I was in the ER 2 weeks ago for a high heart rate and very upset stomach. They sent me home with Pepcid, but since then l've been so weak, can hardly eat, having neck and shoulder pain. My EKG from that ER visit just resulted and it's freaking me out a little. What is ST depression? Should I be worried? I have a GI appointment tomorrow but now I'm concerned this flare is actually my heart.
Result: Sinus tachycardia Rightward axis Cannot rule out Anterior infarct, age undetermined ST & T wave abnormality, consider inferior ischemia Abnormal ECG When compared with ECG of 08-APR-2024 14:37, ST now depressed in Inferior leads T wave inversion no longer evident in Lateral leads

Hi. 30F. 5 feet. 100lbs. Non smoker. On omeprazole. I was in the ER 2 weeks ago for a high heart rate and very upset stomach. They sent me home with Pepcid, but since then I’ve been so weak, can hardly eat, having neck and shoulder pain. My EKG from that ER visit just resulted and it’s freaking me out a little. What is ST depression? Should I be worried? I have a GI appointment tomorrow but now I’m concerned this flare is actually my heart.
Result: Sinus tachycardia Rightward axis Cannot rule out Anterior infarct, age undetermined ST & T wave abnormality, consider inferior ischemia Abnormal ECG When compared with ECG of 08-APR-2024 14:37, ST now depressed in Inferior leads T wave inversion no longer evident in Lateral leads

Please remove if not allowed I have no idea where else to post this.
23F, no related underlying health issues, currently on 40mg Prozac and 20mg Adderall 3-4 days out of the week.
Was in the ER while quitting alcohol and noticed my heart rate was high and wanted to get checked out. I followed up with my PCP today and realized I never saw my test results until I looked in my notes. My last EKG says the following:
SINUS TACHYCARDIA. RIGHT ATRIAL ENLARGEMENT. NONSPECIFIC ST ABNORMALITY. ABNORMAL ECG. WHEN COMPARED WITH ECG OF 06-JAN-2022 10:34,. SINUS RHYTHM HAS REPLACED ECTOPIC ATRIAL RHYTHM. VENT. RATE HAS INCREASED BY 51 BPM. ST NOW DEPRESSED IN INFERIOR LEADS. T WAVE AMPLITUDE HAS INCREASED IN ANTERIOR LEADS
My first ever EKG only said I had a usual P axis and a possible ectopic atrial rhythm. I did follow up with a cardiologist who assured me this wasn’t a serious abnormality and there is no need for further treatment. However, the notes on my most recent EKG seem to be more extensive and is concerning me. I have health anxiety. I do have another appt scheduled with my cardiologist but it’s weeks away, and would appreciate some context from the findings.
Thank you in advance for your time. I know I just need to wait for my cardiologist, but an explanation into the meanings of this stuff would be helpful.

I don't want sexual desires or thoughts, as they are a hindrance and an encumbrance rather than a help. After being blackpilled, I've come to realize, as much as I tried to hide it before, the truth is I hate women and they hate me. The feeling is natural as it is innate. It's an inherent aspect of my being. Ultimately, I consciously hate women. I try my damnedest in real life to avoid women. Whenever I'm on Instagram, I always block any feed with a woman, unless it's something like plastic surgery. I even make sure to steer far away from normie sites. Reddit is normie as fuck, but it's a good therapy place.
My overarching point is that I don't want sexual desires or thoughts. I'm ultimately a narcissist. I care about myself and my looks first. Ultimately, I don't want kids. The whole thing is expensive and ultimately a scam in my opinion.
The issue is my biology. As much as the tossers, the cunts in society act like you can control your sexual desires, they are lying through their fucking teeth.
Down below is what creates a human to be attracted to another human. It's not subjective love, not an ethereal phenomenon. It's a species specification that we share that attracts each other to us to create spawn and offspring.
Pheromone receptor genes: The vomeronasal organ (VNO) expresses receptor genes like V1rs and V2rs that encode vomeronasal receptors (VRs). These VRs bind species-specific pheromones and initiate signaling cascades. For example, the V1rb2 receptor in mice is crucial for male-female recognition.
Olfactory receptor genes: Olfactory receptor (OR) genes like ORs and TAARs encode receptors in the main olfactory epithelium that detect volatile pheromone components. Genetic variation in ORs influences individual differences in odorant perception linked to mate choice.
Neural patterning genes: Homeobox genes (e.g., Lhx6, Emx2) and transcription factors (e.g., Fezf2, Ctip2) regulate the differentiation and connectivity of sexually dimorphic neural circuits involved in processing social/sexual cues.
Now let's look at how these genes influence the underlying neural pathways:
Visual pathway: Retinal photoreceptors transmit signals via the optic nerve to the lateral geniculate nucleus (LGN) of the thalamus. Neurons in the LGN project to the primary visual cortex (V1) where lower-level features are processed.
Ventral stream: V1 projects to extrastriate cortices like V4 for coloform processing and then to the inferotemporal cortex (IT) including the fusiform face area (FFA) specialized for face perception.
Dorsal stream: V1 also projects to the posterior parietal cortex (PPC) crucial for spatial attention and integration of form/motion cues related to biological motion perception.
These ventral/dorsal streams converge in regions like the amygdala, orbitofrontal cortex (OFC), and anterior cingulate cortex (ACC) that assign emotional/motivational salience to socio-sexual cues.
Olfactory/Pheromone pathway: Odorants/pheromones are detected by ORs/VRs which transduce signals to the main/accessory olfactory bulbs. The main olfactory bulb sends projections to the piriform cortex, entorhinal cortex, amygdala, and OFC for conscious odor perception and emotional processing. The accessory olfactory bulb sends VNO projections to the medial amygdala and bed nucleus of the stria terminalis (BNST) which interfaces with the hypothalamus to modulate neuroendocrine responses and motivation.
Integration of Visual/Olfactory inputs: The amygdala, OFC, ACC act as convergence hubs integrating multimodal sensory inputs from vision, olfaction, audition related to conspecific cues. Their outputs to the ventral striatum, particularly the nucleus accumbens, modulate dopaminergic reward/motivation circuits, gating approach/avoidance behaviors.
Hormonal Modulation: Steroid hormones like testosterone, estradiol, and progesterone organize and activate these neural circuits in a sex-specific manner. Testosterone has organizational effects by influencing sexual differentiation of the hypothalamus and amygdala during development. In adulthood, testosterone and its metabolites act as activational signals - binding to androgen receptors (ARs) in the amygdala, BNST, hypothalamus to facilitate male sexual behavior and modulate sensory processing of female cues.
Unfortunately, as of now, there is no technology that can kill off sexual desire. Now let me answer an age-old question asked, "If you could get rid of sexual desire, where would that lead you?"
Simple, no more frustration by women, they wouldn't even register on your mind. I mean, just like how a female lioness doesn't register in my mind, similarly, a female human wouldn't register in mine. In truth, there is no universal beauty. Beauty is always species-specific, and a giga chad in one species isn't a giga chad in another. With no attraction to women, you're no longer being driven by the chains that genes hurl over you. You can actually focus on life without having to deal with all the bullshit nature hurls upon us.

With the development of new, faster, smarter technology, the field of cardiology continues to evolve significantly and attending any Upcoming Cardiology Conference 2024 offers an amazing learning platform to keep up with emerging state-of-the-art technologies in this field.
A Cardiovascular Conference can help you understand and use these trends in your profession. These are great events for researchers, cardiologists, doctors, nurses, medical students, other medical professionals, and cardiovascular device manufacturers who want to stay abreast of the trends.
This blog highlights the top emerging technologies that you will learn about at a good Cardiology Conference in 2024. So, let us get started:

Artificial intelligence (AI) for faster and more accurate diagnosis

Many manual devices, especially those equipped with obsolete technology, may fail to diagnose a patient's heart conditions accurately. This is because older technologies had many limitations that required much innovations to help patients.
AI diagnosis is a brilliant way to understand and analyse medical images faster and more accurately. This technology is helpful in body scans, ECGs, and thoroughly breaking down minute details of patient data. In addition, AI is extremely helpful in determining the heart's working patterns, allowing doctors to provide the best treatment and medications for their patients.
If you attend an International Cardiology Conference 2024, the role of AI in cardiology is one of the most common topics of discussion. At these events, you can learn from esteemed doctors and world-famous medical professionals from around the globe.
3D Imaging and Printing
Having an exact-size model for patients during heart surgery is critical for its success. The technology provides easy-to-understand details of complicated spatial relationships between the heart and vascular system. This understanding is helpful for cardiac surgeons, cardiac radiologists, and cardiologists to successfully manage complex heart diseases.
By attending Cardiology Conferences 2024 India, you will learn how 3D imaging and printing is shaping the cardiology field. Experts, session speakers, industry professionals, researchers, and influential cardiologists attend the Cardiology Conferences 2024 India.

Nanotechnology for cardiovascular disease

Nanotechnology involves working with devices and materials on a nanoscale level, and nanomedicine refers to the use of nanotechnology in healthcare. Nanomedicines are showing huge potential in various cardiac applications because of their unique properties. In simple words, nanomedicines are made of active pharmaceutical ingredients (API) packaged into nano-sized carriers made of excipients.
In addition, Nanotechnology may stimulate angiogenesis from pluripotent or vascular stem cells, resulting in various implications such as wound healing, cardiomyocyte regeneration, and organ recellularization. Similarly, the technology has shown great promise in cardiovascular surgery.
You can learn all about the role of nanotechnology in cardiology health by attending a Cardiology Conference India. These events showcase practical skills and help you gain a clear understanding of nanotechnology in cardiology.

Telecardiology

There are many circumstances where doctors cannot physically check the patient due to remote locations. Telecardiology is a modern medical practice that uses telecommunications for remote diagnosis and treatment of heart patients.
So, telecardiology is a hot topic of discussion at any well-planned International Cardiology Conference. Case studies from cardiologists and researchers will give you exposure to this fastest-emerging cardiovascular technology.

Implantable Cardioverter-Defibrillators (ICDs)

ICDs is a small battery-powered device put in the chest by heart surgeons. The device uses remote monitoring, personalised therapy, and algorithms to detect and prevent arrhythmias. You can learn all about this technology at the Upcoming Cardiology conference 2024. The practical exposure from live workshops and seminars can help you try this technology and learn how to use it in Cardiology.

Final Words

As you see, attending a Cardiology Conference can help you learn about all the latest emerging trends and technologies in this field. Attending these events not only provide knowledge but also give you endless network opportunities with world’s leading cardiologists and researchers. Attend a Cardiology Conference in 2024 today.
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Ive had Afib ONCE before and it was months ago. Ive had palpitations and anxiety since. Tonight Ive been feeling fluttering of my heart and been feeling off. Any ideas about these? ECGs seem normal to me but theres a few spots where it’s different sizes, but heart rate is not high.

This may not adhere to the rules of this subreddit, so please free to delete my post.
I, a Cardiology Technologist, go to a room to do an ECG due to a new tachycardia. I hooked up, and saw the patient was in a SVT of approximately 160-170 bpm. My overconfident brain thought that there was a STEMI going on due to some weird QRS morphology in the inferior leads, and from where I was standing, thought it had ST elevation and I thought the slurred S-waves in lead 1 was reciprocal ST depression. There was a pre-existing RBBB (face palm my dumbass face).
My idiotic self goes to the nurse “please call a doctor asap (who was just around the corner) for a potential STEMI.” Nope, the patient just has a weird RBBB morphology in the inferior leads after pulling up their old ECG.
I jumped the gun, and panicked the nurse and the patient (probably, though the patient was pretty out of it). So, yeah, ... I really need to calm down, read, and interpret the ECG instead of jumping to conclusions. The resident covering was still going to come because of the tachycardia (was unaware of it).
So, yeah… that was a humbling and embarrassing moment in my professional experience. And that resident probably thinks that I'm an idiot now for calling it a STEMI ... Sigh. I should know better.

This may not adhere to the rules of this subreddit, so please free to delete my post.
I go to a room to do a stat ECG due to tachycardia. I hooked up, and saw the patient was in a SVT of like 160-170 bpm. My overconfident brain thought there was a STEMI going on due to some weird QRS morphology in the inferior leads, and from where I was standing, thought it had ST elevation. There was a pre-existing RBBB.
My dumb idiotic brain goes to the nurse “please call a doctor asap (who was just around the corner) for a potential STEMI.” Nope, the patient just has a weird RBBB morphology in the inferior leads after pulling up their old ECG.
I jumped the gun, and panicked the nurse and the patient (probably, though the patient was pretty out of it).
So, yeah. Calm down, read and interpret ECG instead of jumping the gun. The resident covering was still going to come because of the tachycardia (was unaware of it).
So, yeah… that was a humbling and embarrassing moment in my experience.

She says she does not feel right. Palpitations. What do you see?

(Please know that I am not a medical professional, I am only stating what has worked for me and sharing with the hopes it may help someone else who suffers with this)
For about the past 4 years, I’ve (M33) had a really bad particular anxiety/panic attack where whenever I do any kind of physical exertion (ESPECIALLY WALKING UP STAIRS) I would start to hyperventilate. This all started from a really bad panic attack I had at the start of Covid where I ran up a flight of 3 stairs and then I thought I was having a heart attack after. (I have since had an ECG to rule out heart problems and my heart is fine).
For the past 4 years, I’ve literally not been able to go up 2 flights of stairs without starting to feel like I’m gasping for air once I got to the top. This would be followed by me hyperfocusing on my breathing which would always lead to hyperventilating. To cope, I would always stop half way at the platform between to two flights, wait a minute or two and pretend I was looking at my phone to try and avoid embarrassment or not look crazy. 3 flights of stairs, forget it. I would avoid it all together if there was an elevator available because I knew it’d be a full on panic attack waiting.
This controlled my life in many ways. I couldn’t walk any natural inclinations, such as a hill without starting to hyperventilate. I would have to walk up them at a snail’s pace to try and avoid it from happening. I used to love hiking but this made it nearly impossible. I also couldn’t do much exercise without starting to hyperventilate. This had ruined my goals of trying to get in shape.
Well, after 4 long years of suffering with this, it’s finally starting to lose its grip. Just this past week at my job, I was finally able to walk up 2 flights without panicking! And it wasn’t just a fluke, I did it over and over again to make sure.
I found that the answer was in my abnormal breathing😮‍💨 . I’m not new to panic attacks and anxiety, I’ve had it for roughly over a decade. Previously, mental health professionals and other medical professionals would always tell me about taking deep breaths. That used to work, but it didn’t work for this particular anxiety attack -in fact trying to take deep breaths only made it worse. It was actually the opposite - I had to learn to breathe more shallow.
Essentially, every time I walked up the stairs, I would anticipate panic and subconsciously was starting to take in deep breaths. This wasn’t helping as at this point I’m getting too much oxygen. I wasn’t exhaling completely. There was a video I found of a guy on YouTube with the same issue I had and he mentioned the shallow breathing method (I can look for this later if anyone is interested).
Now when I walk up the stairs, I allow my exhale to fully be exhaled, and what I realized is that you don’t have to take in a breath right away, a few exertions can happen before your body naturally feels the need to inhale back in. Idk if what I’m trying to explain makes sense, but essentially my breathing became abnormal overtime through overthinking it, and my panic was causing me to try and inhale as much as I could thinking I wasn’t getting enough air.
I know this is becoming a very long-winded post, but I just wanted to get it out there to someone that you can start to overcome this! I tested it again today with working out, I felt my blood pumping but only felt a little bit of panic for about a second out of a half our workout whereas before I was panicking throughout about 90% of the workout.
Now I am finally looking forward to being able to travel and do things I haven’t been able to these past 4 years! That may sound weird but I would always anticipate that I would have to exclude some place if there were too many steps or any inclinations. I didn’t even feel human. This was particular embarrassing if I was with a group of people as I’d always had to try and play it off like it was something else.
The anxiety is not completely gone yet, I still can feel a minor sense of panic at times, but I am no longer avoiding inclinations like I used to and am looking forward to getting my life back now.
TL; DR : after suffering with air hunger for the past 4 years, I finally came to found it was in my abnormal breathing. I had to learn to start breathing more shallow instead of taking deep breaths.

Went in for pain in the back of neck down the back and in the front a bit (at heart). They did bloods, CT, EKG, said all looks okay! The EKG just got uploaded to MyChart. What is all this?? 47yo, overweight, sedentary, history of lifelong smoking, diabetic, severe stage 3 Hidradenitis Supurritiva (which has deemed me almost immobile). Triponin was 8 then 9. BP is always a little high, 150/90 typical. I’m sitting here on a Saturday night Googling my a$$ off. Ughhh. Any ideas?
Impression Sinus tachycardia Probable left atrial enlargement Borderline ST depression, anterolateral leads ST elevation, consider inferior injury Abnormal ECG
Diagnosis: Sinus tachycardia Probable left atrial enlargement Borderline ST depression, anterolateral leads ST elevation, consider inferior injury Abnormal ECG