Submental gland

Lump on neck swells up and down

2024.05.15 05:57 National-World-2737 Lump on neck swells up and down

23F, occasional marijuana user, no known health issues, takes 50 mg spironolactone for off label tx of acne
So for the last several months to a year or two I have had an area on my neck that swells and goes down periodically. It is located slightly right to the midline of my neck just in the area where my chin meets my neck, maybe near the hyoid bone. I would consider this between submental and submandibular region. When the area swells I can feel my neck quite swollen in this area and that side feels slightly uncomfortable when I swallow on that side. My tonsils are still present but not visually enlarged. The lump is not always present and I cannot seem to connect a trend with it. It comes and goes. It is a hard immovable lump that is not seen visually but can be felt. I had it looked at once last fall when it swelled up by an NP at urgent care. The diagnosis was a swollen parotid gland, however this area is no where near the parotid. I had an ultrasound as well but the results were normal. The lump has returned and I just feel very uneasy about it and have tried researching other possibilities it could be but am stumped. I realize no one can give me a diagnosis, but does anyone have ideas I can look into and perhaps discuss with my doctor? Thank you
submitted by National-World-2737 to AskDocs [link] [comments]


2024.03.26 09:19 skinsecrets46 How to Get Rid of a Double Chin?

How to Get Rid of a Double Chin?

https://preview.redd.it/qdkh9ntc0nqc1.png?width=612&format=png&auto=webp&s=f3afb54f10420538cf37371f73e951dbb7416df1
A submental fat, commonly known as a double chin, can be a stubborn aesthetic concern, forming unsightly layers under the chin and impacting jaw line improvement. This condition often arises with weight gain, but can also be influenced by genetics or natural skin laxity due to aging.
If you’re looking to achieve a more sculpted and youthful appearance, various options are available to address a double chin and enhance jaw line definition. Some methods focus on reducing excess fat, while others target skin tightening and contouring.

Exercises that target a double chin

There’s no scientific proof or data yet that says chin exercises are worthwhile in removing the double chin, however, there is enough anecdotal evidence to get you started.
Here are six workout activities that can boost and tone the muscles and skin around the double chin. Each type of exercise can be done every day 10 to 15 times.
Straight jaw jut
  1. Incline your head back and look toward the ceiling.
  2. Push your lower jaw forward to feel a stretch under the chin.
  3. Hold the jaw jut for a 10 count.
  4. Relax your jaw and return your head to a neutral position.
Ball exercise
  1. Place a 9- to10-inch ball under your chin.
  2. Press your chin down against the ball.
  3. Repeat 25 times daily.
Pucker up
  1. With your head tilted back, look at the ceiling.
  2. Pucker your lips as if you’re kissing the ceiling to stretch the area beneath your chin.
  3. Stop puckering and bring your head back to its normal position.
Tongue stretch
  1. Looking straight ahead, stick your tongue out as far as you can.
  2. Lift your tongue upward and toward your nose.
  3. Hold for 10 seconds and release.
Neck stretch
Tilt your head back and look at the ceiling.
  1. Press your tongue against the roof of your mouth.
  2. Hold for 5 to 10 seconds and release.
Bottom jaw jut
  1. Tilt your head back and look at the ceiling.
  2. Turn your head to the right.
  3. Slide your bottom jaw forward.
  4. Hold for 5 to 10 seconds and release.
  5. Repeat the process with your head turned to the left.

Non-Invasive Procedures to Reduce Double Chin

When the skin is aging, the neck is probably the first place to indicate your age. For most people, fat is more likely to compose underneath the chin. This excess fat and lax skin can affect your overall look. However, a reliable doctor can restore or fix a double chin by employing any of the procedures below:

https://preview.redd.it/ckklgwin0nqc1.png?width=1080&format=png&auto=webp&s=f033d2f99aabb38527e055ebf05a7220b48b05f6

1. Define

Define Cheek and Define Chin are advanced hands-free facial technologies specifically designed to enhance the appearance of the lower face and neck. These headsets utilize a patented technology that employs Radio Frequency (RF) energy to treat facial tissue, resulting in improved aesthetic outcomes.

Key Features:

  • RF Energy: Harnesses the power of Radio Frequency to target facial tissues.
  • Hands-Free Application: Offers a convenient and comfortable treatment experience.
  • Focus on Lower Face and Neck: Specifically targets these areas for rejuvenation.

2. Face-Tite

This is an FDA-certified treatment and is slightly invasive. It delivers radiofrequency (RF) energy underneath the skin’s surface to dissolve fat. Since it facilitates collagen production, the skin tightens within the dermis.
The Face-Tite treatment can be used for areas with serious lines and loose skin on the shoulders. But, it is commonly urged to have firmness to the chin, jowls, cheeks, neck, eyes, mouth, brow, forehead, or jawline.
https://preview.redd.it/8gh6a5sq0nqc1.png?width=1080&format=png&auto=webp&s=a2ca9d26da830636de83b6efe336e4151a28563f

3. Suture Suspension Neck Lift

Suture Suspension Neck Lift is a minimally invasive nonsurgical procedure that shapes and rejuvenates the jawline. Using the ICLED® light guided suture system, Dr. Greta McLaren can easily place a suture support system without incisions that immediately elevate ad approximates underly structures. Suture Suspension Neck Lift can be used as a standalone procedure or in conjunction with submental liposuction and energy-based skin tightening technologies like “FaceTite”.

Am I candidate for Suture Suspension Neck Lift?

  • Early aging of the Chin and Neck
  • Excess fullness or a Double Chin
  • Minimal skin laxity
  • Sagging glands
  • Muscle bands
  • Pronounce Jowling
  • Turkey Neck (Vertical Banding)
  • Subpar Jawline Contouring and Definition

https://preview.redd.it/qpvbayos0nqc1.png?width=450&format=png&auto=webp&s=85391aa4d61ff06c2a00ddc2819021808976d6d5

4. Renuvion Cosmetic Technology

Renuvion is a top-of-the-line, mildly intrusive technology that is FDA-approved for cutting, thickening, and melting soft tissue. This is done during unrestricted and laparoscopic surgical methods.
Renuvion is a cutting-edge energy technology that combines the unique features of cold helium plasma with radio frequency energy. Helium plasma concentrates on RF energy for better tissue impact control, allowing for more accuracy and nearly avoiding unwanted tissue harm. Renuvion offers exceptional accuracy and control in minimally invasive operations by combining the unique features of helium plasma with the efficiency of RF energy.

https://preview.redd.it/l6d3ffsx0nqc1.png?width=738&format=png&auto=webp&s=a781ccdfd9785dfdcd621c5ad839e1e9b34544bb

5. Kybella Injectables

KYBELLA® is an FDA-approved injectable treatment used to reduce moderate to severe fat below the chin, commonly known as a “double chin.” The active ingredient in Kybella is synthetic deoxycholic acid, a molecule that naturally occurs in the body and helps break down and absorb dietary fat. When injected into the fat beneath the chin, Kybella destroys fat cells, resulting in a noticeable reduction in fullness under the chin. Once these cells are destroyed, they can no longer store or accumulate fat, meaning further treatment is not expected once you reach your desired aesthetic goal.
It’s important to note that Kybella should only be administered by a trained healthcare professional, and the number of treatment sessions varies from patient to patient, depending on the amount of submental fat and the patient’s treatment goals. Treatment sessions are typically spaced at least one month apart, and no more than six treatments are recommended.

https://preview.redd.it/lti1tx7z0nqc1.png?width=600&format=png&auto=webp&s=64df3b5f6c5a0661572f1be435e878f97ca3eb74

6. Agnes RF

AGNES Radio Frequency is an energy-based aesthetic and treatment procedure. It is a non-surgical radiofrequency device that treats eye bags without having to go under the knife! It incorporates an enclosed coated microneedle devised for the various deepness of skin with exact pulse and duration control of radiofrequency energy specifically targeting the sebaceous gland (which causes acne), blackheads, syringomas, wrinkles, and fat under the eyes and chin.
Since the needles are developed to a particular depth, they will shield other structures in the skin and also the epidermis while having the treatment. Agnes RF results in minimal disruption and side effects most especially pigmentation.

Cape Coral, FL's Premiere Medical Spa and Anti-Aging Clinic

The face is usually considered as the doorway to the soul. Your anxieties, beliefs, and desires, will be evident on your look in some manner or the other. Whatever consists of your diet and physical activities are being portrayed on the face. One of the worrying features of the face for most people is the double chin.
If you want to have a well-defined face, Skin Secrets does an advanced treatment in Cape Coral, FL to reduce the double chin. We deal with every face in a top-of-the-line technique, utilizing our proficiency in cosmetic remedies to improve your look.
Dr. Greta McLaren is passionate about performing aesthetic practices and agreeably restoring looks for her clients. With well-planned and suitable facial treatments, you will undergo pleasant and naturalistic outcomes.
Our professional and amiable patient care team is willing to meet and discuss with you whatever the skin issues and needs are. We will design a tailored rejuvenative treatment to help you become the best version of yourself.
Contact us today at 239-800-SKIN(7546) to schedule your complimentary consultation.
submitted by skinsecrets46 to u/skinsecrets46 [link] [comments]


2024.03.12 00:20 cats119 I would like to hear someone elses opinion (swollen lymph nodes) F18

I noticed about two months ago a swollen lymph node in left side of my neck, did an ultrasound and i got these results:
Thyroid gland of appropriate position and size, homogeneous of adequate Cd signal with a couple of colloid nodules dominant in the left lobe up to 5 mm. Salivary glands homogeneous Bilateral submandibular single oval igi up to 19 mm visible hilus Submental Igl up to 6 mm visible hilus Along the jugular chain behind m SCMi towards the occipitally more to the left more oval igi up to 11 mm mostly visible hilus Right occipitally towards the cosmatory part oval hypoechoic change in diameter about 10 mm smaller central hyperechoic hilus initially reactive Igl

Doc prescribed me ospamax, that one still didnt go away
I also noticed some swollen lymph nodes in groin but they were movable and kinda soft?
I did thyroid tests, they were in limits of normal so i don't have any thyroid disorder for sure
Did blood results before all that and they also were normal?Only my monocytes and ldh were a little high

I don't want to go to the doctor again bescause i am scared of their reaction, i can't really talk about my symptoms bescause it is kinda messy (it is hard for me to distinguish whether they are different from the ones I had before, bescause i have a neuropathy from nerve compression and now i stopped taking milgamma mono and it is really hard for me bescause i am feeling really tired all the time)
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2024.03.03 21:11 Jaded-Vast-3966 3 Lymph node scans in 1-2 yrs. (results below). cancer possible?

22F, 103 lbs, 5’3
(this is long because i included the Ultrasound and CT scan findings)
I’ve had swollen nodes for a couple years now, at least 4 years. I ignored them at first as I had frequent strep infections with the last time being twice in 2019. Starting at the end of 2019, i started getting fatigued enough to seek help. Doctors keep telling me im fine after extensive bloodwork and a sleep study. I decided to pursue lymph node investigation after hearing stubborn swelling can indicate cancer, especially after years for searching for answers as to why i was so fatigued.
Was referred to ent, and initial US (August 2022) showed this:
TECHNIQUE: Grayscale and color Doppler imaging was performed with a high frequency linear transducer in evaluation of the area of concern of the submandibular region and bilateral neck IIA anatomic regions
COMPARISON: None
COMMENT: In the area of concern in the mid neck in the right and left IIA anatomic regions there are prominent lymph nodes with preserved fatty hila measuring 14 x 4 x 15 mm and 22 x 6 x 19 mm on the right side and 19 x 5 x 15 mm on the left side. Additionally, in the submandibular region there are several prominent lymph nodes with preserved anatomy measuring 7 x 3 x 8 mm and 6 x 4 x 6 mm.
IMPRESSION: Prominent lymph nodes in the neck as described.
ENT said let’s get a FNA to be sure. Unfortunately the radiologist took sample from a node with a long axis of only 9mm, which isn’t one of the stubbornly enlarged ones. (but still in the same anatomical region as the swollen ones) Results came back negative for cancer cells. ENT still said it’s enough and i can forget about the nodes unless there are changes.
I advocated for further investigation, as i feel like a rotting corpse everyday. CT scan showed this (omitted everything else that was found to be unremarkable): (December 2023)
Nodes: No adenopathy seen.Subcentimeter in short axis circumscribed elliptical level 2, level 3 lymph nodes are noted bilaterally largest left level 2 lymph node measures 3.3 x 1.7 x 0.9 cm, with vascular hilum noted.
Salivary Glands: No evidence of mass or other focal abnormality. Palpable markers correspond to the submandibular glands. No dilated ducts, or calcified sialoliths. Another marked lump at the midline corresponds to a nonenlarged submental lymph node measuring 0.3 cm.
IMPRESSION: NO ABNORMAL ENHANCING MASS. NO ACUTE INFLAMMATORY CHANGES. NO SIGNIFICANT ADENOPATHY BY SIZE CRITERIA.
Note, the left level 2 node was more swollen than usual as i had just gotten over a cold when I had this CT done, it has since then went down to its regular swollen size. apart from when im sick, this node waxes and wanes a lot for no reason. It also wasn’t one of the notes i initially felt years ago, so i don’t even know how long it’s been there. It was noticed by my ENT at my first appointment with him.
My ENT then said just to be sure, esp given my symptoms, we’ll biopsy this node. I was finally ready to get this over with and get a solid answer, but then another radiologist who was just suppose to do the biopsy scanned my nodes again via ultrasound and released this report after telling me the chance of malignancy is “close to zero”, as they’re not even considered enlarged, they are apparently symmetrical, and haven’t changed since the initial US.
(February 2023) Imaging of the area of interest in the neck demonstrates no enlarged or morphologically abnormal lymph nodes. This finding is similar to what was seen on the prior digitized CT from December 2023 as well as prior ultrasound of August 2022. There is no pathologically abnormal node for which biopsy can be attempted. IMPRESSION: No enlarged or morphologically abnormal lymph nodes are identified on examination. No biopsy was performed.
Additionally, i got an Xray for scoliosis related issues which i guess would’ve noted anything that could’ve been incidentally found but didn’t.
I was so ready to forget this, but i feel like a rotting corpse everyday. so so sick but symptoms are too general to pursue anything specific. it’s not a sleep issue, or deficiency issue, or mental issue, or anything like that. I am rotting internally. I get anything low grade would be very out of the ordinary for someone my age, but so is feeling like this for no reason.
what really triggers me is seeing stories of people who’ve been told for years they were fine, then ended up with a cancelymphoma diagnosis, and that many doctors told them they were fine but they always knew something was wrong with their body. I feel like my work up for this has been fairly thorough, but at the same time beating around the bush for what really would shut this down: an accurate biopsy. Now, i just feel crazy if i were to ask like the 5th time for another biopsy after even the radiologist told me nothing is abnormal enough to biopsy.
Is this enough of a work up to just forget this issue despite my paralyzing fatigue?
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2024.03.01 12:58 dontwastemityme is TI-RADS 4 always bad?

My health anxiety is skyrocketing right now. I just got my neck US result and the result doesnt seem good. I’m freaking out because of the nodule seen in my thyroid left lobe. I also have swollen lymph node above my right collarbone for more than 2 weeks now. I’ve been taking co-amoxiclav as prescribed by my ENT. im on my 5th day and it hasn’t cleared up. Can someone help me ease my mind pls?
this is my neck and thyroid ultrasound result.
Thyroid: Normal ni size with homogeneous parenchyma and normal vascularity on color Doppler interrogation. Isthmus is not thickened (0.14 cm). Right thyroid lobe - 4.4 x 1.27 x 1.11 cm; Left thyroid lobe - 4.36 x 1.57 x 1.33 cm
A nodule is seen in the left lobe described below: .1 Lower: Solid, Isoechoic, wider than tall with smooth borders and internal calcifications measuring 2.17 x 1.28 x 1.09 cm - TIRADS 4, Tissue correlation (FNAB) is suggested.
No lesions are seen in the right thyroid lobe and isthmus. Parotid and submandibular glands: Unremarkable
Lymph nodes: Prominent sized to enlarged lymph nodes with absent fatty hila are seen in the right supraclavicular region with the largest having a short axis diameter of 1.03 cm. Unenlarged lymph nodes with intact fatty hila are seen in the submental, bilateral submandibular, jugulocarotid, posterior cervical and left supraclavicular regions.
IMPRESSION: Normal sized thyroid gland with left nodule as described above. Cervical lymphadenopathy, right supraclavicular region.
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2024.02.27 01:43 Sevenburgh MZL detected, Radiation planned, Requesting answers for questions inside image from all and Dr /u/Erel_Joffe_MD

MZL detected, Radiation planned, Requesting answers for questions inside image from all and Dr Erel_Joffe_MD submitted by Sevenburgh to Lymphoma_MD_Answers [link] [comments]


2024.02.17 19:15 hunglikebull69 Need help interpreting neck ultrasound results

Hi all,
I just recently had a neck ultrasound on my lymph nodes and thyroid with a lot of feedback. I don’t understand a lot of it and was wondering if anyone could provide some color. I’m just really concerned and didn’t want to wait until follow up appointment if there’s anything to be really worried about.
For reference, I’m a 23 year old male with initial concerns of a mass in the upper neck (near hyoid bone).
CLINICAL HISTORY: Palpable lump in the right neck.
TECHNIQUE: High-resolution soft tissue neck ultrasound is performed.
FINDINGS: In the right neck, there is an intraparotid probable lymph node 8 x 5 mm and 7 x 3 mm with faint fatty hila present. There is a level 2 lymph node that is elongated measuring 31 x 6 mm. Very faint fatty hilus but not enlarged in the short axis. Nonspecific. There is a level 2 lymph node measuring 14 x 6 mm with very faint fatty hilus. Not enlarged in the short axis.
The right thyroid lobe is hypervascular which can be seen with underlying thyroiditis with numerous sub-5 mm thyroid nodules. Recommend dedicated thyroid ultrasound at this time to further evaluate.
Underlying the palpable area in the submental region, there is a level 1 probable lymph node measuring 8 x 4 mm with very faint fatty hilus. Not enlarged in the short axis.
In the left neck, there is a level 2 lymph node measuring 18 x 5 mm with no defined fatty hilus but not enlarged in the short axis. Nonspecific. There is a level 2 lymph node that is elongated measuring 24 x 6 mm with very faint fatty hilus. Not enlarged in the short axis.
The left thyroid lobe is hypervascular which can be seen with underlying thyroiditis with innumerable sub-5 mm thyroid nodules requiring dedicated thyroid ultrasound at this time to further evaluate.
IMPRESSION: Underlying the palpable area in the submental region, there is a level 1 submental lymph node measuring 8 x 4 mm. Not enlarged in the short axis. Recommend further clinical correlation to ensure that the palpable area resolves an ultrasound follow-up of the neck in 3 months to further evaluate.
Additional bilateral cervical chain lymph nodes not enlarged in the short axis. Nonspecific. Recommend attention at follow-up.
Numerous bilateral thyroid nodules with hypervascular thyroid gland which can be seen with underlying thyroiditis. Recommend dedicated thyroid ultrasound at this time to further assess.
Thanks in advance for any support you can provide!
submitted by hunglikebull69 to AskDocs [link] [comments]


2024.01.25 02:37 skinsecrets46 How to Get Rid of a Double Chin?

How to Get Rid of a Double Chin?
A submental fat, commonly known as a double chin, can be a stubborn aesthetic concern, forming unsightly layers under the chin and impacting jaw line improvement. This condition often arises with weight gain, but can also be influenced by genetics or natural skin laxity due to aging.
If you’re looking to achieve a more sculpted and youthful appearance, various options are available to address a double chin and enhance jaw line definition. Some methods focus on reducing excess fat, while others target skin tightening and contouring.

Exercises that target a double chin

There’s no scientific proof or data yet that says chin exercises are worthwhile in removing the double chin, however, there is enough anecdotal evidence to get you started.
Here are six workout activities that can boost and tone the muscles and skin around the double chin. Each type of exercise can be done every day 10 to 15 times.

Straight jaw jut

  1. Incline your head back and look toward the ceiling.
  2. Push your lower jaw forward to feel a stretch under the chin.
  3. Hold the jaw jut for a 10 count.
  4. Relax your jaw and return your head to a neutral position.

Ball exercise

  1. Place a 9- to10-inch ball under your chin.
  2. Press your chin down against the ball.
  3. Repeat 25 times daily.

Pucker up

  1. With your head tilted back, look at the ceiling.
  2. Pucker your lips as if you’re kissing the ceiling to stretch the area beneath your chin.
  3. Stop puckering and bring your head back to its normal position.

Tongue stretch

  1. Looking straight ahead, stick your tongue out as far as you can.
  2. Lift your tongue upward and toward your nose.
  3. Hold for 10 seconds and release.

Neck stretch

  1. Tilt your head back and look at the ceiling.
  2. Press your tongue against the roof of your mouth.
  3. Hold for 5 to 10 seconds and release.

Bottom jaw jut

  1. Tilt your head back and look at the ceiling.
  2. Turn your head to the right.
  3. Slide your bottom jaw forward.
  4. Hold for 5 to 10 seconds and release.
  5. Repeat the process with your head turned to the left.

Procedures to Reduce Double Chin

When the skin is aging, the neck is probably the first place to indicate your age. For most people, fat is more possible to compose underneath the chin. This excess fat and lax skin can affect your overall look. However, a reliable doctor can restore or fix a double chin employing any of the procedures below:

1. Face-Tite

This is an FDA-certified treatment and is slightly invasive. It delivers a radiofrequency (RF) energy underneath the skin’s surface to dissolve fat. Since it facilitates collagen production, the skin tightens within the dermis.
The Face-Tite treatment can be used for areas with serious lines and loose skin on the shoulders. But, it is commonly urged to have the firmness to the chin, jowls, cheeks, neck, eyes, mouth, brow, forehead, or jawline.
https://preview.redd.it/jdvyfo9qphec1.png?width=1024&format=png&auto=webp&s=2d85d3f71ded7436505f66eefc4422630ec572eb

2. Suture Suspension Neck Lift

Suture Suspension Neck Lift is a minimally invasive nonsurgical procedure that shapes and rejuvenates the jawline. Using the ICLED® light guided suture system, Dr. Greta McLaren can easily place a suture support system without incisions that immediately elevate ad approximates underly structures. Suture Suspension Neck Lift can be used as a standalone procedure or in conjunction with submental liposuction and energy-based skin tightening technologies like “FaceTite”.

Am I candidate for Suture Suspension Neck Lift?

  • Early aging of the Chin and Neck
  • Excess fullness or a Double Chin
  • Minimal skin laxity
  • Sagging glands
  • Muscle bands
  • Pronounce Jowling
  • Turkey Neck (Vertical Banding)
  • Subpar Jawline Contouring and Definition

3. Renuvion Cosmetic Technology

Renuvion is a top-of-the-line, mildly intrusive technology that is FDA-approved for cutting, thickening, and melting soft tissue. This is done during unrestricted and laparoscopic surgical methods.
Renuvion is a cutting-edge energy technology that combines the unique features of cold helium plasma with radio frequency energy. Helium plasma concentrates on RF energy for better tissue impact control, allowing for more accuracy and nearly avoiding unwanted tissue harm. Renuvion offers exceptional accuracy and control in minimally invasive operations by combining the unique features of helium plasma with the efficiency of RF energy.
https://preview.redd.it/jav92v4vphec1.png?width=600&format=png&auto=webp&s=c3bf86cbb1af6d078c60b286dc7ac699596101d0

4. Agnes RF

Renuvion is a top-of-the-line, mildly intrusive technology that is FDA-approved for cutting, thickening, and melting soft tissue. This is done during unrestricted and laparoscopic surgical methods.
Renuvion is a cutting-edge energy technology that combines the unique features of cold helium plasma with radio frequency energy. Helium plasma concentrates on RF energy for better tissue impact control, allowing for more accuracy and nearly avoiding unwanted tissue harm. Renuvion offers exceptional accuracy and control in minimally invasive operations by combining the unique features of helium plasma with the efficiency of RF energy.

Cape Coral, FL's Premiere Medical Spa and Anti-Aging Clinic

The face is usually considered as the doorway to the soul. Your anxieties, beliefs, and desires, will be evident on your look in some manner or the other. Whatever consists of your diet and physical activities are being portrayed on the face. One of the worrying features of the face for most people is the double chin.
If you want to have a well-defined face, Skin Secrets does an advanced treatment in Cape Coral, FL to reduce the double chin. We deal with every face in a top-of-the-line technique, utilizing our proficiency in cosmetic remedies to improve your look.
Dr. Greta McLaren is passionate about performing aesthetic practices and agreeably restoring looks for her clients. With well-planned and suitable facial treatments, you will undergo pleasant and naturalistic outcomes.
Our professional and amiable patient care team is willing to meet and discuss with you whatever the skin issues and needs are. We will design a tailored rejuvenative treatment to help you become the best version of yourself.
Contact us today at 239-800-SKIN(7546) to schedule your complimentary consultation.
submitted by skinsecrets46 to u/skinsecrets46 [link] [comments]


2023.12.04 21:01 Key-Produce-9242 NBME CBSE ACTUAL TEST QUESTIONS ANDANSWERS (Quiz bank with all the correct answers)(usmle step 1,2 and 3) Medical examination Full Pack

NBME CBSE ACTUAL TEST QUESTIONS AND
ANSWERS (Quiz bank with all the correct answers)
(usmle step 1) Medical examination
62 year old woman - osteoporosis - a bisphosphonate is prescribed. The expected
beneficial effect of the drug is due to which of the following? -Answer- Decreased
Osteoclast Activity
Cohot Study of elderly women - relative risk ratio for hip fractures among those who
exercise regularly is 1.2 (95% confidence interval of 1.1 to 1.8). Which of the following is
the conclusion about the effect of exercise on the risk of hip fracture? -AnswerStatistically Significant Overall Increase Risk
52 year old man goes to ER with chest pain radiating to his jaw while shoveling snow.
Pulse is 80/min and blood pressure is 130/70. The most immediate treatment
mechanism of action? -Answer- Increased nitric oxide concentration
24 year old woman - spilled hot grease on her left leg while working at a fast-food
restaurant. Exam of leg shows 7cm pink, soft, granular, edematous wound. The
formation of this tissue was most likely caused by increased activity of which? -Answera. Vascular Endothelial Growth Factor
VEGF - stimulates angiogenesiss.
TYPE III Collagen = Blood Vessels - early wound repair
27 year old man- MVC - skull x-ray shows a linear, nondepressed basal skull fracture -
increased serum osm and decreased urin osm. Following desmopressin urine osm
increases. Desmopressin's effect is due to the activation of which of the following? -
Answer- a. Adenylyl Cyclase
Adenylate Cyclase - ATP - CAMP -- PROTEKINASE A -- Ca
v2
A 10 month old boy - 4 day history of fever and cough. He attends day care center.
Chest exam shows intercostal retractions along with bilateral, diffuse wheezes and
expiratory rhonchi. The infectious agent most likely has which of the following
properties? -Answer- Mediation of Cell Entry via a fusion protein
A 17 year old girl in ED - 15 minutes after being stung by a bee. Mild light headedness
but no difficulty swallowing- Bilateral wheezing - Which is most appropriate
pharmacotherapy for this patient? -Answer- B2- Agonist
14 year old boy - 2 day history of sore throat and fever that peaks in late afternoon. 1
week of fatigue. He recentaly had sex with one partner. Physical exam show cervical
lymphadenopathy and pharyngeal erythema with a creamy exudate. DX? -AnswerInfectious Mononucleosis

57 year old man - radiation therapy for squamous cell carcinoma of the lung. Despite
therapy, tumor increases in size and he dies 6 months later. The progressive tumor
growth is due to a defect in cell cycle arrest in which of the following phases of cell
cycle? -Answer- G1
28 year old - lived in sub-Saharan Africa - until he came to the US. Temp of 100.4 -
imaging shows bilateral hydroureter and hydronephrosis. Biopsy shows marked fibrosis
and scattered granulomas. DX? -Answer- Schistosomiasis
A couple with a family history of a-thalassemia. Woman has one gene deletion and man
has two gene deletion. If the two gene is trans - what percentage of offspring will have a
two gene deletion? -Answer- 50%
previously healthy 40 year brought to emergency department by her husband - 2 day
history of fever, lethargy, confusion. PE shows scattered petechiae and ecchymoses
over the lower extremities - 3+ polychromasia and 3+ schistocytes and Low platelets
Dx? -Answer- Thrombotic Thrombocytopenia Purpura
16 year old boy - is admitted to the ER because of a knife wound to the left side of his
chest. An X-ray of the chest shows an air-fluid level in the left side of the chest, partial
collapse of the left lung, and the elevation of the stomach bubble. The mediastinum is
midline. DX? -Answer- Hemopneumpothorax under tension
49 year old woman - coronary artery disease - BP 140/90 - High Cholesterol, High LDL
(190), High triglycerides (350) - TX with atorvastatin and losartan. What are the effects
on HDL and Triglycerides? -Answer- HDL increased
Triglycerides Decreased
73 yeare old - diffuse weakness and tingling of her arms and legs. Sensation and
vibration and position is decreased in all extremities. What vitamin deficiency? -AnswerVitamin B12 - (cyanocobalamin)
Tea and Toast - low B12 in diet
15 year old girl - 3 month history of acne - which is the underlying cause of the patients
acne? -Answer- Stimulation of Sebaceous Glands by androgens
b. ACNE = Propionibacterium ACNE
4 year old from Brazil - PE shows single 12x10cm lesion in the right side of jaw with
diffuse regular edges. Photomicrographs of an incisional biopsy (looks like Burkitts
Lymphoma/ Starry night) - which of the processes most likely to occur in the region
indicated by the arrow? -Answer- Apoptosis
b. Endemic Burkitt lymphoma can happen in Brazil as well as Africa (jaw lesion, puffy
face).

51 year old - lump on tongue - 1 pack smoking history for 30 years. 1.5 cm mass on
apex of tongue. It is most appropriate to evaluate which lymph nodes first for evidence
of metastasis? -Answer- Submental
15 year old boy - ER - 2 hour history of confusion and agitation - fever, headache, stiff
neck, and vomiting - since returned from summer camp - patient is hallucinating -
lumbar puncture - shows cysts and trophozoites- most likely pathogen? -Answer- a.
Olfactory Nerve
Naegleria fowleri
17 year old - ED - 30 minutes after being found with a blank stare. Physical exam shows
rigidity. During exam he becomes hostile and assaults physician - Pt ingested which
drug? -Answer- PCP
Placebo controlled clinical trial - 5000 pts with essential hypertension. 2500 patients
receive new drug and 2500 patients receive placebo. If alpha is set at 0.01 instead of
0.05, which of the following is most likely result? -Answer- Significant findings can be
reported with greater confidence
17 year old - gymnast - comes to hospital because of lack of menstrual period for 6
months. BMI 15 Which is the cause of the amenorrhea? -Answer- Hypogonadotropic
Hypogonadism
A male stillborn is delivered at 32 weeks - Oligohydramnios - absence of a urethral
opening. Which is most likely finding? -Answer- Pulmonary Hypoplasia
A 6 day old - breast fed boy in ED - poor weight gain and irritability since delivery -
Physical exam shows jaundice and hepatomegaly. The concentration of which of the
following metabolites is most likely increased? -Answer- a. Galactose - 1 - phosphate
b. Congenital intolerance to breast milk
A 25-year-old man - comes to ED - severe muscle pain, diffuse, painful swelling of his
neck, underarms, and groin after camping in New Mexico Generalized scattered black
maculae. Examination of the right upper extremity shows erythematous, solid, tender
mass. Mass is draining blood and necrotic material. The most effective antibiotic for
patient disorder will interfere with which of the following processes? -Answer- a.
Ribosomal Assembly
b. Yersenia Pestis
45 year old - progressive weakness - muscle fasciculations of the upper extremities and
weakness of the lower extremity - What additional findings? -Answer- Atrophy
b. ALS = Lou Gherig
A new severe respiratory illness - Why use a killed vaccine vs a live vaccine? -AnswerAvoids Concerns of reversion to virulence
b. Killed vaccines - avoids reversion to virulence

c. live vaccines - can (but rarely do) cause the disease they're designed to prevent
A 33 year old - keratinizing squamous cell carcinoma of cervix. Which of the following
describes pathogenesis of this patient's disease? -Answer- Inactivation of Cellular P53
b. p53 protein = tumor suppressor (it activates apoptosis) - most human cancer
A 54 year old - 40 year history of T1DM - receiving hemodialysis for end stage renal
disease while awaiting a kidney transplant. Receives a drug that induces reticulocyte
release from bone marrow and stimulates a cytokine receptor that signals Jak/Stat
pathway? -Answer- Erythropoietin
Jak Stat - erythropoietin
During a clinical study examining the effects of exercise. The average pulse is 175/min.
Compared with measurement before the session, which is most likely decreased? -
Answer- Total Peripheral Resistance
An 8-year-old boy - 3-day history of fever, sore throat, and itchy eyes. Returned from
week long summer camp that includes hiking trips and swimming - PE shows
conjunctival injection and oropharyngeal edema - Outbreak among other campers.
Which is most likely cause of this patient's symptoms? -Answer- Adenovirus
Fever+ Sore Throat + Itchy Eyes
transmitted via swimming pools
Conjunctivitis Viral = adenovirus
44-year-old woman - 10 month history of wide red streaks over her lower trunk (striae)
and weight gain in face (moonface). Which additional findings? -Answer- Hypertension
and muscle weakness
b. Cushing - hypertension and muscle weakness
12 year old boy - pain below left knee -unable to play soccer - An x-ray shown - Which
structures attached to the abnormal anterior tibial area? -Answer- patellar ligament
b. Osgood-Schlatter
A 65-year-old health maintenance exams - He lives is a single-family home with his cat
and dog. He spend much of his time in his basement woodworking shop. This patient is
increased risk for lung cancer due to which of the following environmental exposures? -
Answer- Radon
54-year-old man - intense overwhelming fear. Which portion of brain stimulated? -
Answer- Amygdala
b. Fear = amygdala
30 year old woman - recurrent URI - Sweat is Salty- Genetic testing for 36 most
common mutations shows detectable G551D in one allele of CFTR - What is patients
clinical phenotype? -Answer- The Second CFTR Gene was not detected by the testing
obtained

submitted by Key-Produce-9242 to u/Key-Produce-9242 [link] [comments]


2023.11.22 18:55 One_Recognition2566 NBME CBSE ACTUAL TEST QUESTIONS AND ANSWERS(Quiz bank with all the correct answers)(usmle step 1)Medical examination

NBME CBSE ACTUAL TEST QUESTIONS
AND ANSWERS
62 year old woman - osteoporosis - a bisphosphonate is prescribed. The expected
beneficial effect of the drug is due to which of the following? Correct answer- Decreased
Osteoclast Activity
Cohot Study of elderly women - relative risk ratio for hip fractures among those who
exercise regularly is 1.2 (95% confidence interval of 1.1 to 1.8). Which of the following is
the conclusion about the effect of exercise on the risk of hip fracture? Correct answerStatistically Significant Overall Increase Risk
52 year old man goes to ER with chest pain radiating to his jaw while shoveling snow.
Pulse is 80/min and blood pressure is 130/70. The most immediate treatment
mechanism of action? Correct answer- Increased nitric oxide concentration
24 year old woman - spilled hot grease on her left leg while working at a fast-food
restaurant. Exam of leg shows 7cm pink, soft, granular, edematous wound. The
formation of this tissue was most likely caused by increased activity of which? Correct
answer- a. Vascular Endothelial Growth Factor
VEGF - stimulates angiogenesiss.
TYPE III Collagen = Blood Vessels - early wound repair
27 year old man- MVC - skull x-ray shows a linear, nondepressed basal skull fracture -
increased serum osm and decreased urin osm. Following desmopressin urine osm
increases. Desmopressin's effect is due to the activation of which of the following?
Correct answer- a. Adenylyl Cyclase
Adenylate Cyclase - ATP - CAMP -- PROTEKINASE A -- Ca
v2
A 10 month old boy - 4 day history of fever and cough. He attends day care center.
Chest exam shows intercostal retractions along with bilateral, diffuse wheezes and
expiratory rhonchi. The infectious agent most likely has which of the following
properties? Correct answer- Mediation of Cell Entry via a fusion protein
A 17 year old girl in ED - 15 minutes after being stung by a bee. Mild light headedness
but no difficulty swallowing- Bilateral wheezing - Which is most appropriate
pharmacotherapy for this patient? Correct answer- B2- Agonist
14 year old boy - 2 day history of sore throat and fever that peaks in late afternoon. 1
week of fatigue. He recentaly had sex with one partner. Physical exam show cervical
lymphadenopathy and pharyngeal erythema with a creamy exudate. DX? Correct
answer- Infectious Mononucleosis

57 year old man - radiation therapy for squamous cell carcinoma of the lung. Despite
therapy, tumor increases in size and he dies 6 months later. The progressive tumor
growth is due to a defect in cell cycle arrest in which of the following phases of cell
cycle? Correct answer- G1
28 year old - lived in sub-Saharan Africa - until he came to the US. Temp of 100.4 -
imaging shows bilateral hydroureter and hydronephrosis. Biopsy shows marked fibrosis
and scattered granulomas. DX? Correct answer- Schistosomiasis
A couple with a family history of a-thalassemia. Woman has one gene deletion and man
has two gene deletion. If the two gene is trans - what percentage of offspring will have a
two gene deletion? Correct answer- 50%
previously healthy 40 year brought to emergency department by her husband - 2 day
history of fever, lethargy, confusion. PE shows scattered petechiae and ecchymoses
over the lower extremities - 3+ polychromasia and 3+ schistocytes and Low platelets
Dx? Correct answer- Thrombotic Thrombocytopenia Purpura
16 year old boy - is admitted to the ER because of a knife wound to the left side of his
chest. An X-ray of the chest shows an air-fluid level in the left side of the chest, partial
collapse of the left lung, and the elevation of the stomach bubble. The mediastinum is
midline. DX? Correct answer- Hemopneumpothorax under tension
49 year old woman - coronary artery disease - BP 140/90 - High Cholesterol, High LDL
(190), High triglycerides (350) - TX with atorvastatin and losartan. What are the effects
on HDL and Triglycerides? Correct answer- HDL increased
Triglycerides Decreased
73 yeare old - diffuse weakness and tingling of her arms and legs. Sensation and
vibration and position is decreased in all extremities. What vitamin deficiency? Correct
answer- Vitamin B12 - (cyanocobalamin)
Tea and Toast - low B12 in diet
15 year old girl - 3 month history of acne - which is the underlying cause of the patients
acne? Correct answer- Stimulation of Sebaceous Glands by androgens
b. ACNE = Propionibacterium ACNE
4 year old from Brazil - PE shows single 12x10cm lesion in the right side of jaw with
diffuse regular edges. Photomicrographs of an incisional biopsy (looks like Burkitts
Lymphoma/ Starry night) - which of the processes most likely to occur in the region
indicated by the arrow? Correct answer- Apoptosis
b. Endemic Burkitt lymphoma can happen in Brazil as well as Africa (jaw lesion, puffy
face).

51 year old - lump on tongue - 1 pack smoking history for 30 years. 1.5 cm mass on
apex of tongue. It is most appropriate to evaluate which lymph nodes first for evidence
of metastasis? Correct answer- Submental
15 year old boy - ER - 2 hour history of confusion and agitation - fever, headache, stiff
neck, and vomiting - since returned from summer camp - patient is hallucinating -
lumbar puncture - shows cysts and trophozoites- most likely pathogen? Correct answera. Olfactory Nerve
Naegleria fowleri
17 year old - ED - 30 minutes after being found with a blank stare. Physical exam shows
rigidity. During exam he becomes hostile and assaults physician - Pt ingested which
drug? Correct answer- PCP
Placebo controlled clinical trial - 5000 pts with essential hypertension. 2500 patients
receive new drug and 2500 patients receive placebo. If alpha is set at 0.01 instead of
0.05, which of the following is most likely result? Correct answer- Significant findings
can be reported with greater confidence
17 year old - gymnast - comes to hospital because of lack of menstrual period for 6
months. BMI 15 Which is the cause of the amenorrhea? Correct answerHypogonadotropic Hypogonadism
A male stillborn is delivered at 32 weeks - Oligohydramnios - absence of a urethral
opening. Which is most likely finding? Correct answer- Pulmonary Hypoplasia
A 6 day old - breast fed boy in ED - poor weight gain and irritability since delivery -
Physical exam shows jaundice and hepatomegaly. The concentration of which of the
following metabolites is most likely increased? Correct answer- a. Galactose - 1 -
phosphate
b. Congenital intolerance to breast milk
A 25-year-old man - comes to ED - severe muscle pain, diffuse, painful swelling of his
neck, underarms, and groin after camping in New Mexico Generalized scattered black
maculae. Examination of the right upper extremity shows erythematous, solid, tender
mass. Mass is draining blood and necrotic material. The most effective antibiotic for
patient disorder will interfere with which of the following processes? Correct answer- a.
Ribosomal Assembly
b. Yersenia Pestis
45 year old - progressive weakness - muscle fasciculations of the upper extremities and
weakness of the lower extremity - What additional findings? Correct answer- Atrophy
b. ALS = Lou Gherig
A new severe respiratory illness - Why use a killed vaccine vs a live vaccine? Correct
answer- Avoids Concerns of reversion to virulence

b. Killed vaccines - avoids reversion to virulence
c. live vaccines - can (but rarely do) cause the disease they're designed to prevent
A 33 year old - keratinizing squamous cell carcinoma of cervix. Which of the following
describes pathogenesis of this patient's disease? Correct answer- Inactivation of
Cellular P53
b. p53 protein = tumor suppressor (it activates apoptosis) - most human cancer
A 54 year old - 40 year history of T1DM - receiving hemodialysis for end stage renal
disease while awaiting a kidney transplant. Receives a drug that induces reticulocyte
release from bone marrow and stimulates a cytokine receptor that signals Jak/Stat
pathway? Correct answer- Erythropoietin
Jak Stat - erythropoietin
During a clinical study examining the effects of exercise. The average pulse is 175/min.
Compared with measurement before the session, which is most likely decreased?
Correct answer- Total Peripheral Resistance
An 8-year-old boy - 3-day history of fever, sore throat, and itchy eyes. Returned from
week long summer camp that includes hiking trips and swimming - PE shows
conjunctival injection and oropharyngeal edema - Outbreak among other campers.
Which is most likely cause of this patient's symptoms? Correct answer- Adenovirus
Fever+ Sore Throat + Itchy Eyes
transmitted via swimming pools
Conjunctivitis Viral = adenovirus
44-year-old woman - 10 month history of wide red streaks over her lower trunk (striae)
and weight gain in face (moonface). Which additional findings? Correct answerHypertension and muscle weakness
b. Cushing - hypertension and muscle weakness
12 year old boy - pain below left knee -unable to play soccer - An x-ray shown - Which
structures attached to the abnormal anterior tibial area? Correct answer- patellar
ligament
b. Osgood-Schlatter
A 65-year-old health maintenance exams - He lives is a single-family home with his cat
and dog. He spend much of his time in his basement woodworking shop. This patient is
increased risk for lung cancer due to which of the following environmental exposures?
Correct answer- Radon
54-year-old man - intense overwhelming fear. Which portion of brain stimulated?
Correct answer- Amygdala
b. Fear = amygdala
submitted by One_Recognition2566 to u/One_Recognition2566 [link] [comments]


2023.02.13 20:06 gecko567 Advice on neck CT results?

Anyone have anything similar? Wondering what I should do next. This is after a neck US with large lymph nodes in December.
PROCEDURE: CT SOFT TISSUE NECK W/
INDICATION: Generalized enlarged lymph nodes R591. .
Bilateral jugulodigastric nodes measuring up to 20 mm level 3. Small nonspecific bilateral cervical lymph nodes not grossly enlarged. Bilateral submandibular mild lymphadenopathy and submental adenopathy is present. Right submandibular lymph node largest up to 10 mm. Several small submental lymph nodes are identified.
Thyroid glands unremarkable.
Small supraclavicular and axillary lymph nodes are noted there are not grossly enlarged. No significant superior mediastinal adenopathy identified.
Lung apices are clear.
Bony structures are intact.
IMPRESSION: Nonspecific cervical adenopathy noted. Likely reactive adenopathy. Most prominent noted in the jugulodigastric chain bilaterally level 3
Small bilateral submandibular and submental lymph nodes and posterior cervical lymph nodes are seen. Clinical correlation advised. Follow-up recommended as necessary.
submitted by gecko567 to lymphnodes [link] [comments]


2022.12.29 21:26 PerspectiveOwn9988 Mono? Blood Cancer? Something(s) Else?

Hey all. I'm a 27 year old caucasian male, ~6ft and ~207 lbs. Regular gym-goer.
There's a big obstacle to my keeping this post short, which is that I’ve been having issues pretty much since July, and I don’t know which events and symptoms that have occurred across the period from then ‘til now are relevant to what’s currently going on and which are not. I will try my best to be concise, but apologies in advance for the fact that the concisely-stated story is still fairly lengthy.
To orient your consideration of the details to come, I’ll say up front that I am currently most concerned about the possibility of my having some kind of blood cancer. To make sense of why I’m concerned about this, here's the story of what’s been going on:
Back in July I developed symptoms of HPV. I was prescribed Imiquimod, and some weeks later - in August - had a full STI panel run to check for any other issues. I unexpectedly tested positive for the HIV antigen. One terrifying week later, I tested negative on two HIV follow-up tests. We – my doctor and I – concluded that the initial result must have been a false positive.
But then I got sick (Sep. 04th). It began with a sore throat, body aches, chills, loss of appetite; then, a night or two later, I had drenching (i.e. clothes-soaking) night sweats. Adding night sweats to the rest of the symptoms scared me into thinking that my negative HIV test was false and that I was undergoing the acute phase of HIV infection. But more HIV tests were done and I remained negative. (As of the present day I now have had a total of 5 or 6 negative HIV tests since the initial positive, including a mix of Ag/Ab tests and RNA Quantitative tests, and so it really doesn't seem likely to be HIV.)
The sore throat and aches went away, and for a couple of days so did the night sweats, chills, and loss of appetite. But then the latter three all came back a few days later. And to my great concern, pretty much that same cycle repeated for all of September - the night sweats, chills, and loss of appetite all coming and going together - with the night sweats varying in severity. Continuing into October, I continued to have intermittent night sweats at basically the same rate, but the chills and loss of appetite ceased to accompany them, and the night sweats were less often drenching.
One symptom that I noticed shortly after Sep. 04th were sensitive red marks on my hands that lasted several days and then vanished. It was due to the addition of this symptom that my doctors at the time concluded that the initial Sep. 04th infection was likely Hand, Foot, and Mouth Disease. But I continued to work with my PCP to figure out why I was continuing to have symptoms. Along the way we learned that I have very high blood pressure (avg. 150+ systolic, sometimes as high as 170s). I also had a kidney stone, and I had two mucoceles across the months of September-October, one of which lasted almost a month. (I mention the oral symptoms because they might be partly relevant to some more recent goings-on.) I also got two head colds: one in October and one in November.
I had lots of tests run in September: COVID, Mono, Monkeypox, CMV, HIV, all of which were negative. I had multiple CBCs over the course of Aug.-October, and everything looked normal on them except – something I'll discuss more shortly – my absolute monocytes, which were always either at the very top of the normal range or just beyond it. I had C-Reactive Protein test and blood sedimentation rate tests done in September, neither of which showed any elevated levels or signs of inflammation. I had a non-contrast CT scan of my kidneys after the kidney stone in October which did not reveal any kidney problems. I did two 24hr urine tests in November to check for a pheochromocytoma, neither of which showed any abnormalities. I most recently - i.e. this December - had an Epstein-Barr test, the results of which I'll discuss shortly.
Since October the night sweats have basically stopped, save for one evening which I'll comment on later because it's noteworthy. For a little while I hoped things had resolved.
But then I discovered that I have swollen lymph nodes. The largest and most numerous of these are in my right-side inguinal region; but I also have them on the left, as well as in my posterior cervical and submental regions, and behind (at least) my right knee. I don’t seem to have them in my supraclavicular, axillary, or epitrochlear regions at the moment. For the most part none of them have been sore, save for some of them recently (which I’ll discuss in more detail shortly). I also realized that my submandibular glands have been variably swollen for awhile. Moreover, I am beginning to suspect that an increasingly persistent dull pain towards the bottom of my left-side ribcage that I have been feeling intermittently for the past week might be spleen-related.
(We're almost done—forgive me and please hang in there!)
Early December I had a fourth CBC (since August) and it came back with high absolute monocytes (again) and bottom-of-the-normal-range neutrophils. I have all along been more concerned about the high monocytes than my doctor has been, but with the addition of the significant drop in neutrophils (relative to where they'd been on the previous CBCs, which was middle-of-normal-range), he had me re-do the test a week later with the idea that if they remained low he would refer me to a hematologist. I did, and the levels were basically the same: neutrophils had risen very slightly, but not nearly to where they'd previously been.
So I currently have, inter alia, (1) swollen lymph nodes in multiple areas, (2) over three months of top-of-range-or-abnormally-high monocytes, (3) possible-but-unconfirmerd spleen enlargement, and (4) high blood pressure. Additionally, for the past several weeks my submental and submandibular regions have been intermittently sore, and my jaw has been intermittently popping. I also recently noticed inflammation of my gum tissue behind my right molar.
Unfortunately my doctor is not yet up to date on (i) the newest CBC results, (ii) the possible spleen enlargement, (iii) the intermittent soreness of my submental and submandibular areas, or (iv) the gum inflammation, as the health center (at which he works) is closed until the new year and so neither he nor any staff are there to be reached. But right before my doctor went on break, he ran the aforementioned Epstein-Barr test on a hunch that Mono might be behind part of this story. The results were ambiguous, but they seemed to suggest a possible recent Epstein-Barr infection insofar as my Anti-VCA IgG levels were high, but I was negative for EBNA antibodies and negative for the Anti-VCA IgM antibodies. Altogether, these results led my doctor to suspect that my night sweats from Sept.-October were due to Mono, even though the Mono test we did in September was negative.
But I suppose I'm unsure about how plausible and comprehensive an explanation Mono is, as the timeline and symptomology seem pretty non-standard for Mono. (Notably, I don’t feel as though fatigue has been a symptom of mine throughout this period.) And I'm sufficiently worried about the closeness of my symptoms to leukemia - particularly CMML - and lymphoma that I am leaning towards thinking I should push to get bone marrow and lymph node biopsies performed to look for these.
Right before I left to visit family for the holidays, my doctor himself wanted me to do a chest x-ray to look for lymph node swelling in my chest; but unfortunately I neglected to bring the physical copy of his test order with me when I left, and, since his office isn’t open, I’m unable to get an emailed copy from them. I’m kind of just anxiously sitting here counting the days, waiting for my doctor’s office to re-open so I can proceed to get a chest x-ray and schedule an appointment with a hematologist.
So that’s what’s been going on. With the debrief now concluded, I figure I can wrap up by rattling off some stray thoughts and questions, which you may feel free to interact with or ignore as you wish.
(1) I mentioned the Imiquimod just because it recently occurred to me that perhaps it could be responsible for some of my symptoms. I discontinued it back in the middle of September, so I don’t know how much of what’s currently going on could possibly be due to it. But maybe it was partly responsible for some of the symptoms characterizing the September-October episode, and perhaps for the more severe lymph node swelling in my right inguinal region (which is nearest to where the Imiquimod was applied). I don’t know what to make of these hypotheses, but my recent research into the topic suggests that Imiquimod can cause lymph node swelling, flu-like symptoms, and decreased neutrophils. More frighteningly, WebMD has an article that lists ‘malignant lymphoma’ as a possible side effect of Imiquimod. But I have been unable to find any other source confirming that attribution.
(2) I mentioned the oral symptoms because my recent discovery of the gum inflammation behind my molar led me to an article on pericoronitis, which sounded quite like what I was experiencing (save for the fact that that is typically caused by wisdom teeth, which I no longer have). The article listed lymph node swelling as a symptom of pericoronitis, which led me to wonder whether it could explain at least some of the swelling and soreness I have recently been experiencing in the relevant areas. And, after rinsing with salt water and cleaning that part of my gum more directly for several days, it does seem as though the soreness and jaw-popping has diminished somewhat. But I have no idea if I’m onto anything here, and even if I did have pericoronitis, I’m not sure how much of what’s going on could possibly be explained by it. On the contrary, it seems as though my having pericoronitis might itself be explained by the low neutrophils insofar as low neutrophils could mean greater vulnerability to infections.
I have also wondered whether there’s any chance the submandibular swelling, conjoined with the mucoceles and the jaw-popping, might indicate some sort of salivary gland infection. But I don’t seem to have other symptoms of this, e.g. dry mouth or pus or anything like that.
(3) I am not really sure how to confirm spleen enlargement. Would the chest x-ray my doctor ordered reveal such a thing, or should I reach out to him after his office re-opens and see if he’d be willing to update that test to a CT scan, or add an ultrasound?
(4) I assume Mono can cause high monocytes, but can Mono also cause decreased neutrophils?
(5) Suppose I really did have Mono back in September. Could it really have taken this long for Mono to cause spleen enlargement?
(6) As I’m sure must be obvious by now, this whole series of events has been extraordinarily stressful. It has seemed plausible to me at various points that stress might be playing a role in things. As perhaps the most compelling example of this, I mentioned above that I have been night-sweat free since the end of October with one exception. The one exception was in early December, and I remember vividly that the day was the first day in awhile that I became extremely concerned that I might actually have CMML. The extreme concern arose the moment I learned that I had low neutrophils, and that CMML can cause high monocytes and low counts of other white blood cells. I remained very distressed for the rest of that day and the next, and on the second night I had non-drenching night sweats. So I feel fairly confident that this instance of night sweats was stress-induced, but it’s unclear to me whether the takeaway should be that stress was the only factor in play at the time, or rather that it was merely an exacerbating agent to some other factor (e.g. Mono, or whatever). Additionally, I wonder whether stress could be partly responsible for the high monocytes. And it seems obvious that stress could be exacerbating the blood pressure, though I wonder to what extent that
If you've made it this far, thank you - I sincerely appreciate your time and consideration.
submitted by PerspectiveOwn9988 to AskDocs [link] [comments]


2022.10.25 13:18 Special-Poem-6163 Smoking cigs with a perforated broken throat where's the smoke going.

Age 48
Sex m
Height 6
Weight 150
Race white
Duration of complaint since 2016 worse present progressive.
Location Ontario
Any existing relevant medical issues lots
Current medications Diclofenac melatonin
Include a photo if relevant
There alot history so please don't bash me. History of Lyme bartonellosis hemoragging neck mass. Phyrygeal dysphagia w aspiration Barrett's esophagus, got worse by covid sars in April. Brought back old symptoms of Lyme bartonellosis. Lose of collagen burning sticky waxy skin with subcutaneous fluid filled tissues bones spinal cord compression stiffness cracked ng brittle bones. Can't speak chew swallow cough clear breathe. Drooling choking regurgitation best nging up acid saliva thick no mucus production. Cauliflower or cellulitis ear swollen red turned hard with tonsil abcess submandibular abcess loss breathing muscles cramping shrinking neck trunk obliques diaphragm Heavy limp belly with lumps everywhere chemical burns across lower ribs sternum down center of abdomen along esophagus stomach bladder radiates into bladder growing hips knees feet numbness burning pressure and pain balls of feet vascular pain chills sweating. Liver lesion thyroid neoplasm follicular. Facial cavity is atrophied with lumps unders cheeks now. Can't stand straight. Abcess ruptured on right forearm. Cramping shrinking tendons ligaments in limbs .swollen burning fishing lips crusting nose skin face thinning and red ear eyes with bacterial sores smoking making everything break down. Never like this before. There's alot more I'm basically walking vegtable jumping around rattle from throat body try to cough and clear breath at same time from shrinking and breaking down of tissues bones and nerves. Belly pulls down on everything.smoking seems to be coming out of skin and causing apoptosis to my internals all connective tissues. Can't touch my tongue tonsils gums and upper neck abcess with my swelling squeeze locking up my back day. Will Post later. Angry at doctors haven't been any where in a year.
Past Diagnosis: not all info here Other imaging at Chatham hospital and clear water imaging
All this since my infections of Bartonella Lyme and coinfections. I believe my first antibiotic for my gum infections 2016 ,brought out infections already in me terrible side effects as discussed with dr Mallette a while back it's a strong theory. All these finding within we short Time. Do math. Never been to drs my whole life pretty much.
Tons of scans injections gadolinium and contrast. Meds. Part of problem as well
Suspected Tia 2012
Hpncc mutation gene stomach colon Lynch syndrome all males on fathers side.
Enhance flair level of the pons T2 MRI 2013
Chronic Anemia bruising balding bleeding into skin difficulty breathing not anxiety tissues muscles circulation spine etc no further investigations infections, Cancers highly suspected based on incomplete biopsy read what happened? Most probably. Never had until Bartonella. RBC hematocrit hemoglobin at times... Was very low in hospital a while back concerning low no investigations into why. That's y I said my chronic infections bacterial fungal or hidden or denied between thyroid liver neck and Throat something s there. Multi systemic conditions happening at same time same background same triggers.
Lymphadenopathy neck and submandibular armpit and groin. Bartonella infection and mixed zoonotic infections reactive with sars covid and new symptoms from covid debilitating. I'm a long hauler now.
Updated I believe I have a strong case of cellulitis systemically you really see it in my right ear affecting all my skin mucosal membranes muscles bones Spine skull blood vessels lymphnodes lymphatic system for sure. Meds and cigs have been complicating more severely after sars covid, especially with broken throat and zoonotic infection past and present every inch of my body. Speaking chewing swallowing breathing choking regurgitation bringing up acid saliva thick mucus. Read symptoms I'm doing well the rattle in throat submandibular is for sure abcess and possibly cellulitis with all other stuff. Eating and deterioration scarring if my innereds mucus membranes skin facial cavity bone marrow all that's stuff connective tissues spine summary.
Bartonella Lyme considered autoimmune infections due to the immunomodulatory effects. They also infiltrate the epithelial cells blood vessels lymphnode proliferation of erythrocytes, live in non accessible compartments outside the immune system, they attack intracellular compromising immune. Also shape shift to anything to avoid immune. The term stealth vectors, is used for this infections. Hard to detect later through blood work especially if immune is low. Laryngeal epithelial proliferation, human umbilical vein, bacillary angiomatosis. Liver pretty much everywhere. Lyme Borrelia OSP C binds to complement C4b lives in the mid gut and everywhere. My labs showed osp c 23 41and 39 I believe.
Scope of stomach original 2017 found rare pylori like organisms. Rare turned out to haemophimus bartonellosis. No proper antibiotics given no investigations of rare pylori like organisms done except by me and dr Quan was advised and agreed. Whole situation could of been totally avoided from start. Condition has turned Chronic with the other co infections to present date on top of everything else going on now.
Phyrngeal dysphagia w aspiration as per hilal. Came with Bartonella Lyme and confections. True neuromuscular disease as he told me. Undiagnosed. Just like connective tissue degenerative changes.
Orthostatic: hypotension, Bartonella Lyme coinfections
Evolving abcess left neck nothing done/again most probable bacterial infection like everything else. 2020 19? Or cancer
Left neck mass and submental masses submandibular, ultra sound 2020 routine thyroid scan of thyroid follicular neoplasm tirade 4 catagory
Left right inguinal hernias indirect and direct getting worse from spine compression. Lumbar facet joint injections L5s1 was wrong type of injection
Tonsilar inflammatory/infectious from CT scan from dr long 2019 no treatments Salivary gland masses and infections 2019 no treatments Abdullah.
Significant ostiomeadal narrowing no info, promenace of the Palestine tonsils infectious inflammatory. No follow up or treatment. July 2019
Gallbladder polys
Hemmoriging neck mass large us shows infection most probable and growing fast 5 x6x8cmv or SCCarcinoma. Biopsy incomplete ruled as inflammatory cause core sample couldn't be taken as contents were drain 2 weeks prior. Poor diagnosis totally wrong. Still stands in my book infections vs SCC as per MRI and ultrasound and high NC ratios and other terms mentioned through biopsies. Core biopsy should have been first not last. Infections cancer are inflammatory. Based on all my symptoms somethings wrong here. Mass was there long before I started moving thyroid cartilage aside slight to swallow now it apparent it completely scarring and narrowed and deteriorated collapsing with esophagus Spine with the shortening of my respiratory muscles and back. Ties into ear eustachian tubes nasal oral throat salivary glands throat esophagus fried dried burning etc. Stomach pain vomitting nausea swelling pain n above naval into upper esophagus. See regurgitation choking swallowing.
Candida like symptoms more dibilatating
connective tissue degenerative wasting secondary causes by chronic bacterial fungal covid infection meds smoking exacerbating causing deteriorating and severe loss of collagen with pigmentation changes brownish painful with subcutaneous lumps. Meds depleting moisture and necessary fluids from glands mucus saliva thick or none and bones.
Chronic ear infections eye infections skin infections sinus tonsils salivary glands throat and now esophagus stomach no treatment and I'm sure my bones joints and spine possibly brain based on symptoms, with Severe inflammation.
Nasal hands telangiectasia, erythromomelanosis coli face and neck, and pressure uticaria vs uticaria vasculitis hands. 2018 Dr Tan, most probable Lyme bartonellosis
Tons CT csans radiation, great for my immune and tissues bones nerve function. Low IgG subclass1 mildly always Low cd 8+ t cells flow cytometry 2019 no follow up, dr zanganah. Another clue hint immune infections meds have copies of everything almost. Severe tonsilar vocal cord asymmetry from either infection or cancer no follow up just like all other infections lymphnodes or possible Cancer. Scanned CT confirmation 2019 inflammatory infectious. Just like everything thing else.
Costochondritis 2019 most probable Lyme bartonellosis. Common findings " mean Lyme my st probable Bartonella Pleural effusion 2016, 17 and now " Chronic knees effusions 2018 2022 " Otitis media 2019 with effusion 2022 " Palmer erythema 2016 2022 worsening " Pericardial effusion 2016 ,17 Bartonella. " infection most probable. No follow up no treatments. No other reasons but infection. Tricuspid regurgitation, enlarged left sided heart valve
Low manganese through 24hr urine blood Zinc Vit d
Diptheria shot one year after deep cut for finger had surgery with no vaccine at time. Things got worse after shot dr zanganah. Eustachian tube disfunction burning inflammation before. Worse now will mucosal membranes affecting ear lobes stiff and bear with sores. He felt something going on with CNS and immune connective. Eustachian tube disfunction was starting up. This after my zoonotic infections a year after or so with my jaw joint ating up with spine and other stuff with gastric and throat and muscles of breathing.
Severe chronic sinus infections strong burn ing odor green to white to clear yet tissues are dried out burning into eyes ears mouth throat salivary glands lymphatic and esophagus massive hernias lumps severe reflux and swelling like achalasia like symptoms regurgitation choking vommit acide cough blood nasal discharge burning drooling uncontrollable.
Polyneuropathy Dr Charron 2018?
Tandem gait ataxia 2018 Dr mustafa
Barretts esophagus stomach polyps Dr Tarabain dialtion months ago made worse esophageal strictures worsening 2022 now Thyroid follicular neoplasm class 4 2016 Tons of liver lesions, suspected hemangiomas or carcinoid metastatic or other unusual pathologies as per Dr Ala not 100 percent accurate. None prior month on MRI w/contrast. Bartonellosis Lyme Most probable, cancer? Perhaps
Pancreatic cysts head and tail no follow up Kidneys cysts, colloid, bosniak level 3 and masses no follow up Possible scarring damage diaphram abdomen wall. Ultra sound tech asked if I had past surgery in that area while scanning 2 years back clear imaging. No notes made
Neuro muscular motor neuron disease Dr Tawil Chatham swallowing issues from scope before hilals official diagnosis Neuro muscular condition Dr hilal from phyryngeal dysphagia. Bartonellosis Lyme coinfections.
Phyrngeal dysphagia with aspiration Hemmoriging neck mass large us shows infection most probable and growing fast 5 x6x8cmv or SCC Neuro muscular condition pharyngeal muscles and other muscles seen Dr Hilal True neuro in origin as per his quoted words when I asked him. immuno neuro inflammatory response mediated , affecting all connective tissues bones joint spine and nerves, infections bacterial fungal viruses then covid sars made everything go to hell. Meds toxins cigs secondarily worsening condition. Dr Mallette no sure acknowledged symptoms. Not the latest updates. She agrees there is definitely something going on.
Follicular neoplasm thyroid 2016 Moderate xerostomia dr staple ford 2018 possible lichen planus dr staple ford. Salivary glands infections and masses no treatment 2018 with complex masses in submandibular region. No treatments or not investigations
Very High cadmium levels urine 2019? Very High antimony metals urine blood 2019?
2012 Vasomotor rhinitis non allergic rhinitis. Severe allergy like symptoms: meds smoking infections for sure. Chronic sinus air way inflammation infections.
Pneumoniae once In awhile Gynecomastia 2014 1x1x1cm Low testosterone blood Dr Banner 2019
Chronic reactive lymphnodes large everywhere. Bartonellosis Lyme cancer?
Bone tumors swelling ribs clavicle left rib near thymus swollen lymphnodes neck above clavicle upper chest Wall near throat. Hips pelvic femur shoulders very painful. All bones joints spine numbness tingling loss sensation ice picks pulsating.
Dermatographism as per dr Aktas started with Bartonella infection/Lyme hga. Came back after covid struck as well other infections bacteria fungi inside outside body with facial droop right sided mouth.
High cd4cd8 ratio most probable infections or cancer based of on biopsy undetermined atypical neck mass or my thyroid neoplasm liver lesions tons of them. Low IgG subclass1 mildly always Low cd 8+ t cells flow cytometry auto immune immune based obviously. Manganese deficiency low via urine blood
Thyroid always on low end .30 avg all most subclinical hyperthyroidism at times Thyroglobulin very high 89 avg Thyroid neoplasm follicular 2016
Always low Vitamin D all year around avg 20 Low IGM Low Cd57 NK cells
Myopathy biopsy 2018 as according to notes on addem from quad biopsy shamisa as the most probable based on addendum not original. It originally said myelopathy which I have with all the stenosis no follow up with the stress fractures, as well confirmed his colleague. Said there's no such thing from biospy it should read myopathy end his quote. could this be an infectious myopathy. Which one is it. No follow up with Dr Mustafa. Lyme bartonellosis? Was told had myelopathy in the past
Chronic skin bacterial fungal infections Tetanus like symptoms and connective tissue type symptoms secondary 95percent if I can't speak well and breath swallow and move funny rattle from hyoid Bone. See other notes. Pinna perichondritis or cellulitis at this point came with Bartonella, covid made worse getting worse systemically.
Cervical and Thoracic lumbar changes subluxation hypertrophy Multi level central stenosis c3c4 c567 c3,4 being worst etc worsening now c1 and2 subluxation guaranteed Grade 3 or worse old report. Spondylolisthesis Causing diaphragm and accessory muscles paralysis from neck down. With a collapsed atrophied infected throat and esophagus. No follow up. Now completely paralysing me. Severe weak cough due to breathing muscles paralyzing neck downward and trachea is cracked and broken at esophagus collapsed major esophageal strictures or cancer infection hernia into belly into bladder groin. Collagen loss not helping Radiculopathy history cervical spine from fall. Bending spine thoracic most visible. Lumbar severe dissection hyperlordosis multilevel stenosis severe bulging and other stuff. old data. Spine is now stiff cracking drying out burning cracking compression causing Major issues in body. Severe bend out ward in spine as a whole. Skull base to tailbone. vertebrates all cracking and crunching with sharp shooting burning pains all the way down from skull base cervical thoracic felt in throat jaws esophagus into lumbar from other muscles, tissues collagen, mucosal membranes breaking down. Chronic gastritis drooling choking regurgitation vomitting acid saliva weak unproductive cough from muscle and Throat. Loss of bladder control numbness tingling and loss sensation of small toes neck limbs vascular pain inflammation phlebitis starting in hand etc. Lumb weakness cramping of legs tendon ligaments muscles with left arm and neck scm muscle bulging back muscles with back neck.
White matter changes brain Chatham hospital. Nothing 1 year prior. Happened after Bartonella 2016/ Lyme anaplasmosis infection 2018 which was all reactive 2years later and again after covid April 2022 Agiogram brain narrowed mca M2 segment left side from either trauma or other forms of vasculopathy infection.
Tarlov spinal cysts
Bleeding pus filled leaking from older neck biopsy from dr Gill under chin. Happened 6 months back from July 2018 no treatment for bacterial infection no treatments IgG subclass deficiency 1&2
Tons of liver suspected hemangiomas 2018 no biopsy. Liver pain present day Infectious inflammatory dystrophic calcification palatine tonsils CT scan 2019 dr ling no treatment. Still active Salivary glands infection no treatments 2019 still active
Tons of CT scans antibiotics for bacterial and Fungal infections 6 in 1 year. Nystatin make my body go wild. Last antibiotic cefuroxime made things worse for suspected nasal infection. Viral? Chronic since med use.
Past Barrett's esophagus. Recent Dilation from scope after made things worse. 3 weeks of stomatitis severe. Current severe gastritis coughing up blood thick chunky with esophagus or trachea from old break and scarring eroding narrowed stricture?. From meds infection cigs. No follow up on Thoracic muscles to re breathing. Respiratory muscles cramping shortening failing and shrinking. Pulling downward. Affecting neck shoulders chest and ribcage expansion back muscles. Inflammation of those tissues causing legs to buckle
No follow up on small hole found in throat from barium swallow. No follow up fractured thyroid cartilage last year. No one knows about completely cracked and perforation of the trachea from hyoid bone with infection or tumor scarring into tongue gums tonsils throat esophagus no one knows except dr Mallette.
Suspicious possible paraneoplastic syndromes which type. Mucosal skin spinal cord bones joints tendons muscles all affected, liver kidneys Throat stomach
High hgfactor 9.0 2018 Shoulder and blades tenosynovitis Possible portal hypertension ascites Umbilical hernia small Liver pain swelling dull sharp stiffness Servere herniation gastritis Esophageal erosion into Throat with infection or possible Cancer. See notes for symptoms. Heavy swollen limp belly pain and lumps under skin multiple. Server chronic gastric swelling burning massive lumps reflux and inflammation Chronic nasal narrowed tonsilar salivary gland lymph nodes swelling burning Chronic infections. Reactive lymphnodes swelling with pain and without chronically neck collar bone under jaw arm pits groin. Possible belly lumps under skin most noticeable nowadays Ear infections Chronic painful drooling can't move speak swallow cough clear Very much at mouth going numbness pain tingling burning fried Bone joint effusions. Spinal cord hypertrophy and subluxation multilevel central stenosis and dissection with spinal crepitus with serve pain amount other concerning findings.
Occipatal nerve pain radiation into neck SCM muscle swollen down into not belly into pelvis groin knees ankles feet with bladder loss and loss of sensation in small toes with burning Chronic swollen lymph nodes neck collar bones armpits groin other areas
Carb and sugars intolerance, grains Enzyme deficiency?
Infections past history: Stomatitis mucositis recent dr aktas Bartonella anaplasmosis highly suspected Lyme disease igx testing CLIA certified lab, fungal and bacterial skin and mouth constant bleeding and fluid filled blisters clear and sticky liKe lips and nose and skin when smoking.
lichens planus as per dr staple ford 2016. This I believe prednisone meds smoking makes worse. Infections and now covid positive april24.
Severe periodontal disease with chronic infections dr Nagle 2016. Severe bleeding gums infections all teeth cracked nerve roots exposed tightening cannot use gum tools to clean anymore my whole body ballistic from infections and pain just tongue tonsils salivary glands upper throat ears into esophagus out into my entire system. Systemic.
Dr Banner London.Dysautonomia, weak immune. Response overload zoonotic infections metal toxicity high
Dr Baker. London Lyme Bartonella rickesii and other zoonotic type infections.
Dr lemmo Windsor. Neurotoxicity autoimmunity or pathogen or a combination thereof all brain, CNS pons medulla mid brain right frontal lobe issues breathing swallowing Shrinkage of belly rectus ab wall diaphram breathing muscles not being used from brain.
Antibiotics: Flagyl tooth abcess still there never removed all teeth deterioration now bleeding Pennicylin bad reaction quad biopsy caused mouth droop and drooling most probable from Lyme and Bartonella Azithromicin suspected sinusitis Amoxicillin sinus infection Doxycycline destroyed me Bartonella Lyme Ciprofloxacin skin internal infection suspected staph for bacterial skin lesions on arms scalp legs shins. It worked but keeps coming back. Ceftriaxone suspected sinus throat stomach esophagus infection. Last Anti destroyed me.
Topical Bacterial and fungal chronic use. Keeps coming back bartonellosis Lyme, crackes perforated throat in throat thinning of veins?
Ssri history never agreed with not only he issue all gave serotonin syndrome. Again not the issue here celexa Zoloft. Made worse with Bartonella Lyme coinfections. Lorazapam for single muscle from infection 2.5 years ago with tramadol famotine. Can't get off pills. Just use for immune response. But making tissues and other things worse. Take 3 halves .05 every 6 hours. Body's used to it interacting with cigs see rest of notes.
All related to bacterial fungal infections all started with Bartonella Lyme, babesia anaplasma coinfections and most likely Covid sars worsening . Haven't stopped all.
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2022.06.29 02:39 machenkaam Lymph nodes in neck swollen for 2 months.

Hi i am 27 years old i am a female from canada
Light Smoker Dont drink No known health issues
Begining of may i noticed a large lump on side of my neck. Didnt think much of it. It is clearly visible and does not hurt and the other side of.my neck has a swollen lymph node as well but its not as big but a bit more tender then the large one. Neither are painful but my neck does get stiff and feel tense like its being stretched..
Went to hospital because of my lymph node not going down and i was experiencing fatigue,shortness of breath(has gotten a bit better) ice pick headaches, tinitus dizziness which im still having and wasnt feeling right mentally.
Had blood work and ultrasound done which i will put results below. They said everything was normal and sent me home nd said a specialist were to call me about my lymph nodes. The ER Doctor looked in my throat and there is no infection or anything abnormal. I however would like to share my reults here and have some other opinions.i do not have a family doctor either.
ULTRASOUND RESULTS
4 x 1.9 x 1.9 cm lymph node on left side, and level 2A lymph node is seen measuring 3.6 x 1.9 x 0.8. It is not getting any better.
There are several prominant lymph nodes in the right neck largest is 3.2 x 1.5 x 1cm
The partoid and submental gland on left side were assesed and seemed nornal.
No abcess was found, enlarged nodes are presumed reactive and clinical follow up is suggested to ensure resolution as an underlying lymphoproliferative disorder is not excluded.
Blood work
HS Troponin <3.0
ELECTROLYTES (ABNORMAL)
SODIUM 137 POTASSIUM 3.7 CHLORIDE 104 TOTAL C02 27 ANION GAP 6
GLUCOSE 4.8 UREA 4.8 CREATIININE 52 EGFR >=90
C-REACTIVE PROTIEN <2.90
CBC(abnormal)
LEUKOCUYTE 9.7
ERYTHROCYTES 4.45
HEMOGLOBIN 123
HEMATROCIT 0.372
MCV 84
MCH 27.7
MCHC 331
RDW 14.4
PLATLETS 479
MPV 7.2
AUTOMATED NUCLEATED RBC 0.1
Please help explain and should i get second opinion
Thank you
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2022.06.24 00:51 machenkaam should i get a second opinion?

Hi i am 26 years old i am a female from canada
Light Smoker Dont drink No known health issues
Begining of may i noticed a large lump on side of my neck. Didnt think much of it. Then had a abcessed tooth early june. Was prescribed amoxocillin
Went to hospital because of mylymph node not going down and i was experiencing fatigue,shortness of breath(has gotten a bit better) ice pick headaches, tinitus and wasnt feeling right mentally. Had blood work and ultrasound which i will put results below. They said everything was normal and sent me home nd said a specialist were to call me about my lymph nodes. I have not gotten a call yet and i want to point out that they are not painful and causs neck tension. Also having sinus issues mostly clear post nasal drip occasionally a bit yellow but do not feel ill or have a headache or congestion.doctor looked in throat and there is no infection or anything abnormal. I however would like to share my reults here and have some other opinions.i do not have a family doctor either.
My oxygen is and was 100% Tempture : 36.8 c
I have a 3.4 x 1.9 x 1.9 cm lymph node on left side, and level 2A lymph node is seen measuring 3.6 x 1.9 x 0.8. It is not getting any better.
There are several prominant lymph nodes in the right neck largest is 3.2 x 1.5 x 1cm
The partoid and submental gland on left side were assesed and seemed nornal.
No abcess was found, enlarged nodes are presumed reactive and clinical follow up is suggested to ensure resolution as an underlying lymphoproliferative disorder is not excluded.
Blood work
HS Troponin <3.0
ELECTROLYTES (ABNORMAL)
SODIUM 137 POTASSIUM 3.7 CHLORIDE 104 TOTAL C02 27 ANION GAP 6
GLUCOSE 4.8 UREA 4.8 CREATIININE 52 EGFR >=90
C-REACTIVE PROTIEN <2.90
CBC(abnormal)
LEUKOCUYTE 9.7
ERYTHROCYTES 4.45
HEMOGLOBIN 123
HEMATROCIT 0.372
MCV 84
MCH 27.7
MCHC 331
RDW 14.4
PLATLETS 479
MPV 7.2
ABSOLUTE 6.7 NEUTROPHILS
ABSOLUTE 2.4 LYMPHOCYTES
ABSOLUTE 0.5 MONOCYTES
ABSOLUTE 0.1 EOSINOPHILS
ABSOLUTE 0.1 BASOPHILS
AUTOMATED NUCLEATED RBC 0.1
Please help explain and should i get second opinion
Thank you
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2022.02.07 01:06 Derkxxx Comparison of protocols and ambulance approach of a similar case between The Netherlands and Chicago Fire Department (blog from a Dutch medic)

Comparison of protocols and ambulance approach of a similar case between The Netherlands and Chicago Fire Department (blog from a Dutch medic)
Just wanted to share a blog I came across of a Dutch medic (ambulance nurse) visiting Chicago Fire Department EMS on a work trip. In the blog, the Dutch medic describes one of the calls he observed during the trip and then made a detailed description of how the situation would have gone in The Netherlands based on the national ambulance protocols here and an actual case. So basically a broad overview of what he saw in the US to a similar case in The Netherlands via the Dutch approach as a simple comparison. This happened in 2015.
A Chicago Fire Department ambulance

Protocols and ambulance approach: US vs NL

6 April 2015 Steven J. Hofman
The case in Chicago
We are just sitting at the table in the firehouse enjoying the lunch the firefighters made when the horn goes again and the computer voice calls out that there is a deployment for ambulance 11 and Engine 42. Paramedic Jeff jumps up and grabs from the cupboard three take-out boxes to take the contents of our plates packed into the ambulance. paramedic John stands in the garage and presses the computer screen to indicate that the notification has been received. a small printer immediately prints out an A4 sheet with the information as it is on the ambulance screen. This system is intended for when the screen in the ambulance gets stuck or does not respond. The address and the call-out information are then known in any case. In downtown Chicago, there is a lady with palpitations. She is conscious and breathing.
We jump into the ambulance, Jeff is behind the wheel and with his right hand he operates the panel which makes all kinds of siren sounds. Since John is the most experienced paramedic, he handles the communication and registration of the deployment. Chicago's street system is clear and numbered, so there is hardly ever a need to use a navigation system. The response time is only a few minutes. Yet at the scene of the incident, there are already three police cars and a fire engine waiting for us with their lights flashing. John gets out of the ambulance and walks towards the patient who is sitting on a chair surrounded by police and firemen. The lady looks a little anxious around but is alert and has a normal skin tone. The EMT sitting on the fire truck does a short handover to John and briefly explains what he found in the primary survey. The patient gets into the ambulance herself and lies down on the stretcher.
John sits at the headboard and Jeff connects a blood pressure monitor to the patient. John asks what happened. The lady states that she has been under a lot of stress lately. She had gone to buy coffee now and started having palpitations with tingling in her arms. It didn't last long, but a passerby saw it happen and called 911 for her. Jeff connected the 4-lead electrodes and took a heart rhythm strip. The blood pressure was 130/80 and the heart rhythm was a sinus rhythm of 88/minute. Oxygen saturation was 100%. The lady anxiously asked what it could have been that made her feel so strange. John replied that he is not a doctor and he could not comment on that. The lady asked if her heart was fine. Jeff explained that he saw no irregularity in the rhythm strip. When asked if her heart had any problems, Jeff could not reassure her. For that, she would have to go to the hospital. The question that now had to be answered by the patient was whether or not she wanted to go to the hospital. The lady asked if that was necessary. Jeff and John both indicated that she should make the decision herself. Preferably a little quickly, as the ambulance was still in the middle of the street, blocking access. The patient was clearly still not reassured and said she feared she had suffered a stroke. Jeff asked her to squeeze both his hands. He also tested the strength in both feet and asked if she would show teeth. The FAST test was negative. The patient was told that this test showed no abnormalities; no statement was made as to what diagnostic significance this then had.
The lady remained anxious and wanted to go to the hospital anyway. Jeff then connected the 12-lead cable and made a heart movie. The Lifepak 15 provided the ECG with a computer diagnostic that said it was a normal ECG with some interference that could affect the data interpretation. Both Jeff and John had had 3 hours of lecture on ECG interpretation and both felt incompetent to review the ECG. I noticed on the ECG that st-elevation was visible in the last complexes in V3-V5. The rest of the complexes in all leads were perfectly according to normal values. Because the isoelectric ECG line was also not straight in those last complexes I saw that it was interference by the movement of the patient that caused the elevations. I was not allowed to make any statements to the patient about that. Jeff inserted a venflon and John gave the lady two chewable tablets of Ascal. He determined blood sugar to be within normal values. John called the telemetry nurse at the hospital while Jeff drove the ambulance with flashing lights and a siren. I noticed that no one in Chicago looks up when an ambulance drives by. An ambulance with flashing lights and a siren is seen as if a city bus is passing by; the most normal thing in the world.
Arriving at the hospital, Jeff grabs a wheelchair. The lady is wheeled inside and taken to the triage nurse. After registration, she may join the crowded waiting room where the waiting time is at least three hours. John fills in all the data on his Panasonic Toughbook where also the measured vital signs of the Lifepak 15 are recorded via BlueTooth. He leaves a printout with the triage nurse.
Chicago Fire Department paramedic
==========
A case with a condition that was slightly more serious, described via the Dutch approach:
Notification:
Not responsive, collapsed in the living room, breathing is present.
On arrival; Patient Assessment Triangle (PAT):
  • Appearance: Lying on back in the living room, anatomically normal position, moaning and with eyes squeezed shut. Moves head from left to right -Work of Breathing: rapid superficial breathing -Circulation to Skin: slightly pale skin color, not clammy. No major bleeding is visible.
Conclusion after PAT: worrisome situation, no scope-and-run scenario
==========
Primary survey according to ABCD method:
  • A: Airway is clear, no loose objects in the mouth, no audible breathing
  • B: Rapid, shallow breathing; 35/minute. Both thoracic halves rise symmetrically. Vesicular breath sounds audible on auscultation left and right.
  • C: Well-filled, regular pulse palpable at the a.radialis, 98/minute. No major bleeding visible, no fractures of long pipe bones visible.
  • D: E2M5V2, pupils PEARL, no signs of meningeal excitation.
==========
Specific anamnesis.
Heteroanamnesis via husband, Mrs. does not respond to being addressed.
After an altercation with her husband, she became increasingly oppressive, losing consciousness after several minutes. She complained of a pressing feeling in her chest with palpitations, which were not present before. She expressed the feeling that she was going to die. The oppressive feeling became worse and tingling developed in both arms. She also complained of tingling around her mouth. Breathing was rapid. According to her husband, at the time of loss of consciousness, she had a pale complexion, and her eyes turned upward. The breathing remained present after the loss of consciousness, the skin color did not change. No twitches were seen on arms or legs. She has not been traveling recently. Medical history:
Blank medication:
  • none
Allergies:
  • none
Diet:
  • Does not eat pork. Last few days low appetite and ate little. The last meal was last night
  • Physical examination
  • General Cared for, a slightly skinny 38-year-old female, appearance older than calendar age.
  • Blood pressure: left 117/75 mmHg; right 112/70. Pulse: 98 /min. Sat O2: 100%, temp 37.1 gr. Celsius tymp. Respiratory rate 35 /min. Weight 68 kg, height 1,60 m.
Head/neck:
  • Eyes: moist conjunctiva, no redness eyelids.
  • Nose: straight, no swellings visible, no pus, mucus, or blood visible. Nostrils are well permeable on both sides.
  • Mouth: lips and corners of mouth clear, cheek mucosa clear, moist mucous membranes, cleaned teeth, no tongue abnormalities, no tongue bite visible, pharyngeal arches symmetrical at rest and on movement
  • Glands Inspection: no swellings visible. No glands palpable: submental,submandibular, pre- and retro-auricular, occipital, anterior and posterior to m.sternocleidomastoidus.
  • Thyroid: no swelling visible, symmetrical shape. Palpation: larynx/thyroid/ring cartilage; symmetrical, no swelling, no palpation pain. No enlargement of the thyroid gland.
Thorax:
  • Inspection: shape symmetrical, no scars and retractions, slightly pale skin color.
  • Palpation: Thorax well mobile and symmetrically retracting on breathing, no throbbing pain vertebrae.
Lungs:
  • Percussion: sonorous percussion across all lung fields, normally mobile lung borders.
  • Auscultation: VAG re=li, no side sounds.
Skin:
  • Inspection: no hematomas, swelling, redness, erythema.
Abdomen:
  • Inspection: slender abdomen, no venous markings. Pants are dry; no leakage of urine
  • Auscultation: sparse peristalsis.
  • Percussion: variable tympanic, lateral liver border thv 9th rib right
  • Palpation: No muscle tension, no défense musculaire. Liver and spleen not palpable. No palpable resistances.
Peripheral vessels:
  • Aa. carotis: pulsations re+/li+, souffles re-/li-
  • Aa. radialis: pulsations re+/li+
  • Aa. femoralis: pulsations re+/li+, souffles re-/li-
  • Aa. tibialis posterior: pulsations re+/li+
  • Aa. dorsalis pedis: pulsations re+/li+
  • No edema or redness on legs
  • Neurological examination
  • Consciousness: decreased consciousness, Glasgow Coma Scale: E2M5V2 (opens eyes on pain stimulus, On supraorbital pain stimulus moves both hands to the site of pain stimulus, speaks unintelligibly, mumbles)
  • No signs of meningeal stimulation: no reflective resistance when bending head in lying position and does not raise legs in the process, no neck stiffness.
Brain Nerves:
  • N.I Smell: not tested
  • N. II Vision: not tested
  • N. III, IV, and VI: Pupils: isocuous, pupil response to light direct and indirect intact. No nystagmus.
  • N.V. Facial sensibility: Corneal reflex intact.
  • N.VII Facial motor skills: corner of the mouth on left slightly lower than right, cannot be instructed to puff up the cheeks or puff up the lips
  • N. VIII Hearing: not tested
  • N. IX and X not tested
  • N. XI not tested
  • N. XII not tested
  • Motor skills: locates with both arms on supra-orbital pain stimulus
  • Sensibility not tested
Summary:
A 38-year-old female with blank history. Now experiencing a sudden drop in consciousness with dyspnea and chest pain after a severe emotional event. Vital signs are within normal range except for a respiratory rate of 35 per minute. The left corner of the mouth appears to be drooping.
==========
Differential diagnoses.
1 Panic attack
DSM-IV criteria panic attack
A circumscribed period of intense fear or feeling of unease, in which four (or more) of the following symptoms occur suddenly, reaching a maximum within ten minutes:
  1. Palpitations, pounding heart, or accelerated heart action
  2. Sweating
  3. Shaking or trembling
  4. The feeling of breathlessness or suffocation
  5. Gasping for breath
  6. Chest pain or discomfort
  7. Nausea or abdominal discomfort
  8. The feeling of dizziness, unsteadiness, lightheadedness, or fainting
  9. Derealization (feeling of unreality) or depersonalization (feeling detached from oneself)
  10. Fear of dying
  11. Paresthesias (numbness or tingling sensations)
  12. Hot flashes or chills Until a few years ago, when these symptoms were encountered, they were referred to as "hyperventilation syndrome." The assumption was that improper breathing caused an imbalance of carbon dioxide/oxygen in the blood, thus causing the symptoms. With the disappearance of scientific evidence for the underlying pathogenesis of hyperventilation syndrome, the diagnosis of "panic attack" has come more to the forefront. However, hyperventilating is often present during a panic attack as an accompanying symptom. Due to the lowered CO2 level in the blood, sensations such as tingling in fingers, hands, and lips occur. This often includes pain or a feeling of pressure in the chest; in extreme cases, cramps of the fingers in an extended position.
Argues for the diagnosis:
  • clear triggering factor
  • Symptoms occur within 10 minutes
  • Palpitations
  • Perspiration
  • The feeling of breathlessness or suffocation
  • Pain or discomfort in the chest
  • Feeling of dizziness
  • Fear of death
  • Paresthesias
2 Pulmonary embolism
A pulmonary embolism is a blockage of a pulmonary artery. It is usually caused by a blood clot (thrombus). The blood clot usually forms in the blood vessels of the legs or pelvis. Through the bloodstream, the blood clot is carried to the lungs.
Arguments for diagnosis include:
  • acute onset of symptoms
  • Occurrence of palpitations
  • increased heart rate
  • accelerated breathing
  • feeling of tightness
Argues against the diagnosis:
  • no pain associated with breathing
  • no coughing or hemoptysis
  • no sweating
  • no suffering from calf pain, redness, or swelling of the lower leg
  • Did not sit still for long periods of time during the past few days
  • does not use birth control pills
  • Does not smoke
3 CVA/TIA
Both ischemia and hemorrhage lead to acute focal neurological deficits. In addition, as a result of edema formation and mass effect of a hemorrhage, increased intracranial pressure may occur, giving rise to a loss of consciousness. The occurrence of this depends on the size and localization of the lesion.
For the diagnosis advocates:
  • decreased consciousness
  • at first sight, a drooping corner of the mouth
Argues against the diagnosis:
  • moves both arms symmetrically in response to a pain stimulus
  • Does not use blood thinners
  • no evidence of trauma to the head.
  • young age
4 Acute coronary syndrome
Acute coronary syndrome (ACS) includes acute myocardial infarction (AMI) and unstable angina pectoris (IAP) with symptoms at rest. Myocardial infarction is usually caused by a blood clot in a coronary artery that cuts off the supply of blood, which was already low, now completely. This can lead to heart failure, death, or life-threatening arrhythmias. If the myocardium does not receive enough oxygen and nutrition when a coronary artery is narrowed, chest pain can occur: angina pectoris.
For diagnosis advocates:
  • oppressive chest pain,
  • pale complexion
  • dyspnea
  • dizziness.
Argues against the diagnosis:
  • Had no vegetative symptoms such as sweating, nausea, and vomiting.
  • Never had chest pain before.
  • No comorbidities such as diabetes present
5 Hypoglycemia
Hypoglycemia is an excessively low blood sugar accompanied by typical symptoms such as a feeling of hunger, trembling, tingling in the hands, feet, or lips, sweating, headache, blurred vision, and palpitations. Signs of severely lowered blood sugar are drowsiness, loss of consciousness, and eventually coma
Pre-diagnosis advocates:
  • Eaten poorly in recent days
  • Had increased glucose demand due to stress moment
  • palpitations
  • dizziness
  • tingling in hands, feet, or lips
Against:
  • not familiar with diabetes
  • complaints arose within 10 minutes
  • no headache as an omen
  • These symptoms occur almost exclusively in diabetic patients who have injected too much insulin

Additional diagnostics
12-lead ECG:
  • The purpose of taking an ECG is: observation and early recognition of cardiac arrhythmias, conduction disturbances, and ischemic heart disease that could cause a drop in consciousness in this case.
  • A meta-analysis of 11 studies describes the testing properties of the ECG made outside the hospital in patients with chest pain. For the diagnosis of AMI (n = 4481), the sensitivity was 76% (95%-BI 54 to 89) and the specificity 88% (95%-BI 67 to 96). For the diagnosis of ACS (n = 4311), these figures were 68% (95%-BI 59 to 76) and 97% (95%- BI 89 to 92), respectively.
  • A sensitivity of 97% to exclude 97% is high but not yet foolproof.
  • When assessing the ECG, it is important to work systematically and to include the clinical presentation of the patient in the final working diagnosis.
  • At the time the ECG was made, the heart rate had already dropped to 65/minute.
A systematic review of the ECG:
1: Heart rate is a sinus rhythm.
Features of normal sinus rhythm:
  • A p top (atrial contraction) precedes the QRS complex
  • Each p wave is followed by a QRS complex
  • The rhythm is regular but varies slightly with respiration
  • The frequency is between 60 and 100/minute.
  • The maximum height of the p Summit is 2.5 mm in II and/or III
  • The p top is positive in II and aVF, and biphasic in V1
----------
2: Heart rate is 65 beats per minute
----------
3: Conduction times:
PQ time: 0.12 seconds (Normal PQ time is between 0.12 and 0.20 seconds)
QRS time: 0.08 seconds (The ventricles normally depolarize within 0.10 seconds. If they take 0.12 seconds or longer, there is a conduction delay)
QT time: 0.38 seconds (The QT time indicates how long it takes for the ventricles to repolarize. The normal value for the QT time is less than 0.45 sec for men and less than 0.46 sec for women)
----------
4: Cardiac axis: Normal cardiac axis
Positive QRS deflection in lead I: the electrical activity points to the left (for the patient)
Positive QRS deflection in lead aVF: the electrical activity points downward.
A normal cardiac axis lies between -30 and +90 degrees.
----------
5: p-top morphology:
Height: 1mm, width 8mm, positive in II and aVF, biphasic in V1. The p-top is normal
Characteristics of a normal p top:
  • The maximum height of the p top is 2.5 mm in II and/or III
  • The width of the p top is normally less than 0.12 second
  • The p top is positive in II and aVF, and biphasic in V1.
----------
6: QRS morphology:
The width of the QRS complex is 0.08 sec, there is no intraventricular conduction disorder. Q waves of 0.01 sec are visible in II, III, and aVF. However, these do not meet the criteria for pathological Q. A pathological Q should be 0.03 sec wide, in this case, there is a physiological q-wave.
----------
7: ST morphology:
The ST segment is isoelectric in all leads.
The ST segment represents ventricular repolarization: the cardiomyocytes are getting ready for the next cardiac action. It extends from the end of the QRS complex to the beginning of the T wave. This segment is normally isoelectric (at the baseline level).
The T tops are of the same polarity as the QRS complexes. A normal T wave top is congruent with the QRS complex and about 1/8th - 1/3rd as high as the QRS complex. Thus, there are no T wave top abnormalities here
The ECG of Mrs. Abdullah shows no abnormalities indicative of ischemia or ACS

Glucose measurement
Glucose levels are determined by finger prick and point-of-care glucose meter. Ms. Abdullah's blood glucose level is 5.7 mmol/l. Hypoglycemia is said to occur when the glucose level is 3.5 or lower. Glucose higher than 14 mmol/l is said to be hyperglycemia. In this case, the blood sugar is not abnormal and cannot be seen as a cause of the drop in consciousness.

End-tidal CO2 measurement
Using the LifePak 12 monitor as used by the Hollands Midden Regional Ambulance Service, it is possible to perform an end-tidal CO2 (ETCO2) measurement. The connector that is normally placed on the endotracheal tube can be placed between the lips of the patient. In this way, a capnogram is seen measuring the ETCO2 content in the exhaled air.
The first measured ETCO2 in Ms. Abdullah is 22 mmHg.
The normal value is between 35 and 45 mmHg.
The low ETCO2 value in combination with the high respiratory rate confirms the suspicion of hyperventilation without the need for metabolic compensation. If there were a normal or elevated ETCO2 value with this increased respiratory rate, there would be metabolic acidosis. When, for example, due to a severe kidney problem or abnormal blood sugar, the blood is too acidic (low pH), the lungs will try to excrete more CO2. This is because CO2 lowers the pH of the blood. By excreting more of this, the pH of the blood will increase and normalize.

Working diagnosis
Panic attack with hyperventilation. The heteroanamnesis in which the argument preceding the collapse was mentioned and the clinical picture at the time of finding give a high suspicion of a panic attack.
The anamnesis and physical examination found no evidence for a pathological/somatic explanation of the shortness of breath and the collapses. The measurement of the ETCO2 confirmed the low CO2 level which could cause somatic complaints.
The additional examination by ECG indicated no indication of cardiac problems at this time. The normal blood glucose level excludes hypoglycemia at this time. The (limited) neurological examination provides sufficient evidence at this time that no major cerebral ischemia is present. The at first glance drooping corner of the mouth cannot be properly objectified
Age is also an important predictor of the final diagnosis. In the age group 25 to 44 years, the final diagnosis of all patients presenting to the general practitioner with shortness of breath is respiratory in nature in 54%. In 13% of them it is a panic disorder and in almost none of them is there a cardiac cause.
To make the working diagnosis of a panic attack in this acute situation, therapy will have to be started to see if the symptoms disappear quickly. According to the LPA8 (national EMS protocol), the symptoms should resolve within 15 minutes of the arrival of the ambulance, otherwise, underlying pathology should still be considered. In the prehospital situation, no additional diagnostics are available to objectify this acute situation.

Prognosis
The prognosis of treatment for a panic attack is generally favorable. If the patient follows the instructions properly, the symptoms may disappear within 15 minutes.
A panic attack can fit within a panic disorder: recurrent, unexpected panic attacks. In older patients, there is a greater risk of a somatic condition explaining the symptoms, while the likelihood of a first anxiety/panic attack is lower in later life. Stay alert even in older patients with a known panic disorder: a new somatic condition may develop. When having an anxiety/panic attack, it is important that the attack is self-limiting. Research shows rebreathing has no added value compared to a calm approach with careful attention to the patient's symptoms.

Treatment plan
Most important starting point: if the symptoms have not completely disappeared within 15 minutes, then transport and transfer for further examination in the emergency room is necessary.
  • A calm approach is essential in the treatment of a panic attack. Provide a low-stimulus environment: refer the family to another room, turn off radio and TV.
  • Provide information about the origin of the symptoms: Anxiety enables the body to react by activation of the autonomic nervous system. The body is primed for a 'fight or flight' response, with accelerated heart rate and breathing, increased muscle tension, and sometimes panic.
  • breathing exercises: The patient is well advised to breathe calmly, breathing in for three seconds each time and then breathing out for six seconds, counting in their thoughts. The end-tidal CO2 measurement can be used for this purpose. It encourages patients to breathe so calmly that the end-tidal CO2 level rises in the capnogram
  • explain the vicious circle according to Clark, in which fear causes physical sensations that reinforce the fear of a somatic abnormality.
  • After normalization of respiratory rate and decrease in symptoms (within 15 minutes) perform the complete neurological examination and tract anamnesis to exclude other causes.
After the family was sent out of the room, Ms. Abdullah was approached calmly yet directly. She was summoned to breathe calmly according to the technique described above. She had to be corrected regularly for this because she kept getting tachypneic whereby she let herself fall back into the panic attack, crying and moaning. After a few minutes, she reacted more adequately and dialogue could be started. In the conversation that followed, she indicated that she was suffering from tensions around housing and her financial situation. Within 15 minutes, the symptoms had subsided to the point that a calm conversation was possible. The complaints have now completely disappeared and she agrees with the decision to stay at home and to look at the problems and possible solutions together with the GP.
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2022.02.05 20:26 Dapper-Bluebird2927 2 Different interpretations of CT scan. Confused.

Female 53 Non Smoker. Lynch Syndrome. No Cancer. Healthy. Dad died from Lung cancer a few months ago. His brother had lung cancer. Nodules removed. Is doing well.
I am a carrier for Anti Trypsin 1 deficiency.
I originally went in about a month ago to doctor to have an x-ray done because my clavicle seemed enlarged. After X-ray and a neck ultrasound they still couldn't figure out 100% if the enlargement was bone related or something else. A Few days ago I went to have a ct scan, and the radiologist that read my x-ray and ultrasound also read this image also. He stated I had a lymph nodes and so on.
Then a different radiologist stated something else. Both Radiologists from the same hospital. I'm really confused. The prominent clavicle issue is all set. Apparently I have scoliosis and that's why my collarbone is prominent. But the other issues I'm concerned with. Do you have any suggestions? Should I have another hospital read these? First reading: DIAGNOSTIC IMAGING REPORT
Some nonspecific lymphadenopathy identified in the mediastinum. No enlarged masses. A nonspecific supraclavicular lymph node on the right is identified. This is seen medially and just anterior to the right lobe of the thyroid. This measures 14 mm. This is identified on the right. Small submental lymph node measuring less than 5 mm is noted off to the left.
IMPRESSION: Prominence of the distal margin of the clavicles with degenerative spurring noted. Changes of what appear to be a slightly prominent right supraclavicular lymph node and a smaller lymph node on the left as well. Suggest further evaluation at some point with ultrasound and attention to the supraclavicular region to evaluate these lymph nodes and determine if they resolve. The one on the right is the largest at 1.36 cm, Small nonspecific mediastinal Iymph nodes are identified also.
The next interpretation:
ADDENDUM: Upon review of the CT examination it would appear that what is described as a right supraclavicular lymph node in actuality represents a seqment of asymmetrically prominent right internal jugular vein. Additionally, the patient has a mild proximal thoracic scoliotic curvature which most likely causes slight asymmetry in the thoracic cage whereby the right sternoclavicular joint is slightly more anterior than that of the left which is suspected to result in a probable mild asymmetric palpable abnormality. These findings were discussed with Dr.
Service Date: 02/03/22 Order#(s): 0203-0008 Reason: R22.2 LOCALIZED SWELLING, MASS AND LUMP, TRUNK 067 8 OTHER CONGENITAL DEFORMITIES OF CHEST CLINICAL HISTORY: Swelling, mass/lump in the trunk, deformity of the chest. PROCEDURE: Multiple axial images obtained without contrast enhancement. Coronal and sagittal reformatted view submitted.
AXILLARY REGIONS: Unromarkable. THORACIC AORTA: Unremarkable. PULMONARY OUTFLOW TRACT: Unremarkable. HEART/PERICARDIUM: Normal. LUNG PARENCHYMA: A few small nonspecific pulmonary nodules measuring less than 3 mm identified in both lung bases. Bony prominence of the distal margin of both clavicles, left slightly greater than right, There are no masses. No definite large soft tissue abnormality. Thyroid gland is intact. No mediastinal adenopathy. LIMITED UPPER ABDOMEN: No acute abnormality.
Please help. Thank you.
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2021.10.10 05:49 teetertotter56 Kimuras disease-from original lymphoma ddx

Hi, so im 20m, 6’ 2, 175. From Canada and i was just diagnosed w this extremely rare disease. Dx was from a excisional biopsy on one of my submental nodes that was about 2.5cm nd round. What im wondering is that how sure can the excisional be? I was pretty shocked to hear as i expecting lymphoma from what i was told for months nd multiple core biopsies. This disease is uncureable, and frankly even my ent had never heard of it. It lines up as the only information online shows early kidney issues and eventually disease as a common complication and i have some protein passing already. Total 5 lymph nodes, only in neck to my knowledge through mri nd ct, largest being on my parotid measuring almost 3.5cm and 2 on my jugular. It is alittle disheartening to accept that i can only control for this as apparently even after excision, they will likely return, along with the kidney, salivary gland and lung complications. Not really willing to accept surgeries and steriods for long term periods or even life. The pathology report shows no information under microscopic description as my core needle biopsies did, just under dx it says Kimuras disease. He has now asked me to return in a month so he can gather more info and try to help me. Even resources on it are rare, no clinical trials or patient organizations on the western side. Can any one give any advice/insight on whether i should attempt another opinion or whether this is a sure thing??
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2020.08.22 01:00 SnooEpiphanies7066 What does this radiology report mean?

Axial CT imaging was performed from the skull base to the upper chest with 5 mm axial, sagittal and coronal reconstructions. 100 mL of Isovue 300 utilized for contrast enhancement. Area of interest is noted with Beekley spot marker.
CT imaging demonstrates normal symmetric parotid and submandibular salivary glands. There are sub-1 cm submental nodes noted. There are 2 adjacent lymph nodes just inferior to the right submandibular salivary gland at the area of interest. The largest measures 8 x 13 mm. Similar nodes are seen in the left submandibular neck with at least one node measuring 13 x 17 mm. No asymmetry of the adenoidal or tonsilar regions. There is no abnormality of the carotid neurovascular bundle on the right or left. There are no enlarged nodes in the anterior or posterior cervical chain. There is no abnormality of the nasopharynx or hypopharynx. The proximal trachea and proximal esophagus are unremarkable. There is no supraclavicular or superior mediastinal lymphadenopathy. There are normal symmetric thyroid glands. Bone windows do not demonstrate abnormality of the cervical spine.
RADIOLOGY RESULTS Impression 2 small adjacent lymph nodes are seen inferior to the right submandibular salivary gland in the area of interest. The largest measures 8 x 13 mm. Similar nodes are seen in the left submandibular neck with at least one node being enlarged measuring 13 x 17 mm. Bilateral anterior cervical chain nodes likely representing reactive nodes. Clinical correlation and follow-up as clinically indicated.
submitted by SnooEpiphanies7066 to u/SnooEpiphanies7066 [link] [comments]


2020.07.15 05:48 TomBulju Lumps under my chin cause pain when I yawn

I can't post a clear pic of this because it happens rarely, unpredictably, and I have a beard anyway so it's probably unnoticeable.
So a couple years ago I started having this weird problem when I yawned. Occasionally, these two lump-like things (salivar glands or submental lymph nodes, perhaps?) stick out from under my chin and get stuck under my jawbone, causing acute pain. Most of the time I have to physically push them back in with my fingers to make the pain stop. I've looked online and I doubt it's some form of eagle syndrome, since that's usually felt in the back of the jaw, or some sort of tumor, since I cannot feel them at all when I'm not yawning.
All it does is cause pain for a couple seconds at most, not interrupting my breathing or anything, so I've lived with this problem for some time now, but I finally decided to get it checked out once this whole covid situation calms down. Until then, you guys have any idea what it might be?
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2020.07.02 18:50 nbmehero Step 1 - Free120 [July 2020] - Question and Answer Discussion

Hello /NBME,
This post will contain the answers to the Step 1 - Free120 July 2020 Exam for educational discussion purposes. More info may be found here.
Please remember to read the following rules carefully before contributing:
  1. Read the Comment Rules and Policies found here.
  2. Do not quote, link to, or otherwise reproduce any exam content.
  3. Paraphrase as much as possible.
  4. Remember to be kind and thank you for your contribution in advance.
Block 1
  1. Binding to the 30s ribosomal protein
  2. Decreased activity of UDP glucuronosyltransferase
  3. Cholinesterase inhibitor
  4. Formation of antibodies to RhD
  5. BP 85/60, Pulse 120, JVP – increased, Pulsus Paradoxus – increased
  6. Metformin
  7. Petechiae
  8. Immune complex deposition in tissues
  9. Reassure the patient that her chance of becoming addicted to narcotics is minuscule
  10. CD28 on T lymphocytes  CD80 on epidermal Langerhans cells
  11. Fatty acid oxidation
  12. Open-labeled clinical trial
  13. Hypercoagulability from advanced malignancy
  14. Sertoli-Leydig tumor
  15. Separation of endothelial junctions
  16. Upward gaze
  17. Atrial fibrillation
  18. Hair follicle
  19. Elaboration of proteases and urease with local tissue destruction
  20. Common fibular (peroneal)
  21. Pouch of Douglas
  22. TNF-alpha
  23. Basement membrane
  24. Atropine
  25. Aspiration
  26. “You sound upset. Tell me a little more about that.”
  27. Third-degree atrioventricular block
  28. Parathyroid gland
  29. C
  30. “It is tough to change your diet and fluid intake, but what sorts of things were you doing at first when you were following the recommendations?”
  31. Gene rearrangement
  32. Androgen insensitivity
  33. E. Coli
  34. “Could you tell me your thoughts about the hormone treatment option we have discussed?”
  35. 0.2
  36. Foreign peptides bound to class I MHC are recognized by CD8+ T lymphocytes
  37. Baroreceptor output
  38. Type I pneumocytes – decreased, Type II pneumocytes – increased, fibroblasts – increased
  39. Alpha-toxin
  40. Graft-versus-host disease
Block 2
  1. P2 louder than A2
  2. Accumulation of acetaldehyde
  3. Infraspinatus
  4. Depolymerization of microtubules
  5. It is a polymorphism
  6. “Thank you for waiting. How can I help you today?”
  7. Arterial Baroreceptor Firing Rate – decreased, systemic vascular resistance – increased, pulmonary vascular resistance – increased, systemic capillary fluid transfer – absorption
  8. Omental bursa (lesser sac)
  9. 0.67
  10. Telephone Interpreter
  11. Increased hydrostatic pressure
  12. Trinucleotide repeat expansion
  13. Branch of thyrocervical trunk
  14. Adverse drug effect
  15. Splitting
  16. Case series
  17. Myeloperoxidase
  18. DNA virus
  19. Ingestion of soil
  20. Antigenic variation
  21. The patient’s DMD carrier status is uncertain because of random X inactivation
  22. Von Willebrand Disease
  23. Membranous
  24. C5a
  25. Cyclin-dependent kinases
  26. Metabolism
  27. Tampons
  28. Superficial inguinal
  29. Normal development
  30. Sensation over the upper lip
  31. Orthostatic hypotension
  32. Gastric mucosa
  33. Acting out
  34. ACE inhibitors
  35. Leukotrienes
  36. “Tell me what you know about steroid inhalers and how they are supposed to work.”
  37. Glycogenolysis
  38. Crohn Disease
  39. Decreased activity in enzyme that reduces Fe3+ to Fe2+
  40. Strep pyogenes (group A)
Block 3
  1. Decreased osteoclast activity
  2. Statistically significant overall increase in risk
  3. Increases NO concentration
  4. Vascular endothelial growth factor
  5. Adenylyl cyclase
  6. Mediation of cell entry via fusion protein
  7. Beta-2 agonist
  8. Infectious mononucleosis
  9. G1
  10. Schistosomiasis
  11. 50%
  12. TTP
  13. Hemopneumothorax
  14. HDL – increased, Triglycerides – decreased
  15. Vitamin B12
  16. Stimulation of the sebaceous glands by androgens
  17. Apoptosis
  18. Submental
  19. Olfactory nerve
  20. PCP
  21. Significant findings can be reported with greater confidence
  22. Hypogonadotropic hypogonadism
  23. Pulmonary hypoplasia
  24. Galactose-1-phosphate
  25. Ribosomal assembly
  26. Atrophy
  27. Avoids the concern for reversion to virulence
  28. Inactivation of cellular p53
  29. Erythropoietin
  30. Total peripheral resistance
  31. Adenovirus
  32. Hypertension and muscle weakness
  33. Patellar ligament
  34. Radon
  35. Amygdala
  36. The second CFTR mutation was not detected by the testing obtained
  37. “You must miss your daughter very much. Tell me about her.”
  38. Normal development
  39. Basal ganglia
  40. Normal findings
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2019.10.04 01:56 ChiaReally Lymph Nodes Enlarged 2 Months+

Hello Reddit doctor community, I'm now extremely concerned about the lymph nodes on the side of my neck being enlarged. I'll be as thorough as I can with my explanation of symptoms.
General info: 28 y/o male, 5'8", 130lb. Pack/day smoker for 10 years. Daily marijuana use. No other drugs.
May of this year I had the lower wisdom tooth on the right side removed. I had a few issues that the DMD told me was nothing to worry about (tongue went numb on the right side occasionally, metallic taste in mouth, enlarged taste bud on the right side of my tongue, swollen salivary glands on the same side). Those problems did seem to go away as healing progressed, but I do still get the numb tongue rarely.
July of this year, as I was moving from one apartment to another, 3 of my cervical lymph nodes on the right side swelled up. The upper most was painful without touching it, the other 2 below were just enlarged. None of them have ever been larger than the size of a pea. The next day, the pain without touching it went away, but the upper most and the middle one were still painful to the touch. They still are now on very rare occasions.
I took 2 rounds of antibiotics (clindamycin, 300mg then 150mg) because I thought the lymph nodes were caused by oral infection. The wisdom tooth was impacted and infected before removal, and if I remember correctly so was the tooth next to it, but that was 2, almost 3 months prior. That tooth had a root canal that I'm still in the process of getting crowned (temporary crown in now, permanent goes in on Monday Oct 7)
Not sure if correlation = causation, but the lymph nodes did seem to get a little smaller after the antibiotics but weren't entirely back to normal. I was also having other issues, it felt like there was pressure in the right side of my neck and like something was stuck in my throat on that side when I swallowed. Speaking of swallowing, I had to basically force myself to swallow "on that side" (if that makes sense. If felt like something was blocking/resisting air and saliva on that side)
That's when I decided to book an appointment at a clinic, and then another just to get a second opinion. Nobody really gave me a straight answer, citing everything from allergies, physical/mental stress, and bacterial/viral infection.
Unsatisfied and still with enlarged lymph nodes, I pushed to have bloodwork and an ultrasound done. Bloodwork and ultrasound results came back normal. At this point, I asked for a referral to an ENT. Saw the ENT, got a nasal endoscopy which showed everything was normal. A few days prior to this, I noticed there was a lump by my submental node on the right side. I'm fairly certain it wasnt there before, because I had been obsessively checking my mouth/neck/throat for the whole 2 months this ordeal had been going on for.
I now have an appointment for a CT scan this upcoming Thursday (Oct 10). I'm not worried that its cancer or anything like that, but I am still concerned. Hence this post.
Also, when I swallow, I feel... maybe a muscle or a tendon moving around? But wouldn't the endoscopy have shown something like that? Also, I cant get my ear on that side to "pop" when performing the Valsalva maneuver. The left side lets a little burst of air out, but the right side... I'm lucky to get anything out of. Even just generally swallowing I can feel air come out the left but rarely the right.
I'm a mechanic, and my hair is the perfect length to let sweat drip into my ear when I'm working. I'm thinking maybe some dirt got in my ear that way, or even a sliver of rusty metal? I dunno.
The summarized version:
What could the potential cause(s) be at this point? Will my lymph nodes ever go back to normal? Will my throat/neck ever feel normal again?
Will update with CT scan results when I get them. If CT scan shows everything is normal, and the symptoms/nodes dont subside, what's my next course of action?
EDIT: my right ear "pops" when I swallow if I lay down and angle my neck certain ways, or after pulling/digging around my ear.
Thank you very much in advance, I hope the information was helpful/thorough enough.
submitted by ChiaReally to AskDocs [link] [comments]


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