
• Better control diabetes
• Lower triglyceride levels
• Stop consuming alcohol

There are currently no approved medications to specifically treat NAFLD and NASH. Some doctors give their fatty liver patients vitamin E and thiazolidinediones (a class of drugs, including rosiglitazone and pioglitazone, commonly taken for diabetes) when alcohol is not the cause. However, these drugs can cause adverse effects and some say they don’t make enough of a difference long-term.
When NASH leads to cirrhosis, conventional treatment typically includes certain medications and possibly operations or procedures. When cirrhosis leads to liver failure, a liver transplant can be required.

Fatty Liver Diet and Supplementation

Are you wondering, how can I reduce my fatty liver? Some of the main natural tactics include following a healthy fatty liver disease diet loaded with whole foods. There are also a number of supplements that can be helpful as well.

1. Foods to Boost Liver Function

What foods to eat if you have a fatty liver? In general, you will want to incorporate more plant-based diet foods and choose helpful fatty liver foods including vegetables, fruits and healthy fats.
The following helpful foods are known to decrease inflammation while also aiding the body in its use of insulin, which is often a problem for people with a fatty liver:

• Foods high in monounsaturated fats like olives, olive oil, avocados and nuts.
• Foods rich in omega 3 fatty acids including wild-caught fish like salmon and sardines, chia seeds, flaxseeds and walnuts.
• High antioxidant foods that can help to protect the liver cells from damage, especially items rich in vitamin E, which some research shows can improve the state of a fatty liver. Some healthy foods high in vitamin E include sunflower seeds and almonds.
• Liver boosting vegetables including artichokes and bitter, leafy greens like mustard greens, chicory, arugul, and dandelion.
• Green tea, which is rich in catechins, has been shown to help decrease body fat and discourage obesity.

2. A Mediterranean and Ketogenic Diet

You may want to consider following a Mediterranean diet, which some some studies have suggested can decrease fat in the liver. This diet emphasizes vegetables, fruits, whole grains, legumes, beneficial fat sources like extra virgin olive oil, and healthy proteins like sardines and other omega-3 fatty acid rich fish.
You can take it one step further and follow a Mediterranean ketogenic diet. A pilot study published in 2011 in the Journal of Medicinal Food finds that this type of combined diet plan which focuses on whole foods (especially healthy fats, proteins, and vegetables) can help people overcome metabolic syndrome, which is closely associated with nonalcoholic fatty liver disease.
So for people struggling with a fatty liver linked to metabolic syndrome, this study shows that the potential therapeutic properties of a Mediterranean ketogenic diet can improve metabolic syndrome along with NAFLD.

3. What Foods to Avoid

If you’re looking to follow a fatty liver diet menu and lifestyle that can help to improve the state of your liver, there are some things you’ll definitely want to avoid. Of course, excess alcohol is a huge no-no especially if you have alcoholic fatty liver disease.
There are also a number of foods and other items known to have a negative impact on the liver including:

• Too much alcohol or caffeine
• Packaged goods that contain refined vegetable oils, artificial ingredients, sweeteners and colors
• Fruits and vegetables heavily sprayed with chemical pesticides and herbicides (non-organic crops)
• Factory-farm animal products, farm-raised fish or conventional dairy (that has been pasteurized and homogenized)
• Refined sugar as well as too much fructose
• Refined grains
• Uncooked shellfish
• Certain medications (especially acetaminophen)
• Eating poisonous wild mushrooms
• Exposure to harsh chemicals especially endocrine-disrupting chemicals

You can check the LiverTox database provided by the National Institutes of Health to see if any medication, herb or supplement may be linked to liver injury so that you can avoid them as well.

3. Supplements that Boost Liver Health

According to a scientific review published in 2015, there are some supplements that have specifically been shown to help in the treatment of non-alcoholic fatty liver disease including:

• Vitamin E and vitamin C: Since oxidative stress can play a significant role in the development of pathogenesis of NAFLD, antioxidant agents like vitamin E and vitamin C could be beneficial in the treatment of a fatty liver. However, so far study results are unclear. For example, Vitamin E may only be helpful for adults with NASH who do not also have diabetes or cirrhosis.
• Vitamin D: Research has shown that a vitamin D deficiency can result in insulin resistance, metabolic syndrome and NAFLD.
• Resveratrol: Found in red wine as well as foods like red grapes, raw cocoa (dark chocolate), berries and also available in supplement form, resveratrol appears to decrease the severity of NAFLD in animal models.
• Anthocyanin: Animal research points towards anthocyanin‘s ability to decrease fat accumulation in the liver and counteract liver inflammation.
• Green tea extract: Both in vitro and in vivo experiments demonstrate that green tea and EGCG (a type of catechin) may prevent steatosis by decreasing dietary absorption of fats and carbohydrates.
• Garlic: Garlic-derived S-allylmercaptocysteine (SAMC) appears beneficial to nonalcoholic fatty liver disease thanks to its ability to regulate fat storage and glucose metabolism.
• Ginger: May help to prevent NAFLD or slow down its progression to more severe liver diseases.
• Probiotics and prebiotics: As the review points out, “The gut-liver axis is an important pathway in NAFLD development, which is associated with small intestinal bacterial overgrowth and increased intestinal permeability.” So in other words, SIBO and a leaky gut appear to be contributing factors to NAFLD, which is why prebiotics and probiotics can be so beneficial. You can incorporate more prebiotic and probiotic foods into your diet as well as high quality probiotic supplements.
• Cinnamon: May improve insulin resistance and oxidative stress.
• Curcumin: The active component of turmeric, curcumin, may reduce fat deposits in the liver. It may also prevent fatty liver progression with its “potent antioxidant and anti-inflammatory activities.”
• Quercetin: Available as a supplement and also found in foods like apples and onions, quercetin may help to reduce inflammation and may be particularly helpful for people with a fatty liver as well as obesity and/or diabetes.

This review also points out how both epidemiological and animal studies have shown that drinking coffee (opt for organic to eliminate pesticide residues!) on a regular basis can decrease the risk of type 2 diabetes development, which is a known cause/risk factor for fatty liver disease.

4. Detox Your Liver

Check with your doctor first, especially if you have diabetes or struggle with blood sugar issues.

Key Points About Fatty Liver Disease

• What is a fatty liver? A liver which has a fat storage of five to 15 percent of its total weight. This equates to a diagnosis of fatty liver disease.
• A fatty liver can result in no obvious symptoms or it can cause one or more common fatty liver symptoms such as abdominal pain, abdominal swelling, jaundice, nausea, loss of appetite, fatigue, weakness and itching.
• Nonalcoholic fatty liver disease affects people who drink little to no alcohol while alcoholic fatty liver disease is the result of excessive alcohol consumption.
• For people who don’t excessively consume alcohol, other fatty liver causes include being overweight or obese, insulin resistance, high blood sugar and high levels of fats, particularly triglycerides, in the blood.
• A healthy diet for fatty liver should include plenty of whole foods like liver-boosting artichokes and bitter leafy greens, fish rich in omega 3s and olive oil, high vitamin E foods like sunflower seeds and almonds, and green tea.
• Supplements like probiotics, resveratrol, curcumin, vitamin E, vitamin C and vitamin D may help to naturally improve a fatty liver.
• It’s also important to get regular exercise, which can help you to maintain a healthy weight and decrease fat accumulation in the liver.
• Talk with your healthcare provider before changing your diet or trying any new supplements if you have been diagnosed with fatty liver disease.

Everything you need to know about liver cancer
Liver cancer is cancer that begins in the cells of your liver. Your liver is a football-sized organ that sits in the upper right portion of your abdomen, beneath your diaphragm and above your stomach.
Several types of cancer can form in the liver. The most common type of liver cancer is hepatocellular carcinoma, which begins in the main type of liver cell (hepatocyte). Other types of liver cancer, such as intrahepatic cholangiocarcinoma and hepatoblastoma, are much less common.
Not all cancers that affect the liver are considered liver cancer. Cancer that begins in another area of the body — such as the colon, lung or breast — and then spreads to the liver is called metastatic cancer rather than liver cancer. And this type of cancer is named after the organ in which it began — such as metastatic colon cancer to describe cancer that begins in the colon and spreads to the liver. Cancer that spreads to the liver is more common than cancer that begins in the liver cells.

Types:

Hepatocellular carcinoma

Symptoms:

Most people don’t have signs and symptoms in the early stages of primary liver cancer. When signs and symptoms do appear, they may include:

• Losing weight without trying
• Loss of appetite
• Upper abdominal pain
• Nausea and vomiting
• General weakness and fatigue
• Abdominal swelling
• Yellow discoloration of your skin and the whites of your eyes (jaundice)
• White, chalky stools

When to see a doctor

Make an appointment with your doctor if you experience any signs or symptoms that worry you.

Causes

It’s not clear what causes most cases of liver cancer. But in some cases, the cause is known. For instance, chronic infection with certain hepatitis viruses can cause liver cancer.
Liver cancer occurs when liver cells develop changes (mutations) in their DNA — the material that provides instructions for every chemical process in your body. DNA mutations cause changes in these instructions. One result is that cells may begin to grow out of control and eventually form a tumor — a mass of cancerous cells.

Risk factors

Factors that increase the risk of primary liver cancer include:

• Chronic infection with HBV or HCV. Chronic infection with the hepatitis B virus (HBV) or hepatitis C virus (HCV) increases your risk of liver cancer.
• Cirrhosis. This progressive and irreversible condition causes scar tissue to form in your liver and increases your chances of developing liver cancer.
• Certain inherited liver diseases. Liver diseases that can increase the risk of liver cancer include hemochromatosis and Wilson’s disease.
• Diabetes. People with this blood sugar disorder have a greater risk of liver cancer than those who don’t have diabetes.
• Nonalcoholic fatty liver disease. An accumulation of fat in the liver increases the risk of liver cancer.
• Exposure to aflatoxins. Aflatoxins are poisons produced by molds that grow on crops that are stored poorly. Crops such as corn and peanuts can become contaminated with aflatoxins, which can end up in foods made of these products. In the United States, safety regulations limit aflatoxin contamination. Aflatoxin contamination is more common in certain parts of Africa and Asia.
• Excessive alcohol consumption. Consuming more than a moderate amount of alcohol daily over many years can lead to irreversible liver damage and increase your risk of liver cancer.

Prevention

Reduce your risk of cirrhosis

Cirrhosis is scarring of the liver, and it increases the risk of liver cancer. You can reduce your risk of cirrhosis if you:

• Drink alcohol in moderation, if at all. If you choose to drink alcohol, limit the amount you drink. For women, this means no more than one drink a day. For men, this means no more than two drinks a day.
• Maintain a healthy weight. If your current weight is healthy, work to maintain it by choosing a healthy diet and exercising most days of the week. If you need to lose weight, reduce the number of calories you eat each day and increase the amount of exercise you do. Aim to lose weight slowly — 1 or 2 pounds (0.5 to 1 kilograms) each week.
• Use caution with chemicals. Follow instructions on chemicals you use at home or at work.

Get vaccinated against hepatitis B

You can reduce your risk of hepatitis B by receiving the hepatitis B vaccine, which provides more than 90 percent protection for both adults and children. The vaccine can be given to almost anyone, including infants, older adults and those with compromised immune systems.

Take measures to prevent hepatitis C

No vaccine for hepatitis C exists, but you can reduce your risk of infection.

• Know the health status of any sexual partner. Don’t engage in unprotected sex unless you’re certain your partner isn’t infected with HBV, HCV or any other sexually transmitted infection. If you don’t know the health status of your partner, use a condom every time you have sexual intercourse.
• Don’t use intravenous (IV) drugs, but if you do, use a clean needle. Reduce your risk of HCV by not injecting illegal drugs. But if that isn’t an option for you, make sure any needle you use is sterile, and don’t share it. Contaminated drug paraphernalia is a common cause of hepatitis C infection. Take advantage of needle-exchange programs in your community and consider seeking help for your drug use.
• Seek safe, clean shops when getting a piercing or tattoo. Needles that may not be properly sterilized can spread the hepatitis C virus. Before getting a piercing or tattoo, check out the shops in your area and ask staff members about their safety practices. If employees at a shop refuse to answer your questions or don’t take your questions seriously, take that as a sign that the facility isn’t right for you.

Ask your doctor about liver cancer screening

For the general population, screening for liver cancer hasn’t been proved to reduce the risk of dying of liver cancer, so it isn’t generally recommended. The American Association for the Study of Liver Diseases recommends liver cancer screening for those thought to have a high risk, including people who have:

• Hepatitis B and one or more of the following apply: are Asian or African, have liver cirrhosis, or have a family history of liver cancer
• Hepatitis C infection and liver cirrhosis
• Liver cirrhosis from other causes, such as an autoimmune disease, excessive alcohol use, nonalcoholic fatty liver disease and inherited hemochromatosis
• Primary biliary cirrhosis

Discuss the pros and cons of screening with your doctor. Together you can decide whether screening is right for you based on your risk. Screening typically involves an ultrasound exam every six months.
​
Source: http://www.hiwamag.com/health/fatty-liver-disease/

26F. 5'7. 149lbs.
Onset/duration: 4 days ago, approximately 8 hours after whole blood donation.
Disclaimer - I am a paramedic, but am not sure what to do at this point.
Vitals taken while at work today: HR 110, Sinus tach on the cardiac monitor, no abnormalities to my 12-lead. BP 118/80, respirations 22, SpO2 96% on room air.
Past Medical History: Inappropriate sinus tachycardia, ADD, allergy induced asthma, insomnia - post TBI.
Current Medications: Cardizem CR 180mg daily, Adderall XR 20mg, Zovia 1/35mcg (BC), Singulair, Ambien CR, Benadryl 25-50mg per day.
Complaint: I donated whole blood on Friday 11/29 with no significant issues. I have donated plenty of times previously with no issues. Approximately 8 hours after donation I began having noticeable fatigue, then had symptoms of low blood pressure (felt hot, slightly sweaty, dizzy, nauseous). I layed down, continued to drink plenty of fluids (2-3 liters), and ate a protein rich meal. Felt off the rest of the day.
Saturday and Sunday I experienced the same symptoms of hypotension despite continiued fluid intake. Have since lost my appetite and am struggling to eat more than 800 calories a day which is abnormal. Symptoms worsened after sitting for prolonged periods of time.
Monday I did light exercise and had a sustained heart rate of 170+, confirmed manually. I'm used to having my heart rate get into the 170-180 range during physical activity and rest periods. I normally sit 160-170 during activity with decreaseto the 150s during rest periods. I also felt short of breath, similar to before the tachycardia was controlled through medication, diet, and exercise. Through the rest of the day I felt weak/"gassed", short of breath, and continued to have a resting heart rate of 130-140.
Today I felt okay in the morning and slowly began feeling weak and jittery. Symptoms not quite as bad as the previous 3 days.
I have never felt like this after giving blood and am not sure if I need to contact my cardiologist or give it a few more days. I've felt symptoms from my inappropriate sinus tachycardia before, but it has never been sustained for days at a time.

I'm a new grad on an adult medicine unit and I was genuinely feeling pretty positive about how I was doing until a couple days ago when one of my patients started going down hill. He was a 80 something year old male, s/p abdominal surgery, stable BP with a HR sustaining in the 50s. I was going about my shift until PT called me, saying that he became orthostatic when he stood up. I checked his BP and it was 60s/40s, HR 100. Paged the docs, they wanted to bolus him with 1L LR wide open. Up until this point, it all felt pretty straight forward. He hadn't been eating/drinking much because he didn't like the clear liquid diet foods we were offering him. I thought we were going to be fine after the bolus.
I come back after the bolus was done. His pressures were 110s/70s, but now his HR was 140-160, and he was complaining of chest pain and headache. At this point I started getting anxious. My preceptor basically carried me and it all seems like a blur; everything happened so fast that I felt like I didn't have time to think. We did an ECG and he was in afib, rapid response came, and they were talking about giving metop/cardizem, and after several pages, the surgery resident finally came. Long story short he converted back to NSR without any meds (and then he continued to have episodes of afib 2 days in a row, eventually transferred to TCU).
I felt like everything was moving at 100 miles an hour and I didn't even have time to think before my preceptor started doing things. This was my first experience with a patient declining this quickly and I felt almost useless. Any advice on how to be better? I know it takes experience but I'd like to be more prepared for situations like these when they happen again.. How long did it take you to feel comfortable on your unit?

Age: 23
Sex: Female
Height: 5'5"
Weight: 240
Race: Caucasian
Medication: None
Issue: pressure, pain, cannot inhale fully without discomfort or pain
Location (of issue): Upper right abdomen, lower right chest
Duration: 2 days. I don't know exactly when it began as when I went to bed 2 nights ago it didn't exist but when I woke up it did and has ever since.
Details:
This is fairly long. Thank you so much in advance for reading or contributing.
I have scheduled an appointment with a doctor in my PCP's office (not my PCP) because they said it was the first that'd be available on Monday. I am wondering if, based on my symptoms, I should seek sooner consultation/treatment either by going to an urgent care center or ER.
My main symptoms: I cannot inhale fully without a feeling of great pain, pressure, and discomfort in the area on the right hand side that's in my upper abdomen, lower chest. The first day, this discomfort was more on the right and across a wider area (blue). Now it's more centralized, but a smaller area (black). Here's a color-coded picture to help clarify the areas.
I've had no injuries, falls, accidents, trauma, etc.
The discomfort is not constant. Most of the time I feel somewhat normal so long as I do not deeply inhale, and there are others where I feel pain in even shallow breaths (infrequent). Even if I don't deeply inhale, I still feel like my right side in that area is "heavier". Exhaling and coughing does not cause pain, only inhaling. The pain is like a deep sore/dull pain. Similar to the pain of pushing on a large bruise. It fluctuates in intensity. Sometimes it's extremely uncomfortable when sitting, other times it only is felt when deeply inhaling. I am most uncomfortable lying down; side is least uncomfortable (barely noticeable), stomach more (pressure, but no pain unless I move), and on back is most uncomfortable (pressure and pain without moving). Standing or sitting upright makes it less noticeable, but if I deeply inhale in any position it hurts and is uncomfortable.
I don't have a fever or chills. No known allergies, asthma, heart, respiratory, liver, etc. problems. I had blood work done in December for a routine physical and all came back normal except cholesterol (201 mg/dL) and LDL (130) mg/dL which were marked as high. Triglycerides were marked as 66 mg/dL, but I was not told if that was normal or not.
Very brief overview of my medical history since last October:
Early October: Found a PCP because I had what I remember as pulling, twitch-like sensations in my chest (around same area as I'm having issues now). I'd have occasional jaw, neck, ear pain. She suspected heart problems, did EKG, but came back fine. She said to take Advil and see if it helps.
That night, I woke up at 2 AM with a racing pulse, dimmed vision, and felt like I was going to pass out. Was unlike anything I'd ever felt before, immense feeling of doom. Face burning hot and splotchy, body shivering. I was driven to the ER where my blood pressure when taken in was ~220/160 and my pulse was 185. At the doctors office earlier that day it was normal, 115/110. An IV was inserted and was put on oxygen and given a beta blockecalcium channel blocker (forgot which). X-ray, blood work, urinalysis, second ekg, came back fine and was sent home with suspected SVT.
The day after, I went to a cardiologist. Third ekg came back fine, but he said sinus tachycardia. Blood tests were ordered, echocardiogram, stress test, and holter. All results were said to have come back fine. Was prescribed cardizem to take if I have another episode. I have not used it since it was prescribed to me, as I have not had any episodes like those (racing heart, severe chills, etc.)
Three years ago I was extremely healthy, worked out 5x a week, healthy diet, ran often, and was in school. I now weigh 100 pounds heavier and am working to lose weight as I do not want medical problems, I let go of myself and quickly fell into depression.
I am lost as to what to do. Almost always I feel like I can wait until Monday, but when I lie down to sleep I can hardly be in any position without pressure or discomfort. And when I switch around to move, I feel the pain. It's now 6 AM and I've not yet slept. Any ideas what this could be? Does it warrant going to an urgent care or an ER?

I had my blood pressure taken this morning by a nurse. It was 160 over 98 with pulse 60. That is high. I am in my mid fifties. I do not smoke. I need some Reddit advice. I am not strictly speaking asking for medical advice though if any medical people are here I appreciate any advice. I had thought I could deal with this by exercise and diet alone but have failed and now think I need medical help. I have been exercising by bicycling close to 100 miles per week. I have tried to cut back on sodium and eat more fruits and vegetables. Nothing helps. Now there is a secondary issue. I was laid off and have no medical insurance. It was a choice between COBRA insurance and food. My wife is covered by insurance but cannot cover me. With that background, I have some questions I would like to ask.

1. Is there any best medicine for blood pressure reduction that people use around here? Medicine available through Kroger $4 medicine program?
2. How can I get a free visit to a hypertension specialist in Ohio with no insurance?
3. How can I get free medicine? Note that Johnson and Johnson offered my wife some free medicine but we did not need it due to her having insurance. It was wildly expensive medicine like $2000 per dose.
4. Should I call my normal family doctor and tell them I was laid off and cannot pay?
5. The mayoclinic web site mentions a thiazide diuretic that might be the best first treatment for high blood pressure. I wonder if that might be too weak to impact me. They also mention the below:
   Angiotensin-converting enzyme (ACE) inhibitors. Frequently prescribed ACE inhibitors include captopril (Capoten), lisinopril (Prinivil, Zestril) and ramipril (Altace).
   

Angiotensin II receptor blockers. Frequently prescribed angiotensin II receptor blockers include losartan (Cozaar), olmesartan (Benicar) and valsartan (Diovan).
Beta blockers. Frequently prescribed beta blockers include metoprolol (Lopressor, Toprol XL), nadolol (Corgard) and penbutolol (Levatol). (Sounds dangerous)
Calcium channel blockers. Frequently prescribed calcium channel blockers include amlodipine (Norvasc), diltiazem (Cardizem, Dilacor XR) and nifedipine (Adalat, Procardia). (I love drinking gallons of milk.)
I hate taking medicine. My mother and father who are both still alive take gobs of medicine. I do not want my exercise life interrupted. Would any of the above medicines make it hard for me to exercise?
Thanks for any good advice. I know I should and will see a doctor (somehow) but would like to be prepared before that.