Bursa contusion

Shoulder MRI results

2024.04.26 17:08 musical_spork Shoulder MRI results

39, female 8+yrs of shoulder pain, arm & hand weakness, & numbness & pain.
Can someone dumb down these MRI results for me? If I'm reading it right I have at least one year in my rotator cuff and a torn labrum? I'm not sure what the #2 under impressions is. The mass effect thing.
FINDINGS:
Rotator cuff: Moderate supraspinatus tendinopathy more pronounced anteriorly where there is some thinning of the anterior supraspinatus secondary to bursal sided fraying. Mild tendinopathy anterior infraspinatus. Teres minor is intact. Insertional tendinopathy of the subscapularis moderately with low grade partial thickness tearing series 401 image 11. Rotator cuff musculature signal and bulk are intact.
Subacromial/subdeltoid bursa: Trace bursal fluid suggesting bursitis.
Long head biceps tendon: No evidence of significant tear or tendinopathy. No dislocation or subluxation.
AC joint: Moderate arthritic change. There is type 1 acromion. There is mass effect on the superior margin of the supraspinatus suggesting external impingement. No separation. Coracoclavicular and coracoacromial ligaments appear intact.
Bone marrow: No bone contusion or fracture.
Labrum: There is a small tear involving the posterior labrum mid to upper aspect. Remainder intact.
Cartilage: No focal chondral defects are detected.
Joint space: No significant joint effusion.
Soft tissues: No masses or cysts in the suprascapular notch, spinoglenoid notch, or quadrilateral space.
IMPRESSION: 1. Supraspinatus and subscapularis tendinopathy as above. Bursal sided supraspinatus tendinopathy and low grade insertional partial thickness tearing at the subscapularis. 2. External impingement with mass effect on the supraspinatus associated with AC joint arthritis. 3. Suspect small tear posterior labrum.
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2024.03.24 01:54 scorpiofiredragon76 Would appreciate someone please explaining the severity of my right shoulder MRI results, thank you!

Hi everyone, I’d really appreciate someone helping explain my MRI results. I've been in a lot of pain for a long time and am worried. My rheumatologist (I have psoriatic arthritis in my wrist) ordered the MRI after I let him know about the shoulder pain.
I waited 3 mos for the MRI insurance approval and MRI appt to be available. After the MRI, my rheumatologist referred me to a specialist (an MD in Orthopedic Surgery dept) and after waiting several weeks, that appt is next Wed. I also called the hospital's PT/Rehabilitation dept and have an initial appt in 3 weeks.
My rheumatologist, who's not a shoulder expert, told me the following (my notes from our call): "good news is that there is no swelling, not seeing frozen shoulder, that’s a longer recovery. The inflammatory arthritis is not that bad. We have these 2 other things to deal with (rotator cuff and labrum). Physical therapy is one resource for the rotator cuff, an orthopedic shoulder specialist should look at the labrum to see if they recommend a procedure. Tears do heal, some need surgical intervention. Psoriatic arthritis can predispose you to this."
MRI test results pasted below. Thanks for any thoughts or advice you can provide:
MR SHOULDER WO CONTRAST RIGHT
TECHNIQUE: Coronal fat-saturated T2W, sagittal T1W and fat-saturated T2W and axial fat-saturated PD images of the shoulder were obtained.
FINDINGS:
Bones: There is no acute fracture, bone contusion, avascular necrosis, or suspicious marrow lesion.
Acromioclavicular joint: The joint is normally aligned without significant degenerative changes, separation, or ligamentous injury.
Rotator cuff: There is a low-grade articular surface partial thickness tear of the critical zone of the infraspinatus tendon measuring about 1.5 x 1.0 cm. The supraspinatus and subscapularis tendons are unremarkable. Muscles: The cuff muscles are normal in signal and bulk.
Long head of the biceps tendon: The long head of the biceps tendon is intact.
Glenohumeral joint: The glenohumeral joint is normal in alignment. There is no glenohumeral joint effusion or synovitis.
  • Labrum: There is chronic appearing tearing of the anterior and anterior superior labrum. No discrete paralabral cyst.
  • Cartilage: No discrete high-grade or measurable chondral defect.
Subacromial/subdeltoid bursa: No bursal fluid.
Other: No axillary lymphadenopathy. There are no space-occupying lesions within the quadrilateral space.
IMPRESSION:
  1. Low-grade partial-thickness tear of the infraspinatus tendon.
  2. Chronic tearing of the anterior and anterior superior labrum.
  3. No joint effusion or synovitis.
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2024.03.21 05:04 scorpiofiredragon76 Would appreciate someone please explaining the severity of my right shoulder MRI results, thank you!

Hi everyone, I’d really appreciate someone helping explain my MRI results. I've been in a lot of pain for a long time and am worried. My rheumatologist (I have psoriatic arthritis in my wrist) ordered the MRI after I let him know about the shoulder pain.
I waited 3 mos for the MRI insurance approval and MRI appt to be available. After the MRI, my rheumatologist referred me to a specialist (an MD in Orthopedic Surgery dept) and after waiting several weeks, that appt is next Wed. I also called the hospital's PT/Rehabilitation dept and have an initial appt in 3 weeks.
My rheumatologist, who's not a shoulder expert, told me the following (my notes from our call): "good news is that there is no swelling, not seeing frozen shoulder, that’s a longer recovery. The inflammatory arthritis is not that bad. We have these 2 other things to deal with (rotator cuff and labrum). Physical therapy is one resource for the rotator cuff, an orthopedic shoulder specialist should look at the labrum to see if they recommend a procedure. Tears do heal, some need surgical intervention. Psoriatic arthritis can predispose you to this."
MRI test results pasted below. Thanks for any thoughts or advice you can provide.
---------------------------------
MRI SHOULDER RIGHT WO CONTRAST
Narrative & Impression
MR SHOULDER WO CONTRAST RIGHT
TECHNIQUE: Coronal fat-saturated T2W, sagittal T1W and fat-saturated T2W and axial fat-saturated PD images of the shoulder were obtained.
FINDINGS:
Bones: There is no acute fracture, bone contusion, avascular necrosis, or suspicious marrow lesion.
Acromioclavicular joint: The joint is normally aligned without significant degenerative changes, separation, or ligamentous injury.
Rotator cuff: There is a low-grade articular surface partial thickness tear of the critical zone of the infraspinatus tendon measuring about 1.5 x 1.0 cm. The supraspinatus and subscapularis tendons are unremarkable.Muscles: The cuff muscles are normal in signal and bulk.
Long head of the biceps tendon: The long head of the biceps tendon is intact.
Glenohumeral joint: The glenohumeral joint is normal in alignment. There is no glenohumeral joint effusion or synovitis.
- Labrum: There is chronic appearing tearing of the anterior and anterior superior labrum. No discrete paralabral cyst.
- Cartilage: No discrete high-grade or measurable chondral defect.
Subacromial/subdeltoid bursa: No bursal fluid.
Other: No axillary lymphadenopathy. There are no space-occupying lesions within the quadrilateral space.
IMPRESSION:
  1. Low-grade partial-thickness tear of the infraspinatus tendon.
  2. Chronic tearing of the anterior and anterior superior labrum.
  3. No joint effusion or synovitis.



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2024.02.16 04:01 Agitated-Air8874 MRI Shoulder w/o Contrast Right

Hello, any help to better understand what's ahead of me? Yet to meet the orthopedic
FINDINGS: ROTATOR CUFF: Supraspinatus tendon: Mild tendinosis with tiny partial width partial-thickness tear of the supraspinatus tendon at the footprint (series 7 image 10) Infraspinatus tendon: Mild tendinosis with a high-grade tear.
GLENOHUMERAL JOINT: Labrum: No labral tear or detachment. No paralabral cyst. Bone and cartilage: Marrow edema is present at the greater tuberosity without definite underlying fracture. No significant chondral abnormality.
BURSA: Mild subacromial subdeltoid bursitis.
IMPRESSION: 1. Mild rotator cuff tendinosis without high-grade tear. Tiny low-grade tear of the supraspinatus tendon at the footprint. 2. Mild subacromial subdeltoid bursitis. 3. Marrow edema is present at the greater tuberosity without definite underlying fracture. Nonspecific but may be bony contusion given history of prior trauma.
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2024.02.01 03:28 IrishNova32 This is the results of my MRI of my knee. Is this kind of meniscus tear repairable without surgery? I'm seeing a doctor in the next day or two

STUDY PERFORMED: MRI of the Left Knee without contrast
TECHNIQUE: Magnetic resonance imaging was performed. Sequences acquired include axial T2, sagittal proton density and fat suppressed T2 , as well as coronal T1 and fat suppressed T2 sequences CLINICAL INFORMATION: Pain in left knee.
COMPARISON: None
FINDINGS:
Medial Compartment: Horizontal undersurface tear of posterior horn of the medial meniscus which communicates with para meniscal cyst along the posterior medial joint capsule. Para meniscal cyst measures 1.9 x 1.9 x 0.5 cm. Lateral Compartment: The lateral meniscus is intact. The lateral collateral ligament is intact. There is no significant joint surface abnormality. Intercondvlar Space_and_Cruciate_Ligaments: The cruciate ligaments are intact
Extensor_Mechanism:
The quadriceps and patellar tendons are intact. Lateral patellar tilt. Mild thinning of articular cartilage medial patellar facet. Linear full-thickness articular cartilage defect median patellar ridge which measures 1.7 mm transversely. Nonspecific prepatellar edema The TT-TG distance is 12 mm Patella alta. Insall Salvati ratio is 1.2 cm. Small amount of fluid in deep infrapatella bursa Bone: There is no fracture, contusion pattern or osseous lesion. Svnovium_and_Other Soft Tissues: There is no significant joint effusion or popliteal cyst formation Muscle signal is normal.
IMPRESSION:
Horizontal undersurface tear posterior horn of the medial meniscus which communicates with a para meniscal cyst about the posterior medial joint capsule. Mild chondromalacia patella. Full-thickness fissure within articular cartilage median patellar ridge.
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2024.01.19 13:20 jaymavs Any recommendations on how best I can recover from this? Happened due to a ski accident.

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2023.11.20 07:01 my58vw MRI “probable” Labral tear… help with results

Looking for a bit on understanding on MRI results. Injury occurred due to a fall on outstretched arm after stopping an assault on an individual at work, dislocation, with two repeat dislocations in the following few days.
MRI results
The current exam is mildly limited secondary to patient motion and patient's large body habitus.
There is a moderate to large Hill-Sachs deformity at the posterior lateral aspect of the humeral head with associated moderate patchy subjacent marrow edema. There is probable associated tear at the anterior inferior glenoid labrum with mild extension into the posterior inferior quadrant not excluded. Correlate clinically and if indicated, a dedicated MR arthrogram would provide more detailed evaluation.
There is mild tendinopathy versus rotator cuff contusion involving the supraspinatus and subscapularis tendons without a significant discrete rotator cuff tear identified. The infraspinatus and teres minor tendons appear intact.
The long head of the biceps tendon and the biceps labral anchor are intact. The biceps tendon is situated within the bicipital groove. There is a small-to-moderate glenohumeral joint effusion with probable mild debris and/or synovitis. No discrete intra-articular bodies are seen. There is also mild fluid within the subacromial/subdeltoid bursa, possibly bursitis.
Does probable mean “likely but we can’t know 100% the severity? The MRI seems to indiciare a Bankart lesion…
I lost significant ROM in the 5 weeks since the injury, could not life my arm straight in front of my body. The initial evaluator who looked at the MRI was a complete a$$ and would not give me any information, other than to criticize my ROM, and ordering PT. The doctor asked the office to refer me to the “doctor that can do the surgery.”
I am working hard to get ROM back, and have seen significant progress in the last week.
On a side note… I got trapped in the MRI machine (too large shoulders, BMI, and had to be pulled out of the machine… all with my shoulder in some major intense pain…
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2023.11.06 17:10 Equal_Ad8255 Recovering fast without surgery

I injured my knee around 18 days ago 1st photo MRi was next day after injury And 2 nd Mri was taken 14 days after injury At first it was very hard for me to walk around There was so much swelling . I could not bend my knee all the way . Then I applied ice twice a day for 12 days After seeing my 2nd MRi report my doctor said that I don't recommend surgery , you'll do fine without it . He checked himself by siting on leg and pulling toward himself , my leg didn't go up so he said looks like your ACL is not completely teared After 18 days I can walk normally with zero percent pain . I can jog a little, squat a little but still there is just one problem I can't put my heel on ground for more than 3,4 second But at early days I couldn't put weight on my heel for 0.5 second Then it improved from 0.5 to 3,4 second I hope I don't go under surgery and recover from just excercise
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2023.10.15 18:07 Equal_Ad8255 How bad is it look is surgery necessary please help 🙏

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2023.10.15 08:49 Equal_Ad8255 Can't it be treated without surgery

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2023.08.22 18:28 Overall-Savings-1424 Can my shoulder dislocation be treated without a surgery? Added the points from my MRI scan report. I have consulted a few doctors, most of them advised for a surgery and one of them told dislocation can happen even after the surgery, so I have to start with PT and learn to live with it.

30
Male
168 cm
80 kg
Indian
2 weeks India
Shoulder dislocation
Physiotherapy exercises
MRI report: Focal depression in the anteromedial aspect of humeral head with underlying edema / contusion
Posterior labrocapsular periosteal sleeve avulsion
Moderate to severe glenohumeral join effusion with fluid in subcoracoid and subscapularis bursae and biecep long head tendon sheath
Edema / contusion in teres minor and infraspinatus muscles
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2023.05.31 18:50 Sea_Instance3391 How bad is this?

Feeling incredibly stressed about my knee at the moment. However, I’m not experiencing any pain when walking and have a relatively large range of motion in it. Injured it playing sport 2/3 weeks ago. It just buckled underneath me while turning. Thanks in advance.
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2023.05.19 01:40 Atomic-Kitty Help Understanding MRI Results Please

I received access to the report for my left knee MRI today but my followup appointment isn’t until 5/25. Can someone give me a brief synopsis of the findings? It’s full of really big words that sound like surgery in my future.
FINDINGS: Menisci: Complex medial meniscal tear with a high-grade tear involving the posterior root complex tearing extending to the posterior horn with extruded medial meniscal body.. There is no high-grade lateral meniscal tear.
Cruciate Ligaments: Normal anterior and posterior cruciate ligaments.
Collateral Ligaments: Normal lateral collateral ligaments. Mild reactive edema along the MCL without tear.
Tendons: Normal.
Articular Cartilage/Synovium: Small joint effusion. Increased signal within the prepatellar bursa. - Medial: Full-thickness cartilage defect measuring up to 2 cm within the medial femoral condyle. No associated marrow edema. Additional full-thickness cartilage defect within the periphery of the medial tibial plateau with mild osseous edema associated. - Lateral: Multifocal cartilage thinning without focal high- grade defect. - Patellofemoral: Multifocal high-grade cartilage loss with focal cartilage fissuring along the lateral patellar facet and with associated edema (series 3, image 12). Central trochlear multifocal cartilage fissuring
Bones: Osseous contusions as above, otherwise no suspicious marrow signal.
Muscles: Normal.
Miscellaneous: No popliteal cyst or other soft tissue mass.
IMPRESSION: 1. Complex medial meniscal tear with a high-grade tear of the posterior root and significant extrusion of the meniscal body. 2. Multifocal, high-grade cartilage loss with full-thickness defects in the patellofemoral and medial compartments as detailed above.
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2023.03.18 03:38 waffleear Should I get this repaired?

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2022.12.13 13:17 TemporaryBee7314 What Does a Sprained Wrist Feel Like?

What Does a Sprained Wrist Feel Like?
Do you ever get a "sprained wrist" that will leave you asking, "What does a Sprained Wrist Feel Like?" I do, often. Just recently my wrist got twisted (kind of funny coming from a chiropractor), and now I can barely type, let alone walk. In this article I'm going to explain what a Sprained Wrist feels like, and how you can get one today without waiting over the weekend to see a doctor. Buy Twitter Accounts. When you buy Yahoo Accounts at Medical Discount Services you're signing up for an entire month of what does a Sprained Wrist Feel Like benefits.
Sprained wrists happen a lot. One common injury is tennis elbow. Tennis Elbow causes similar symptoms to those of a sprained ankle. The wrist band or wrist brace tightens, similar to when you get a contusion. The pain is usually worse in the morning, but better by the afternoon. The wrist pain usually goes away after two days, but you might need to take an aspirin.
Another injury is tennis elbow. This usually happens on the non-dominant hand. Usually the forearm pain comes first, and then the elbow pain follows. My forearms are still hurting a few days after the initial treatment. The same goes for the other hand.
Tennis Elbow usually affects your swing, causing your wrists to lock, your grip to change, or you may drop a racquet or hit a ball with your non-dominant hand. The pain usually first manifests in the morning, or sometimes in the early afternoon. It's worse when you first hit the courts or when you first get back to playing. After about a week the pain subsides, but it can come back. If you play sports like tennis a lot, this can be a big problem, since you spend a lot of time on the courts. You may also injure your wrist while playing tennis, which will lead to chronic wrist pain. Buy Google Voice Accounts.

https://preview.redd.it/gdb3khloqn5a1.jpg?width=1200&format=pjpg&auto=webp&s=320f32986a5e23dcd4398fa630b42464b3cc4e2e
One of the worst pains I've ever experienced is from carpal tunnel syndrome. This syndrome is caused by compression of the median nerve, which is in your wrist. You will experience the pain in your thumbs, index finger, middle and even ring fingers. It can be a very sharp pain that shoots through your arm, and sometimes it feels like your hand is going to burn. It can last for weeks or months. Buy Edu Emails.
A torn rotator cuff is another injury that can cause what does a Sprained wrist feel like. Your rotator cuff is made up of four muscles. When it is injured, it can be very painful. Usually it happens when you lift your arm overhead, or if you throw a ball with your injured hand. You might also get pain while running or doing some other activity that requires you to use your shoulder as much as possible.
Sometimes what does a Sprained wrist feel like happens when you accidentally twist your wrist. When this happens you can feel a sudden sharp pain that shoots through your arm. When you twist your arm, your wrist gets bent backwards and can put a lot of pressure on the tendons that are attached to your forearm bones. When you injure your wrist in this way, you need to rest it immediately and get plenty of ice to reduce swelling.
Another injury that can cause what does a Sprained wrist feel like is if you accidentally bump your elbow into something. This can really crack the delicate tendons and bursa (small sacs of fluid) that cover the tendons. When a bursa is cracked, it causes a lot of friction and inflammation between tissues, tendons, bones and muscles. The more inflammation there is, the more pain and discomfort you will feel. Some people may actually experience enough pain that they will need to visit the doctor to get some treatment for what does a Sprained wrist feel like. pvabuynow.blogspot.com Remember that if your injury is severe enough to restrict your motion or your ability to feel any pain, then you really need to go to the doctor and get the proper treatment for your injury.
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2022.09.19 11:06 lukafromchina How to correctly view "mycotoxins" in animal husbandry industry

How to correctly view
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With the advent of winter, the use of mold removers is becoming more and more popular, but how to use mold removers, how to use them reasonably, and how much should be used, this should be the most concerned issue as a farmer, but in the market of mold removers In the past, too much profit-seeking sales will not only make people with vague concepts misinterpret the effect of mold release agents, but also cause adverse effects on the health of pigs in the case of unreasonable use.
1.What are mycotoxins?
Mycotoxins are naturally produced by molds. Molds lead to local decay and deterioration during the harvesting and storage of crops. In this process, the toxins produced are called mycotoxins. The toxins produced produce large numbers of propagules (spores) that enter the air and cause diseased infections in the body as the animal feeds and breathes. For example, animal death caused by typical acute flatulence disease also exists because of feed mycotoxin poisoning. Animals are also smart. In the case of mildew in the feed, its feed intake will drop significantly, which is a state of self-protection. When such clinical manifestations occur, it is necessary to check whether the feed is mildewed and so on.
  1. What are the hazards of mycotoxins to animals?
The growth of various mycotoxins has different effects on livestock.
Effects of aflatoxin on livestock: growth retardation, decreased feed rate, jaundice, rough coat, hypoalbuminemia, depression, anorexia, acute liver disease, liver cancer, immunosuppression; effects on poultry: bursa and thymus Atrophy, subcutaneous hemorrhage, poor immune response, decreased antibody power, vaccine failure, increased susceptibility to disease, smaller eggs, decreased yolk weight, decreased fertilization rate, hatching rate, and increased embryonic death.
Effects of ochratoxin on livestock: attack on kidneys, immune system and hematopoietic system, liver becomes fragile, mild renal disease, decreased weight gain, severe thirst, growth retardation, azotemia, polyuria, diarrhea, diabetes; Effects on poultry: inhibition of kidney, immune and hematopoietic system, incomplete absorption of calcium and phosphorus, fragile bones, incomplete eggshell calcification, high egg breaking rate, subcutaneous hemorrhage, and easy contusion.
Effects of DON on livestock: damage to the intestines, bone marrow, spleen, reduced feed intake, easy secondary infection by bacteria, vomiting, and refusal to feed; effects on poultry: damage to the digestive tract, glandular stomach and intestinal lesions , decreased feed intake, refusal to feed, and reduced egg production.
Effects of T-2 toxin on livestock: damage to the digestive tract, oral cavity, stomach and intestinal tract, reduced feed intake, irritated oral cavity and skin lesions, food refusal, vomiting, neurological disorders, immunosuppression; effects on poultry : Decreased egg production rate, poor feather growth, oral ulcers, decreased feed intake, food refusal, neurological disorders, suppressed immunity.
Effects of citrinin on livestock: damage to the kidneys, resulting in renal lesions, decreased feed intake, polyuria, soft feces, diarrhea; effects on poultry: inhibition of kidneys, increased urinary excretion, soft feces, diarrhea.
Effects of F-2 toxin on livestock: estrogen hyperactivity, irregular or no estrus, false estrus in gilts, pus before mating, sow labia, uterus enlargement, reduced conception rate, vaginitis, abortion, Stillbirth, valgus legs, red and swollen labia, prolapse of the anus, prolapse of the uterus, and decreased semen quality of boars; effects on poultry: ovarian atrophy, decreased egg production rate, and decreased egg fertilization rate.
Clinical observation: sick pigs generally show normal body temperature or decreased body temperature. Acute poisoning is manifested as a large number of pigs suffering from disease, loss of appetite or even extinction. The pigs died abnormally. The dead pigs generally showed swelling of the stomach, stiff limbs, purple skin and so on. Male sows and growing pigs are seriously ill, die within a few days, and have a high mortality rate. Pregnant sows have a large number of abortions and stillbirths, and boars have dead sperm and no sperm. Chronic poisoning is mainly manifested as abnormal estrus, false estrus, return to estrus, increased proportion of stillbirths and weak litters, unsatisfactory parturition data of breeding pigs; significantly reduced litter size and reduced milk production of sows. Herds have reduced feed intake and slow growth. The vulva of the newborn piglets is red, swollen and weak, and the hind legs are turned outwards in the shape of "splayed feet". The vulva of the growing pig is red and swollen, prolapse of the anus, ataxia, yellow-stained skin, and rough coat. Loss of libido, mammary gland enlargement, foreskin edema, and testicular atrophy in boars. Erythema appears on the skin of the mouth, ears, medial and ventral sides of the extremities, and in severe cases the skin is ulcerated and crusted. Individual pigs vomited significantly. Commercial pigs will have various immunosuppressive diseases, such as blue ear, ring, parapig, etc. The occurrence of such diseases is often caused by mycotoxins. At the same time, it is easy to see the symptoms of vomiting, rectal prolapse, and vulvar swelling in clinical practice. The overall health of the pig herd is in a sub-healthy state, the diarrhea rate of suckling piglets is high, and the antibody test after the vaccine is not ideal, etc., it is necessary to consider whether mycotoxins are at fault. The immune function of the body is decreased, and secondary infection is obvious.
  1. How to minimize the impact of mycotoxins on the body, and how to "treat the disease before it is cured"?
  2. Selection of raw materials
The origin of high-quality feed raw materials determines the main factor for grain management, which is also the quality requirement of feed processing enterprises for raw material suppliers. Procurement teams of large companies will also make procurement plans based on the degree of mildew of mycotoxins. At the same time, in the process of planting feed raw materials, the quality of the harvest will be reduced due to "force majeure" factors, such as typhoons and floods. This situation is unavoidable, so how to deal with the harmful effects of mycotoxins plays a key role in the requirements of a feed enterprise and the choice of origin.
  1. Processing of corn
If the corn is not dehydrated in time after the harvest, it will also cause the growth of mycotoxins. The dehydration of corn is generally based on drying and drying. To do cleaning, there are sieving, washing and other operation methods. Generally, the processing moisture of feed corn is between 14-15 moisture, and corn exceeding this moisture standard is not conducive to storage, transportation or processing.
  1. Detailed management of feeding
In the process of feeding, the management is not in place, such as: rushing the pen to wet the feed, the free feeding trough is not cleaned and fed, and the wet feed is not eaten and shoveled away, especially the sow feed trough is not clean. cleaning residue. The reduction of the feed-to-meat ratio can actually be improved through reasonable management. For example, the feed that falls on the ground in slatted farming can be collected and fed to poultry. Before feeding, develop the habit of smelling the odor of the feed and observe the color of the feed. At the same time, in the wet and rainy season, add a mildew remover to the feed to prevent and control.
  1. Management of warehousing environment
Unreasonable placement, such as: the feed is placed directly on the ground without a partition in the middle. The warehouse is leaking, the feed is approaching its expiration date, and so on. Such situations must be solved, because this can avoid the production of storage toxins (aflatoxin, ochratoxin); after the feed is bought back, in addition to reasonable storage arrangements, it must also be combined with the actual situation of the pig herd. Do a good job in the prevention and control of mycotoxins.
  1. The frequency of feeding corn is best controlled within 2 weeks, not more than one month;
  2. Add a really effective mold remover in the process of raising pigs. The principle of mold release agent, one is clay adsorbent, which uses the ionic polarity formed by the tetrahedral interlayer porous structure and the surface to strongly adsorb mycotoxins that also have ionic polarity. The strong adsorption force comes from the large surface area and Electrostatic adsorption; the second is yeast cell wall extract: using the chemical structure of glucomannan in the yeast cell wall and the affinity of mycotoxins that also belong to the organic category.
  3. How to reasonably degrade the effects of mycotoxins on animals?
  4. Application of feed mildew inhibitor
The use of antifungal agents is mainly used in grain storage, because the starch resistance of corn needs to be transformed for a period of time, and the content of resistant starch is highest in the new harvest period, which in turn reduces the digestion and absorption rate. Therefore, feed processing enterprises generally choose natural dry corn to process into piglet feed. For the storage of natural dry corn, the most important point is the prevention and control of mycotoxins, and the common method is to add the application of antifungal agents. The main activity of the antifungal agent is mainly the organic acid in the non-dissociated state. Antifungal agents include propionic acid and its salts, fumaric acid and its salts (mainly dimethyl fumarate), sodium diacetate (sDA) and other organic acids such as formic acid, propionic acid, lactic acid, sorbic acid , citric acid, benzoic acid and their salts [00]. The fungicides currently on the market are mainly mixtures of the first three or more.
Proper use of antifungal agents can prevent mildew in the feed, but when used improperly, it will not only fail to prevent mold, but also damage the quality of the feed, and even cause harm to the health of livestock, poultry and people. Be sure to pay attention to the following issues when using:
(1) Antifungal agent can only inhibit the growth of mold but cannot eliminate the mold that has been produced, so it is best to add antifungal agent when the feed and raw materials are not moldy. Feed manufacturers generally add antifungal agent to the feed to do a good job. To ensure the quality of the shelf life, adding a mildew remover (Mumeiqing or Chumoubao) is a preventive measure based on the actual situation to find the existing mildew;
(2) Pay attention to safety, add in strict accordance with the recommended amount, and add antifungal agents based on the principles of low toxicity, low cost and good effect;
(3) Each antifungal agent has its own suitable antifungal range, and the mixed use of multiple antifungal agents is more effective than using a single antifungal agent;
(4) If one antifungal agent is used for a long time, the bacteria will develop drug resistance, so a variety of antifungal agents should be used in rotation;
(5) Most antifungal agents are acidic substances and cannot be mixed with alkaline substances, otherwise the efficacy of antifungal agents will be reduced;
(6) Strengthen management to prevent mildew of feed. Mix well with feed when using mold inhibitor.
There are other measures to prevent mold in market feed:
(1) Control the water content of the feed and keep the environment dry. When the moisture content of cereal feed is 17%-18%, it is the most suitable condition for mold growth and reproduction. Therefore, the crops should be dried quickly after harvesting, and the drying must be uniform and uniform. The moisture content of corn, sorghum and rice should be controlled below 14%, the moisture content of soybeans and their meal, wheat and bran should be controlled below 13%, rapeseed meal, cottonseed meal and peanut kernel meal should be controlled under 13%. , fish meal, meat meal and meat and bone meal water content should be controlled below 12%.
⑵ physical mildew method. Mainly include temperature control of storage environment, closed oxygen barrier storage, controlled atmosphere storage, low temperature ventilation storage and radiation method.
Application of mold remover
At present, the mature mold release agents used in the market are mainly mineral adsorbents and yeast cell wall extracts (mannose oligosaccharide and glucan).
  1. Mineral adsorbents: mainly hydrated aluminosilicate, montmorillonite, bentonite, among which hydrated aluminosilicate has been confirmed by the US FDA as a mold release agent in food and feed, and these products are of good quality The key to the bad is the purity of the product. The lower the purity, the worse the adsorption selectivity, which will affect the content of vitamins, amino acids and trace elements. It has a strong adsorption effect on polar aflatoxin. The disadvantage is that the dosage is large. According to relevant data reports, 3kg should be added per ton of feed. At the same time, it also has an adsorption effect on polar vitamins and amino acids. When adsorbing aflatoxin, it also takes nutrients out of the body.
  2. Yeast cell wall: mainly mannose oligosaccharide, which adsorbs part of mycotoxins through electrochemical action, but its adsorption effect is significantly lower than that of mineral products. In vivo experiments, the symptoms of animal mycotoxins cannot be relieved alone. The disadvantage is that it cannot adsorb non-polar zearalenone, falomycin and vomitomycin, which are the most harmful to sows. For various toxins that have been absorbed into the circulatory system by the intestinal mucosa, it cannot fundamentally solve the mycotoxin poisoning of the main feed. Therefore, the effect is not good in the case of clinical reactions to mycotoxins.
3 Simple determination of the quality of mineral adsorbents:
  1. The fineness of high-purity montmorillonite is more than 300 mesh, and the production cost is relatively high. Ordinary bentonite and zeolite powder are generally only within 100 mesh, and the cost is low. Therefore, when you touch it by hand, you can feel that ordinary bentonite and zeolite powder are rough, while high-purity montmorillonite feels fine and smooth.
Second, montmorillonite has strong water absorption, put your hand in it, the moisture on the skin will be absorbed instantly, and then the hand will be exposed to the air, there will be obvious burning sensation. Bentonite and zeolite powder do not have such characteristics.
Regarding the application of mold remover, it can be concluded that for the routine prevention of mycotoxins, yeast cell walls can be selected; for mold poisoning treatment, minerals can be selected, and it is best to use minerals during seasonal humidity. Classes are more practical.
  1. Application of detoxification products
Decomposing molds: The active ingredients are active enzymes such as aflatoxin decomposing molds, which can decompose some mycotoxins in a targeted manner. However, due to their strong requirements on temperature and environment, their stability is not good, and only for one type of mold. Toxins are expensive, and the toxicity of the decomposed products to the body needs to be studied.
Chinese herbal medicines: mainly by improving the liver's ability to detoxify toxins to achieve the purpose of removing mycotoxins. And according to the clinical thinking of traditional Chinese veterinarians, mycotoxin poisoning is only a poisoning reaction. It is treated by combining Chinese herbal medicine according to specific clinical reactions, not just using a certain prescription to relieve or alleviate mycotoxin poisoning. treatment purpose. At the same time, there are also many studies based on the data extracted by the single prescription drug laboratory, but the clinical manifestations are different or because of the type of breeding animals, the incentives of the disease are mixed with other factors, the TCM veterinarians need to take into account the above situations. Prescribing medicines also reminds Chinese veterinarians that they are rigorous in judging diseases and increase their confidence in the efficiency of medicines. And in the long-term use of Chinese herbal medicine for health care, the detoxification reaction of the body's internal environment is also invisibly moved, thereby gradually improving the health of the pig herd. This is why foreign pig breeds are introduced in the process of breeding and cultivation The excellent genes of Chinese pig breeds. I think one of them may be the importance of the disease resistance genes of our pig breeds.
Although mycotoxins are ubiquitous and affect the development of our aquaculture industry, modern scientific research combines the unique experience of ancient medical research in my country, and then develops into the application of aquaculture industry. Then we can greatly reduce the indirect or direct economic losses caused by mycotoxins in the breeding industry, minimize the harm of mycotoxins as much as possible, and also speed up the economic development of my country's animal husbandry industry.
https://www.arshinefeed.com/
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2022.08.13 20:37 mariamine3 Meniscal Tear Dissapeared?

So here’s what happened. August 5th 2020, I was standing in front of a couch (my left leg was touching it, knee around the seat, I was facing down the length of the couch, left knee touching couch, right knee facing rest of room). My brother approached me, so I turned my right leg and body towards him (now left foot is facing forward with my hips, but chest and right foot have turned to the right to face him) and he lightly pushes me by the shoulders I assume to make me sit down onto the couch as an annoying brother does passing by. However since my left foot was planted, when I fell/sat, my right knee bended regularly as I sat but my left knee made three loud pops as it was forced to bend, not at the knee, but towards the inside of my leg where I assume the meniscus is. I couldn’t get an MRI, knee so swollen in the bent position my foot couldn’t touch the ground for a month and half and I eventually left canada to go home (diff country) where I got an MRI and spoke with heads of orthopaedic deps, as well as head of physiotherapy at one of the biggest hospitals. They essentially said, ur 20, you don’t have any major sports to play nor movement to do since everything’s online so let’s PT and build ur strength. By Jan I was bending down to get stuff out of the fridge. By April ‘21 I was doing burpees (carefully). Came back to canada and around Nov/Dec my knee started hurting again. It progressively got worse despite my keeping it warm and Pt. Now it hurts when I sit cross legged or stand or bend down. I can but it’s tender and very noticeable plus I even get pain when I press lightly on the inner side of my left knee. So I met my fam doc and for scheduled for an MRI and lo and behold my doc calls me and says there’s absolutely no injury? Is this possible? They themselves said if it was a mensical tear it’s not possible to heal like that but the MRI shows absolutely perfect ligaments. They weren’t there in the other country obviously so don’t know my original condition but it was traumatizing. Also, my first mri was an hour long but the second was fifteen minutes. Barely. I attached my original report, I don’t have details but this part I screenshot Ed so it was in iCloud and I’m copying it’s text. I’m in pain and my doctor isn’t helping Al all. Any advice?
THE ORIGINAL MRI IMPRESSION:
IMPRESSION: Lateralization of patella with increased patellar inclination angle, focal areas of cartilage loss with bone edema/contusion in medial articular facet of patella and anterolateral aspect of lateral femoral condyle, slightly thickened medial patellar retinaculum with intra-substance increased T2W/STIR signal and peri-ligamentous fluid……Injury pattern is concerning for lateral patellar translation/ dislocation with kissing contusions involving patella and femoral condyle. Details in body of report Joint effusion is seen with extension into supra patellar bursa. Edema is seen in pre patellar and superficial infrapatellar bursa. Edema is also seen in Hoffa's fat pad. Fluid is seen around medial collateral ligament concerning for sprain as attachment sites are intact. Grade 2 tear posterior horn of medial meniscus Please correlate clinically.
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2022.07.17 19:33 engacad Can someone knowledgeable tell me if shoulder-joint issue can cause pain along medial-border of scapula?

41m.
I've had diffuse/burning/sharp pain along the top-part of the shoulder (near supraspinatus muscle area) and the medial-border of scapula (which is the border opposite from the shoulder side of scapula).
Doctor ordered an MRI of shoulder and it shows a few issues in the shoulder joint a) humeral head subchondral bone marrow edema b) superior labrum degeneration c) supraspinatus tendinopathy d) inferior glenoid osteophyte.
Can the shoulder-joint issue cause pain along the medial-border (not lateral) of the scapula? What issue in shoulder-joint can translate to pain on the opposite border of the scapular bone?
It's confusing as it's been >2 years since the issue began, and doctors initially said that my pain was due to left C5-6 moderate foraminal stenosis that showed in Cervical mri. But when I went to surgeon for opinion on surgical procedure for C-spine, he said the my pain along medial-border of scpaula seems unusual, and ordered left-shoulder MRI. Anyone knowledgeable in this, can this MRI explain the medial-border scapular pain?
Impressions **
Normal bulk and signal intensity of the rotator cuff muscles. No mass lesion along the expected course of the suprascapular nerve.
Mild supraspinatus tendinopathy with low-grade fraying. No high-grade partial or full-thickness rotator cuff tear.
Subchondral bone marrow edema of the humeral head articular surface. Findings are most likely consistent with degenerative bone marrow edema, although other differential considerations include bone contusion from an acute injury or stress reaction from chronic repetitive microtrauma.
** FINDINGS **:
A-C JOINT AND CORACOACROMIAL ARCH: No significant degenerative changes of the acromioclavicular joint. Coracoacromial and acromioclavicular ligaments are intact. No os acromiale.
ROTATOR CUFF: Mild supraspinatus tendinopathy with low-grade bursal surface fraying. Infraspinatus, subscapularis, and teres minor tendons are intact. No significant fatty replacement or atrophy of the rotator cuff muscles.
BICEPS TENDON AND ANCHOR: Intact.
LABRUM: Degeneration of the superior labrum without evidence of a displaced labral tear or paralabral cyst.
FLUID/OTHER: No significant fluid within the subacromial/subdeltoid bursa. No significant joint effusion.
BONES AND CARTILAGE: Subchondral bone marrow edema of the humeral head articular surface with probable overlying chondral thinning, although evaluation is limited on larger than normal field of view images. Small marginal osteophytes of the inferior glenoid.
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2022.07.09 04:13 engacad Can issue in shoulder joint in this report explain upper-back pain between spine-scapula

I had a cervical MRI showing C5-6 left-foraminal stenosis of "moderate" level.
But my pain was mainly in the shoulder and the border of the scapula near the spine (not so much pain in the arm).
So the surgeon asked for a scapulashoulder MRI to exclude "suprascapular nerve palsy". MRI report below.
Normal bulk and signal intensity of the rotator cuff muscles. No mass lesion along the expected course of the suprascapular nerve.
Mild supraspinatus tendinopathy with low-grade fraying. No high-grade partial or full-thickness rotator cuff tear.
Subchondral bone marrow edema of the humeral head articular surface. Findings are most likely consistent with degenerative bone marrow edema, although other differential considerations include bone contusion from an acute injury or stress reaction from chronic repetitive microtrauma.
** FINDINGS **:
A-C JOINT AND CORACOACROMIAL ARCH: No significant degenerative changes of the acromioclavicular joint. Coracoacromial and acromioclavicular ligaments are intact. No os acromiale.
ROTATOR CUFF: Mild supraspinatus tendinopathy with low-grade bursal surface fraying. Infraspinatus, subscapularis, and teres minor tendons are intact. No significant fatty replacement or atrophy of the rotator cuff muscles.
BICEPS TENDON AND ANCHOR: Intact.
LABRUM: Degeneration of the superior labrum without evidence of a displaced labral tear or paralabral cyst.
FLUID/OTHER: No significant fluid within the subacromial/subdeltoid bursa. No significant joint effusion.
BONES AND CARTILAGE: Subchondral bone marrow edema of the humeral head articular surface with probable overlying chondral thinning, although evaluation is limited on larger than normal field of view images. Small marginal osteophytes of the inferior glenoid.
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2022.07.07 00:42 engacad Can this report explain pain/spasms along the medial-border of Scapula

I have a general question related to imaging report, and I'm not looking for a specific diagnosis or medical advice.
I had a cervical MRI showing C5-6 left-foraminal stenosis of "moderate" level, and had pain in the scapular region mainly. After PT for 1 year didn't help, I was referred to surgeon not for surgery but for a consult.
My pain was mainly in the shoulder and the border of the scapula near the spine (not so much pain in the arm).
So the surgeon asked for a scapulashoulder MRI to exclude "suprascapular nerve palsy". MRI report below.
Impressions **
Normal bulk and signal intensity of the rotator cuff muscles. No mass lesion along the expected course of the suprascapular nerve.
Mild supraspinatus tendinopathy with low-grade fraying. No high-grade partial or full-thickness rotator cuff tear.
Subchondral bone marrow edema of the humeral head articular surface. Findings are most likely consistent with degenerative bone marrow edema, although other differential considerations include bone contusion from an acute injury or stress reaction from chronic repetitive microtrauma.
** FINDINGS **:
A-C JOINT AND CORACOACROMIAL ARCH: No significant degenerative changes of the acromioclavicular joint. Coracoacromial and acromioclavicular ligaments are intact. No os acromiale.
ROTATOR CUFF: Mild supraspinatus tendinopathy with low-grade bursal surface fraying. Infraspinatus, subscapularis, and teres minor tendons are intact. No significant fatty replacement or atrophy of the rotator cuff muscles.
BICEPS TENDON AND ANCHOR: Intact.
LABRUM: Degeneration of the superior labrum without evidence of a displaced labral tear or paralabral cyst.
FLUID/OTHER: No significant fluid within the subacromial/subdeltoid bursa. No significant joint effusion.
BONES AND CARTILAGE: Subchondral bone marrow edema of the humeral head articular surface with probable overlying chondral thinning, although evaluation is limited on larger than normal field of view images. Small marginal osteophytes of the inferior glenoid.
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2022.07.06 18:08 engacad Can this report explain pain/spasms along the medial-border of Scapula

41M. I had a cervical MRI showing C5-6 left-foraminal stenosis of "moderate" level, and had pain in the scapular region mainly. After PT for 1 year didn't help, I was referred to surgeon not for surgery but for a consult.
My pain was mainly in the shoulder and the border of the scapula near the spine (not so much pain in the arm).
So the surgeon asked for a scapulashoulder MRI to exclude "suprascapular nerve palsy". MRI report below.
Impressions **
Normal bulk and signal intensity of the rotator cuff muscles. No mass lesion along the expected course of the suprascapular nerve.
Mild supraspinatus tendinopathy with low-grade fraying. No high-grade partial or full-thickness rotator cuff tear.
Subchondral bone marrow edema of the humeral head articular surface. Findings are most likely consistent with degenerative bone marrow edema, although other differential considerations include bone contusion from an acute injury or stress reaction from chronic repetitive microtrauma.
** FINDINGS **:
A-C JOINT AND CORACOACROMIAL ARCH: No significant degenerative changes of the acromioclavicular joint. Coracoacromial and acromioclavicular ligaments are intact. No os acromiale.
ROTATOR CUFF: Mild supraspinatus tendinopathy with low-grade bursal surface fraying. Infraspinatus, subscapularis, and teres minor tendons are intact. No significant fatty replacement or atrophy of the rotator cuff muscles.
BICEPS TENDON AND ANCHOR: Intact.
LABRUM: Degeneration of the superior labrum without evidence of a displaced labral tear or paralabral cyst.
FLUID/OTHER: No significant fluid within the subacromial/subdeltoid bursa. No significant joint effusion.
BONES AND CARTILAGE: Subchondral bone marrow edema of the humeral head articular surface with probable overlying chondral thinning, although evaluation is limited on larger than normal field of view images. Small marginal osteophytes of the inferior glenoid.
submitted by engacad to AskDocs [link] [comments]


2022.07.06 05:05 engacad Can this report explain pain/spasms along the medial-border of Scapula

I had a cervical MRI showing C5-6 left-foraminal stenosis of "moderate" level.
But my pain was mainly in the shoulder and the border of the scapula near the spine (not so much pain in the arm).
So the surgeon asked for a scapulashoulder MRI to exclude "suprascapular nerve palsy". MRI report below.
Normal bulk and signal intensity of the rotator cuff muscles. No mass lesion along the expected course of the suprascapular nerve.
Mild supraspinatus tendinopathy with low-grade fraying. No high-grade partial or full-thickness rotator cuff tear.
Subchondral bone marrow edema of the humeral head articular surface. Findings are most likely consistent with degenerative bone marrow edema, although other differential considerations include bone contusion from an acute injury or stress reaction from chronic repetitive microtrauma.
** FINDINGS **:
A-C JOINT AND CORACOACROMIAL ARCH: No significant degenerative changes of the acromioclavicular joint. Coracoacromial and acromioclavicular ligaments are intact. No os acromiale.
ROTATOR CUFF: Mild supraspinatus tendinopathy with low-grade bursal surface fraying. Infraspinatus, subscapularis, and teres minor tendons are intact. No significant fatty replacement or atrophy of the rotator cuff muscles.
BICEPS TENDON AND ANCHOR: Intact.
LABRUM: Degeneration of the superior labrum without evidence of a displaced labral tear or paralabral cyst.
FLUID/OTHER: No significant fluid within the subacromial/subdeltoid bursa. No significant joint effusion.
BONES AND CARTILAGE: Subchondral bone marrow edema of the humeral head articular surface with probable overlying chondral thinning, although evaluation is limited on larger than normal field of view images. Small marginal osteophytes of the inferior glenoid.
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